All patients with STEMI, NSTEMI, and UA should be discharged on a(n): intensive HMG-CoA reductase inhibitor oral unfractionated heparin oral suspension of acetaminophen oral sodium channel blocker
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All patients with STEMI, NSTEMI, and UA should be discharged on a(n):
- intensive HMG-CoA reductase inhibitor
- oral unfractionated heparin
- oral suspension of acetaminophen
- oral sodium channel blocker
Step by step
Solved in 3 steps
- In people with diabetes mellitus Type 1, explain the reason for:(a) ketoacidosis, and (b) ketonuria.Indicate whether the following sentences is True or False: During prolonged starvation or in uncontrolled diabetes mellitus, the formation of acetyl-CoA exceeds oxaloacetate supply, forming ketone bodies Hypoglycaemia may be observed in Cushing syndrome patients The range of albuminuria that is considered as a reversible and treatable is 30 – 300 mg/24hr (20 – 200 μg/min) Urea is the major nitrogen-containing metabolic product of protein catabolism in humans, accounting for > 75% of the nonprotein nitrogen eventually excreted GLUT5 is found in small intestine and other organs to transport fructoseAlcoholism is often associated with hepatomegaly (due to lipid deposits) and lactic acidosis. Explain the metabolism
- Individuals with hyperammonemia are given a-ketoacids as a treatment. Explain.Elevation of serum amylase and lipase is commonly seen in: Acid reflux disease, Acute appendicitis,Acute pancreatitus, or Gallbladder disease?In 2-page worth of words (around 500), discuss CYCLAMATE's regulation, allowable levels, and what group of people are at high risk for the side effects of CYCLAMATE in the body.
- What is the most dangerous adverse effect following use of biguanides?A. Lactic acidosisB. HypoglycaemiaC. Diabetic ketoacidosisD. HyperosmolalityE. HyperglycaemiaDefine the following terms:a. prenylationb. steroidc. digitalisd. lipoproteine. apolipoproteinHow does the pathophysiology of diabetes ketoacidosis differ from hyperosmolar non-ketonic coma? (easy and simple)
- Many patients with glucose 6- phosphatase deficiency have high serum levels of urate. Hyperuricemia can be induced in normal people by the ingestion of alcohol or by strenuous exercise. Propose a common mechanism that accounts for these findings.Indicate what will happen ( increase, decrease or no effect) a. release of glucagon in the blood to the activity of carnitine acyl transferase 1 b. high malonyl CoA to the activity of carnitine acyl transferase I C. Epinephrine to the activity og glycogen synthase d. high citrate to the activity of acetyl CoA carboxylase e. high acetyl CoA to ketogenesisA person is suspected with diabetes mellitus. He checks his blood glucose level by gluceose oxidase method. After the experiment, OD of his blood sample was obtained as 0.08. Based on the glucose standard curve seen in the virtual lab, calculate his blood glucose level. Is he had diabetes mellitus? (Note: Normal blood glucose level in our body is 70-110mg/dl). Find out the role and chemical reactions of the following reagents in the blood glucose level determination by glucose oxidase method. a) Sodium sulphate – Zinc sulphate solution b ) Glucose oxidase reagent c) Peroxidase A person did estimation of blood glucose level by glucose oxidase method by referring the virtual lab procedure. But he forgot to add ortho - toluidine reagent during glucose oxidase reagent preparation. a) What is the observation of the result?…