Repeated injections of very small amounts of antígen (a) Mast cell- IgE antibody- Antigen Granule containing mediators IgG antibody (b) FIGURE 17.3 Immunotherapy for IgE Allergles (a) Repeated injections of very small amounts of antigen are given over several months. (b) This regimen leads to the formation of specific IgG antibodies. The IgG reacts with antigen before it can bind to IgE, and therefore it blocks the IgE reaction.

Repeated injections of very small amounts of antígen (a) Mast cell- IgE antibody- Antigen Granule containing mediators IgG antibody (b) FIGURE 17.3 Immunotherapy for IgE Allergles (a) Repeated injections of very small amounts of antigen are given over several months. (b) This regimen leads to the formation of specific IgG antibodies. The IgG reacts with antigen before it can bind to IgE, and therefore it blocks the IgE reaction.

Human Heredity: Principles and Issues (MindTap Course List)

11th Edition

ISBN:9781305251052

Author:Michael Cummings

Publisher:Michael Cummings

Chapter17: Genes And The Immune System

Section: Chapter Questions

Problem 29QP

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Similar questions

An infant appears healthy until about 9 months of age, when he develops severe bacterial infections, one after another. Fortunately, theinfections are treated successfully with antibiotics. When infectedwith the measles and other viral diseases, the infant recovers withoutunusual difficulty. Explain the different immune responses to theseinfections. Why did it take so long for this disorder to become apparent? (Hint: Consider IgG.)
Whatresults in the specificity of an antibody (i.e. one antibody can bind only one particular antigen)? Whichtype of lymphocyte makes antibodies?
Why is it necessary for immature T lymphocytes toundergo a two-step selection process through whichantigen-reactive cells are first selected followed bythe elimination of cells that react strongly withself-antigens?
Identify cells that can function as antigen-presenting cells (APCs)
A given B cell expresses only maternally or paternally derived heavy chains but never both. This observation is the result of A. antibodydiversity.B. isotypeswitching.C. allelicexclusion.D. affinitymaturation.E. randomVJgenerearrangement.
It would be disastrous if a complement attack werenot confined to the surface of the pathogen that is the tar-get of the attack. Yet, the proteolytic cascade involved inthe attack liberates biologically active molecules at severalsteps: one that diffuses away and one that remains boundto the target surface. How does the complement reactionremain localized when active products leave the surface?
a. Describe what could be summarized as the three Rs of immunity:recognize, react, remember.b. Why would it be necessary for the T cells to bind both antigen andself (MHC) receptors?
Describe the typical shape and structure of an antibody. Distinguish between antibodies and antigens. Be able to draw an antibody and label the variable (antigen-binding) region and the control region. What results in the specificity of an antibody (i.e. one antibody can bind only one particular antigen)? Which type of lymphocyte makes antibodies?
If a series of genetic mutations prevented some, blit not all, of the complement proteins from binding antibodies or pathogens, would the entire complement system be compromised?
________triggers immune responses. a. Cytokines d. Antigens b. Lysozyme e. Histamines c. Antibodies f. MHC markers
If a series of genetic mutations prevented some, but not all, of the complement proteins from binding antibodies or pathogens, would the entire complement system be compromised?
if someone had dysfunctional plasma cells that were unstable to secrete immunoglobulins, what pathogen/s would they be most at risk from and why? Thank you!