At the axon terminus, gates for a new ion open. What is the ion and which direction does it flow across the axon membrane? Neurotransmitters move into the cell Chloride ions move out of the cell Calcium ions move out of the cell. Neurotransmitters move out of the cell. Calcium ions move into the cell. Chloride ions move into the cell.

At the axon terminus, gates for a new ion open. What is the ion and which direction does it flow across the axon membrane? Neurotransmitters move into the cell Chloride ions move out of the cell Calcium ions move out of the cell. Neurotransmitters move out of the cell. Calcium ions move into the cell. Chloride ions move into the cell.

Biology: The Dynamic Science (MindTap Course List)

4th Edition

ISBN:9781305389892

Author:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Publisher:Peter J. Russell, Paul E. Hertz, Beverly McMillan

Chapter39: Information Flow And The Neuron

Section: Chapter Questions

Problem 1TYK

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What causes Hyperpolarization following the spike of the action potential? Voltage-gated K+ channels close slowly allowing K+ ions to leave the cell The opening of leaky Na+ channels The Na+/K+ pump briefly reverses the direction of ion flow Both Na+ channels and K+ channels become inactivated Na+ and Cl- enter the cell until the Na+/Cl- pump closes
When a neurotransmitter binds with receptors at the membrane, it may cause sodium channels to open. An inflow of sodium causes the membrane to partially depolarize and become less negatively charged. For example, the voltage might change from -70 mV to -65 mV. This partial depolarization of the dendrites and cell body acts as: an EPSP, exciting the cell and increasing the likelihood of action potentials. an IPSP, inhibiting the cell and reducing the likelihood of action potentials. none of these options; depolarization has no excitatory or inhibitory effect on a neuron. both an EPSP and and IPSP.
If the neuron is stimulated such that its membrane potential changes to 50 mV, what is the sign of the driving force for Cl- ions and in which direction will these ions move across the membrane? Positive driving force; Cl- moves into the cell Positive driving force; Cl- moves out of the cell Negative driving force; Cl- moves into the cell Negative driving force; Cl- moves out of the cell
Which of the following describes a threshold potential? A brief reversal of the charge difference across a neuron's plasma membrane The membrane potential at which voltage-gated sodium channels in a neuron axon open, causing an action potential The membrane potential of a neuron at rest
If you inhibit the closing of sodium channels what do you predict the effect on neuronal function will be? If you inhibit the closing of sodium channels what do you predict the effect on neuronal function will be? Hyper-excitability: more sodium ions flow into the cell, causing neurons to fire more readily Hypo-excitability: more sodium ions flow into the cell, causing neurons not to fire as readily Less sodium is pumped out of the cell, slowing down return to the resting state Sodium ions are toxic to neurons, leading to cell proliferation
An action potential is generated at the... trigger zone of the axon postsynaptic membrane cell body axon terminal
The changes in membrane potential when a neuron is transmitting information is called ..... threshold action potential chemical potential resting potential
Which of the following is FALSE about action potentials? At resting potential, there are more potassium channels open and most sodium channels are closed. Depolarization is due to the flow of Na+ out of the axon via voltage-gated Na+ channels. Hyperpolarization is due to continued loss of K+ from the axon. During repolarization, K+ flows out of the axon along its concentration gradient. Resting membrane potential is primarily established by the Na+/K+ pump.
Which of the following is NOT an example of communication that happens from one neuron to another neuron? A. Endocrine signaling (ex. hormones secreted into the blood stream travel and bind to receptors on dstant cells). B. Retrograde neurotransmission (ex.the dendrite releases a neuroactive substance that binds to receptors on the axon terminal of a presynaptic neuron). C. Electrical communication via propagation of action potential from one node of ranvier to the next node of ranvier. D. Volume transmission (ex. neuropeptides diffuse away from the synapse and bind to receptors on adjacent cells) E. Electrical communication through gap junctions.
How can molecules pass through a closed gate such as in ligand-gated channel protein? Group of answer choices Receptor must receive pressure to open the gate. Receptor must receive electrical signals to open the gate. Receptor must receive signals or stimulus from the neurotransmitter to open the gate. The ligand-gated channel protein is always open, and it does need any stimulus
The voltage produced by a single nerve or muscle cell is quite small, but there are many species of fish that use multiple action potentials in series to produce significant voltages. The electric organs in these fish are composed of specialized disk-shaped cells called electrocytes. The cell at rest has the usual potential difference between the inside and the outside, but the net potential difference across the cell is zero. An electrocyte is connected to nerve fibers that initially trigger a depolarization in one side of the cell but not the other. For the very short time of this depolarization, there is a net potential difference across the cell, as shown. Stacks of these cells connected in series can produce a large total voltage. Each stack can produce a small current; for more total current, more stacks are needed, connected in parallel. In an electric eel, each electrocyte can develop a voltage of 150 mV for a short time. For a total voltage of 450 V, how many electrocytes must…
What is responsible for action potentials propagating in one direction? The inactivation of voltage gated sodium channels The sodium potassium pump and the potassium leak channels The inactivation of voltage gated potassium channels The inactivation of ligand gated sodium channels The inactivation of ligand gated potassium channels