Soil bacteria such as Streptomyces spp. produce the bulk of known antibiotics. It has been suggested that in nature antibiotics serve as signaling molecules, allowing members of the same species or strain to communicate. If this is the case, what might these molecular signals be “saying” and how would such a message be transmitted?

Soil bacteria such as Streptomyces spp. produce the bulk of known antibiotics. It has been suggested that in nature antibiotics serve as signaling molecules, allowing members of the same species or strain to communicate. If this is the case, what might these molecular signals be “saying” and how would such a message be transmitted?

Biology 2e

2nd Edition

ISBN:9781947172517

Author:Matthew Douglas, Jung Choi, Mary Ann Clark

Publisher:Matthew Douglas, Jung Choi, Mary Ann Clark

Chapter9: Cell Communication

Section: Chapter Questions

Problem 3VCQ: Figure 9.17 Which of the following statements about quorum sensing is false? Autoinducer must bind...

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Why might bacteria use quorum sensing to regulate genes needed for virulence? How might this reason be related to the rationale behind using quorum sensing to establish a symbiotic relationship?
What are the 3 pillars of signal transduction that are employed by bacteria for communication? Describe, compare, and contrast each type. Cite an example and elaborate the pathway (i.e., molecular process).
Identify which of the following statements is a lie? Select one: a. Bacterial biofilms due to quorum sensing can be found on medical equipment and implants such as hip or knee replacements or heart pacemakers. b. Quorum sensing uses autoinducers as signaling molecules. Autoinducers are signaling molecules secreted by bacteria to communicate only with the same species of bacteria. c. The first evidence of bacterial communication was observed in a bacterium that has a symbiotic relationship with Hawaiian bobtail squid.
In quorum sensing, why might a bacterium synthesize more than one type of signaling molecule?
Which of the following best describes the mechanism of action of the antibiotic streptomycin? A. it disrupts protein synthesis in resistant bacteria. B. Streptomycin inhibits cell wall synthesis in all bacterial species. C. Streptomycin creates mutations in bacteria which cause them to become resistant. D. It disrupts protein synthesis in nonresistant bacteria.
If you were developing a new “broad-spectrum” antibiotic to kill a widevariety of bacteria, which cell structures and pathways would you target?Which of those targets also occur in eukaryotic cells, and why is thatimportant? How would your strategy change if you were designing a new“narrow-spectrum” antibiotic active against only a few types of bacteria?
Please describe the defense role of RNAs in eubacteria and archaea by explaining how mechanistically protects organisms? Add information about similar functional RNAs in eukaryotic organisms by giving details about the cellular mechanism.
Figure 9.17 Which of the following statements about quorum sensing is false? Autoinducer must bind to receptor to turn on transcription of genes responsible for the production of more autoinducer. The receptor stays in the bacterial cell but the autoinducer diffuses out. Autoinducer can only act on a different cell: it cannot act on the cell in which it is made. Autoinducer turns on genes that enable the bacteria to form a biofilm.
Figure 14.23 shows a genetic switch that controls the choice between the lytic and lysogenic cycles of phage ?. What is a genetic switch? Compare the roles of a genetic switch and a simple operator site (like the one found in the lac operon) in gene regulation.
The pathway shown below is used by a fungus to create Arginine from a precursor starter molecule. A nonsense mutation occurs in the gene that encodes enzyme 2. This fungus is grown on media that only contains the precursor. Discuss the effects of this mutation on:(i) The function of Enzyme 2 (ii) The potential of the fungus to produce Ornithine
Which of the following is NOT a method by which bacteria can be resistant to antibiotics? Select one: a. Utilise an alternate pathway of gene expression that is not targeted by the antibiotic. b. Alter the binding site of the antibiotic, to either prevent it binding, or remove it from the site. c. Production of enzymes that bind to and break down the specific antibiotic. d. Production of a cell membrane pump that translocates antibiotics outside the cell. A potential side effect of antibiotic use is a secondary bacterial or fungal infection, usually associated with regions like the skin, upper-respiratory tract or genitalia. What is the most likely reason for the formation of these secondary infections? Select one: a. Antibiotics do not work on viruses, so they are able to establish infections in these locations. b. Organisms that are antibiotic resistant are usually more virulent, meaning they can survive and cause infection. c. The antibiotic kills off normal flora, providing an…
For a bacterial exotoxin with a typical A-B type toxin structure, removal of the A portion of the toxin would result in which of the following? The B portion remaining would still be able to bind to the target cell, but would not have toxic effects The remaining B portion would enter the cell and carry out its toxic activity, such as ADP-ribosylation The remaining B portion would cause inhibition of protein synthesis Nothing would happen because the A portion is responsible for binding to the target cell. The remaining B portion would still induce cell lysis Please answer asap and in short and content should not be palgarised please