tep 1 Staphylococcus aureus is a bacteria, that is now resistant to commonly used antibiotics, and it is now resistant to the antibiotic methicillin (narrow spectrum beta lactam class). step 2 Describe the physiological mechanism used by individual bacteria to resist the antibiotic listed in above of this Thought Question. For example, the mechanism could be modification of drug, modification of target, prevention of drug penetration,

tep 1 Staphylococcus aureus is a bacteria, that is now resistant to commonly used antibiotics, and it is now resistant to the antibiotic methicillin (narrow spectrum beta lactam class). step 2 Describe the physiological mechanism used by individual bacteria to resist the antibiotic listed in above of this Thought Question. For example, the mechanism could be modification of drug, modification of target, prevention of drug penetration,

Curren'S Math For Meds: Dosages & Sol

11th Edition

ISBN:9781305143531

Author:CURREN

Publisher:CURREN

Chapter10: Reconstitution Of Powdered Drugs

Section: Chapter Questions

Problem 4.2P

See similar textbooks

Similar questions

Results from a Kirby Bauer antibiotic assay on a Gram-negative bacterial culture are described as follows: A) the bacterium is resistant to penicillin, an antibiotic that targets synthesis of the peptidoglycan cell wall and B) the bacterium is resistant to tetracycline, an antibiotic that targets the small subunit of the ribosome, inhibiting protein synthesis. Which of the results represents intrinsic resistant and which represents acquired resistants?
You perform a Kirby-Bauer assay with two antibiotics. Antibiotic X has a zone of inhibition of 9 mm. Antibiotic Y has a zone of inhibition of 11 mm. Which antibiotic is better at killing this particular microorganism? Group of answer choices 1Antibiotic Y 2Antibiotic X and Y, which have identical antimicrobial activities 3Antibiotic X4 4It is impossible to tell from the information given
In what ways can you use aseptic technique principles in your daily life to preserve a healthy lifestyle, particularly in light of the CoVID-19 pandemic? Explain in four to five sentences.
The journal article "On the rapidity of antibiotic resistance evolution facilitated by a concentration gradient [See #1 for reference]" used the methods of "Mutational pathway determines whether drug gradients accelerate evolution of drug-resistant cells [See #2 for reference]". Reference used #1 : Hermsen, R., Deris, J. B., & Hwa, T. (2012). On the rapidity of antibiotic resistance evolution facilitated by a concentration gradient. Proceedings of the National Academy of Sciences, 109(27), 10775–10780. doi:10.1073/pnas.1117716109 #2 : Greulich, P., Waclaw, B., & Allen, R. J. (2012). Mutational Pathway Determines Whether Drug Gradients Accelerate Evolution of Drug-Resistant Cells. Physical Review Letters, 109(8). doi:10.1103/physrevlett.109.0881 QUESTIONS: 1. Based on Biological and Science manner, what is your comment/feedback on how the research articles/author presented their used material and methods?
Bacterial species that are harmful, as well as others that are beneficial, are found living in the human body. To slow the rate of developing antibiotic resistance in bacteria, physicians are being encouraged to use "narrow-spectrum" antibiotics- those that target only a few bacterial types. How can the information learned by antibiotic sensitivity testing be used by physicians who must choose antibiotics that inhibit the growth of bacteria causing disease but that do not interfere with beneficial bacteria?
A bacterium can protect itself against antibiotics in different ways. Describe 4 fundamentally differentmechanisms of antibiotic resistance. Discuss whether there are any basicdifferences or similarities between these 4 mechanisms and, if so, describe them.
With detail, compare and contrast the following 5 real-time assays; Taqman, SYBR Green, Molecular Beacon, FRET, and Scorpion. Please write complete sentences in paragraph form. Identify how the assays are similar but more importantly, what are the distinguishing features of each of the technologies.
Induced pluripotent stem cells have allowed scientists to model various diseases and screen drugs in these in vitro models. Please provide information on induced pluripotent stem cells and their use as described above (model various diseases and screen drugs in these in vitro models). Please explain clearly and nicely. THANKS
At what stage of the drug development pathways can molecular conjugation andbioconjugation based drug optimisations take place?
RESEARCH ARTICLE TITLE: "On the rapidity of antibiotic resistance evolution facilitated by a concentration gradient" Full Article Reseach: Hermsen, R., Deris, J. B., & Hwa, T. (2012). On the rapidity of antibiotic resistance evolution facilitated by a concentration gradient. Proceedings of the National Academy of Sciences, 109(27), 10775–10780. doi:10.1073/pnas.1117716109 QUESTION: 1. In biological/science manner, what are your comments on how the authors constructed their ARTICLE/RESEACH TITLE (see article).
Will the results of an in vitro antibiotic sensitivity test always agree with the results of an in vivo use of the antibiotic? Why or why not?
When using combination antibiotic therapy, it is important to administer drugs that work synergistically as possible. Which of the following represents a combination with known synergism? A beta-lactam and an aminoglycoside A penicillin and a cephalosporin Two drugs in which the second drug will displace the first from plasma protein-binding sites. Two drugs that are eliminated by different routes Two drugs that work on the same step in a metabolic pathway.