1. 'Betamix B' is a herbicide produced by the drug company Bayer. The chemical is designed to kill broadleaf weeds, but not the plant that farmers want to grow and produce a bountiful harvest/yield → e.g. sugar beet and strawberry. Betamix B helps these two plants by binding to the PQ (plastoquinone) molecules of certain weeds and stopping them from passing electrons in a certain membrane. Describe how blocking this shuttle/carrier molecule from performing its job will eventually kill the weed, and refer to the two types of light dependent electron pathways PQ is involved in. Do not feel that you need to mention each electron shuttle molecule in the redox reactions. If you wish, sketch a diagram to help explain your answer.

1. 'Betamix B' is a herbicide produced by the drug company Bayer. The chemical is designed to kill broadleaf weeds, but not the plant that farmers want to grow and produce a bountiful harvest/yield → e.g. sugar beet and strawberry. Betamix B helps these two plants by binding to the PQ (plastoquinone) molecules of certain weeds and stopping them from passing electrons in a certain membrane. Describe how blocking this shuttle/carrier molecule from performing its job will eventually kill the weed, and refer to the two types of light dependent electron pathways PQ is involved in. Do not feel that you need to mention each electron shuttle molecule in the redox reactions. If you wish, sketch a diagram to help explain your answer.

Name: 1. 'Betamix B' is a herbicide produced by the drug company Bayer. The
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Description: 1. 'Betamix B' is a herbicide produced by the drug company Bayer. The chemical is designed to kill broadleaf weeds, butnot the plant that farmers want to grow and produce a bountiful harvest/yield → e.g. sugar beet and strawberry. Betamix Bhelps the

Biology (MindTap Course List)

11th Edition

ISBN:9781337392938

Author:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Publisher:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Chapter6: Cell Communication

Section: Chapter Questions

Problem 4TYU

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You are researching the regulation of cell-size control and discover that a signaling pathway that acts through a particular enzyme-coupled receptor is important for the size growth (enlargement) of rat kidney cells. Receptor activation causes activation of adenylyl cyclase, which ultimately leads to the activation of PKA. PKA then phosphorylates a transcription factor called BTS on threonine 84. This phosphorylation is required to allow BTS to bind to specific regulatory DNA sequences, increasing the transcription of Zrt2, a gene encoding a protein vital for kidney cell growth. You find that kidney cells without this receptor are 25% smaller than normal cells. When you measure cells expressing a constitutively active version of PKA, you find they are 25% bigger than normal kidney cells. Based on these results, predict whether a kidney cell’s size would be bigger or smaller than normal kidney cells in each of the following circumstances. Explain why in 1-2 sentences for each. You add…
1. Embryonic stem cells continuously divide and remain undifferentiated. In order to maintain this pluripotency and continue to divide, which gene set(s) must be expressed? What specific components of cell communication activate the expression of these gene sets? There are 2 parts to this question, be sure to answer both.
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