The body produces a normal amount of testosterone. However, a particular cell you are studying has an unusually low number of testosterone receptors. How might the cell's response to testosterone be affected by its low number of testosterone receptors? A. The cell will activate more protein kinases in response to testosterone exposure than a cell with a normal number of testosterone receptors. B. The cell will make less mRNA in response to testosterone exposure. C. The cell will make less cAMP in response to testosterone exposure. D. The cell’s response to testosterone exposure will be identical to the response of a cell with a normal number of testosterone receptors.

The body produces a normal amount of testosterone. However, a particular cell you are studying has an unusually low number of testosterone receptors. How might the cell's response to testosterone be affected by its low number of testosterone receptors? A. The cell will activate more protein kinases in response to testosterone exposure than a cell with a normal number of testosterone receptors. B. The cell will make less mRNA in response to testosterone exposure. C. The cell will make less cAMP in response to testosterone exposure. D. The cell’s response to testosterone exposure will be identical to the response of a cell with a normal number of testosterone receptors.

Biology (MindTap Course List)

11th Edition

ISBN:9781337392938

Author:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Publisher:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Chapter6: Cell Communication

Section6.4: Signal Transduction

Problem 3C

See similar textbooks

Similar questions

A particular drug inhibits the protein kinase that is responsible for phosphorylating the CREB protein. How would this drug affect the following events? A. The ability of the CREB protein to bind to CREs B. The ability of extracellular hormones to enhance cAMP levels C. The ability of the CREB protein to stimulate transcription D. The ability of the CREB protein to dimerize
What would be the likely result of a mutation of the bcl-2 gene on the level of apoptosis? a. No change b. A decrease in apoptosis c. An increase in apoptosis d. An initial decrease, followed by an increase in apoptosis
A mutation in p53 promoter that prevents p53 from being expresses Causes? Why? A. Increased proliferation B. Decreased proliferation
For cellular transduction, why is it useful that phosphorylation cascades have a series of protein kinases? a. they amplify the original signal b. they always lead to the same cellular response c. the number of molecules used is small and fixed d. they are cell specific
A mutation in Ras that prevents Ras from hydrolyzing GTP. Would cause? Why? A. Increased proliferation B. Decreased proliferation
How does an intracellular receptor regulate gene expression in the cell? Describe briefly what happens when a ligand like a hormone binds to one of these receptors
The frog Xenopus laevis has often served as a model system for the study of apoptosis. Can you think of some reasons that frogs are particularly good models for the study of apoptosis?
Explain how a kinase cascade works. Why is this advantageous in the context of hormone signaling?
What are the two major mechanisms through which cell signaling leads to a change in cell behavior?
A drug that inhibits the kinase activity of AKT would MOST likely have which of the following effects? A. Increased GSK-3b activity and increased CyclinD synthesis B. Increased GSK-3b activity and decreased CyclinD synthesis C. Decreased GSK-3b activity and increased CyclinD synthesis D. Decrease GSK-3b activity and decreased CyclinD synthesis
Why are steroids able to diffuse across the plasma membrane? a. Their transport protein moves them through the membrane. b. They are amphipathic, allowing them to interact with the entire phospholipid. c. Cells express channels that let hormones flow down their concentration gradient into the cells. d. They are non-polar molecules.
You stimulated the cell with a ligand and analyzed the response. Upon ligand treatment, the cell increased the expression of gene α and decreased expression of gene β. When you inhibit cyclic AMP production by treating the cell with an inhibitor of Adenylyl cyclase, the increased expression of gene α was not observed but the decrease gene β expression was still observed. a) What you can expect for gene α regulation by this ligand? What signaling mechanism could be used? b) To determine how gene β is regulated by the ligand via what signaling mechanism, provide your hypothesis and indicate a potential experiment that you can perform to test that.

SEE MORE QUESTIONS

Recommended textbooks for you

Biology (MindTap Course List)

Biology

ISBN:

9781337392938

Author:

Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Publisher:

Cengage Learning

Biology (MindTap Course List)

Biology

ISBN:

9781337392938

Author:

Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Publisher:

Cengage Learning

SEE MORE TEXTBOOKS

GET THE APP

About FAQ Academic Integrity Sitemap Document Sitemap

Contact Bartleby Contact Research (Essays)High School Textbooks Literature Guides Concept Explainers by Subject Essay Help Mobile App

GET THE APP

Privacy

Your CA Privacy Rights

Your NV Privacy Rights

About Ads

Manage My Data

bartleby, a Learneo, Inc. business

	A.	The cell will activate more protein kinases in response to testosterone exposure than a cell with a normal number of testosterone receptors.
	B.	The cell will make less mRNA in response to testosterone exposure.
	C.	The cell will make less cAMP in response to testosterone exposure.
	D.	The cell’s response to testosterone exposure will be identical to the response of a cell with a normal number of testosterone receptors.