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- The protein on the right called desmolaris, also called Draculin, found in bats. It is a non competitive inhibitor of Factor Xa. So what is this protein for? Why inhibit Factor Xa?Endocrine signals are transmitted more slowly than paracrine signals because.___ the ligands are transported through the bloodstream and travel greater distances the target and signaling cells are close together the ligands are degraded rapidly the ligands don't bind to carrier proteins during transport6)During the epinephrine signaling pathway we learned about in class, the enzyme glycogen phosphorylase is regulated by: Zymogen activation Competitive inhibition Allosteric regulation Covalent modification
- You can choose one or more than one option Arginase is an enzyme that: is expressed in the cytoplasm of hepatocytes shows two isoenzymes in mammals. found in the liver and other tissues catalyzes the formation of urea and ornithine from arginine. induces hyperammonemia when it is overexpressed is inhibited by basic amino acids such as lysine and ornithine.1a. Contrast the activation of cytokine receptors to the activation of receptor threonine kinases. b. We discovered a new cytokine that stimulates cancer cell growth. Now we want to determine if the JAK/STAT pathway is activated after stimulation. In the lab we have a small molecule that can competitively bind to SH2 domains. Explain where and how in the pathway this inhibitor would be effective.Coenzyme Q-10 (Ubiquinone) is one of the most important and crucial substances within the human body. The heart muscle normally has very high levels of CoQ-10, and when these levels drop by 75%, it will stop pumping. When CoQ-10 (Ubiquinone) levels drop by 25% within the body, diseases usually occur. Why does this happen?
- Explain the function(s) of Hsp90. Name two factors which lead to the increase of this protein in cells.Histamine binds to the H1 G-protein-linked receptor to initiate the itchiness and airway constriction associated with an allergic response. If a mutation in the associated G-protein’s alpha subunit prevented the hydrolysis of GTP how would the allergic response change? More severe allergic response compared to normal G-protein signaling. Less severe allergic response compared to normal G-protein signaling. No allergic response. No change compared to normal G-protein signaling.Can you explain the regulation of PFK-1 by ATP and AMP?
- 1) Individual with the genetic disease of Familial hypercholesterolemia have higher levels of cholesterol in the blood, which of the following could cause this disease: (A) deregulated biosynthesis of low-density lipoprotein (LDL) receptors (B) high dietary cholesterol (C) plaque formation in the arteries. (D) high cholesterol biosynthesis in liver tissue. (E) deregulated biosynthesis of low-density lipoprotein (LDL) 2) The enzyme inorganic pyrophosphatase utilizes ____ as its substrate. A). ATP B). ADP C). AMP D). PPi E). phosphateI come across a sentence "Myc mutations increase the lethality of Ras inhibitors", but am not sure if an increase in the lethality of Ras inhibitors is a good thing or bad thing. I should be grateful if the expert could help and advise.What is the effect of an inhibitor binding an enzyme? The enzyme is degraded. The enzyme is activated. The enzyme is inactivated. The complex is transported out of the cell
