In a series of infection experiments, a researcher discovers that the ID50 value for the infectious bacterium Parasiticum mucoides is 2,000, and that the ID50 for another infectious bacterium, Donoteatum thisbacterium, is 150. Given these data, a person exposed to 1,000O bacteria of each type would be more likely to be infected by which bacterium? Parasiticum mucoides Both infections are equally likely Donoteatum thisbacterium There is no way to know given the information provided
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- In a series of infection experiments, a researcher discovers that the ID50 value for the infectious bacterium Parasiticum mucoides is 100, and that the ID50 for another infectious bacterium, Donoteatum thisbacterium, is 15,000. Given these data, a person exposed to 1,000 bacteria of each type would be more likely to be infected by which bacterium? Group of answer choices There is no way to know given the information provided Both infections are equally likely Parasiticum mucoides Donoteatum thisbacteriumDescribe the lines of proof Robert Koch used to definitivelyassociate the bacterium Mycobacterium tuberculosis with thedisease tuberculosis. How would his proof have been flawedif any of the tools he developed for studying bacterialdiseases had not been available for his study of tuberculosis?Suppose you spilled two cultures of Salmonella typhimur-ium (each containing 100,000 cells) on your lab bench. You im-mediately applied the same disinfectant to both cultures at thesame time. One culture had been freshly grown for 36 hours andthe other culture is 2 weeks old. If the kill ing rate of the disin-fectant is 90% per minute, do you think that microbes in bothcultures will be completely killed after 6 minutes? Why or whynot?
- Suppose you do the Kirby-Bauer test on a hypothetical Staphylococcus species with penicillin and tetracycline. You record diameters of 20mm for tetracycline and 24mm for penicillin. Which antibiotic is most effective against this bacterium and why? Please explain and interpret these results.If a bacterial species is not susceptible to an antibacterial drug at the concentration present in a particular disk, does that necessarily mean the species is completely resistant to the drug? Explain your answer. Did you notice any colonies (isolated mounds of cells) growing within any of the zones of inhibition? If so, which plate(s) and which drug(s)? What would cause growth of colonies within a zone of inhibition? please answer both questions.There have been recurring cases of mad-cow disease in the United Kingdom since the mid-1990s. Mad-cow disease is caused by a prion, an infectious particle that consists only of protein. In 1986, the media began reporting that cows all over England were dying from a mysterious disease. Initially, there was little interest in determining whether humans could be affected. For 10 years, the British government maintained that this unusual disease could not be transmitted to humans. However, in March 1996, the government did an about-face and announced that bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, can be transmitted to humans, where it is known as variant Creutzfeldt-Jakob disease (VCJD). As in cows, this disease eats away at the nervous system, destroying the brain and essentially turning it into a spongelike structure filled with holes. Victims experience dementia; confusion; loss of speech, sight, and hearing; convulsions; coma; and finally death. Prion diseases are always fatal, and there is no treatment. Precautionary measures taken in Britain to prevent this disease in humans may have begun too late. Many of the victims contracted it over a decade earlier, when the BSE epidemic began, and the incubation period is long (VCJD has an incubation period of 10 to 40 years). A recent study concluded that 1 in 2,000 people in Great Britain carry the abnormally folded protein that causes VCJD. In spite of these numbers, the death rate from VCJD remains low. It is not clear whether this means that the incubation period for the disease is much longer than previously thought, or whether they may never develop the disease. How can a prion replicate itself without genetic material?
- How does Escherichia coli O157:H7 end up in groundbeef? To what class of pathogenic E. coli does this strainbelong? How does this class differ from other classes?A recent metagenomic study analyzed the microorganisms present on surfaces within the entire subwaysystem of New York City. The researchers found hundreds of bacterial species in the subway, most of themnonpathogenic. Interestingly, almost half of all DNAfound in the subway matches no known organism.a. The scientists found that different subway stationshad characteristic microbiomes. How might thisobservation be useful to the police?b. Because the majority of the subway DNA that couldbe identified was bacterial, the researchers presumethat most of the DNA fragments that could not bematched to a known organism are bacterial. Why doyou think that so many bacterial species are unknownto us? What feature of these unknown bacteriamight prevent us from studying them?write a brief summary on the KPC bacteria, What clinical significance does this organism have for humans? (What disease/infections does it cause in humans?) What is the mode of transmission to humans? (How do you get an infection with this organism?) What is the worldwide geographic distribution for this organism? (Where is it found, have there been any reported cases or outbreaks lately?)
- Why is E. coli O157:H7 an organism of concern in contaminated foods? The strain is represented as O157:H7. Which particular “parts” of the bacterial cell do the “O” and “H” refer to?You are treating swimming pools with ozone. You are using 0.02 ppm ozone to kill adenovirus, with an exposure time of 30 minutes. Will this treatment be effective on 2 log adenovirus ? Will this treatment also kill E. coli in the same water ? Explain your answer with calculationsAn infectious dose of several million cells in enteric infections seems like a lot. a. In terms of the size and abundance of microbes, could you see a cluster containing that many cells with the naked eye? b. About how long would it take an average bacterial species to reach the infectious dose of a million cells starting from a single cell? c. Why do enteric diseases require a relatively higher infectious dose than nonenteric diseases? d. Explain why antimicrobic therapy used on gram-negative bacterial infections could actually lead to illness rather than cure it