exaplin in detail the concept of the multi-hit hypothesis of adenocarcinoma in regards to bowel cancer. (colon/recum) Provide full explanation Give exmaples and steps
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exaplin in detail the concept of the multi-hit hypothesis of adenocarcinoma in regards to bowel cancer. (colon/recum)
Provide full explanation
Give exmaples and steps
Step by step
Solved in 2 steps
- Explain the importance of MSH2 and MSH6 in regards to adenocarcinoma colon cancerExplain detail the multi hit hypothesis for Adenocarcinoma and gastrointestinal stromal tumors. Provide examplesexplain and proivde anylyses what are the diagnoses and current and potential future treatments for adenocarcinoma tumours colorectal cancer (colon). And Make linkadge too CIN, MSI, CIMP pathways. Give detailed examples of treatments for the disease.
- Explain What are the advantages and disadvantages of CCL22, SERPINE1, TIMP1, CX3CL1 genes to adenocarcinoma tumours in regards to bowel cancer. Provide evidence.Provide evidence on the sequelae on how adenocarcinoma tumours (bowel cancer) can be develp in the human body.explain how do the CIN MSI CIMP pathways all link together in develpment of adenocarcinoma tumours colorectal cancer. Give full explanation if there do or do not link.
- The development of colon cancer is particularly well- studied, and often has a predictable order of mutations. Briefly describe the most common steps in the progression of colon cancer in the order of key genes mutated.explain and analyse each gene mutaition in the CIN, MSI, CIMP pathways and what are the causes of these mutations in regards to adenocarcinoma tumours colorectal cancer. Provide detailed analysis with exampleswhat are the possile outcomes, pros and cons of adenocarcinoma tumours regards to bowel cancer to the human body. provide examples and back with evidance.
- Explain and describe the pros ans cons of adenocarcinoma tumours in regards to bowel cancer.1. the wnt story:- a) Discuss the role of Wnt pathway in the normal development of epithelia within the colon – stem cells, differentiated epithelia, and shedding epithelia. b)Then discuss how this path gets altered in the familial cancer FAP – familial adenomatous polyposis? How does it get impacted in non-hereditary colon cancer? Your discussion should explain how the altered wnt path leads to hyperplastic epitheliaEXPLAIN the consistency and colour of the contents of an ostomy pouch for an ileostomy versus a sigmoid colostomy and WHAT is the reason for this difference? *