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- 10) One of the field of studies listed includes the rest: a) Viral Nucleic acids type b) Viral classification c) Capsid symmetry d) Presence of an envelope e) Absence of an envelope. 11) The cells that line our respiratory tract, and one celled paramecium both have cell membrane modifications called: a) flagella b) microfilaments c) centrioles d) cilia e) pili. 12) A proteasome degrades proteins that are “tagged” with: a) Phosphates b) Ubiquitin c) Methionine d) CoQ enzyme e) Poly A tailThere have been recurring cases of mad-cow disease in the United Kingdom since the mid-1990s. Mad-cow disease is caused by a prion, an infectious particle that consists only of protein. In 1986, the media began reporting that cows all over England were dying from a mysterious disease. Initially, there was little interest in determining whether humans could be affected. For 10 years, the British government maintained that this unusual disease could not be transmitted to humans. However, in March 1996, the government did an about-face and announced that bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, can be transmitted to humans, where it is known as variant Creutzfeldt-Jakob disease (VCJD). As in cows, this disease eats away at the nervous system, destroying the brain and essentially turning it into a spongelike structure filled with holes. Victims experience dementia; confusion; loss of speech, sight, and hearing; convulsions; coma; and finally death. Prion diseases are always fatal, and there is no treatment. Precautionary measures taken in Britain to prevent this disease in humans may have begun too late. Many of the victims contracted it over a decade earlier, when the BSE epidemic began, and the incubation period is long (VCJD has an incubation period of 10 to 40 years). A recent study concluded that 1 in 2,000 people in Great Britain carry the abnormally folded protein that causes VCJD. In spite of these numbers, the death rate from VCJD remains low. It is not clear whether this means that the incubation period for the disease is much longer than previously thought, or whether they may never develop the disease. How can a prion replicate itself without genetic material?There have been recurring cases of mad-cow disease in the United Kingdom since the mid-1990s. Mad-cow disease is caused by a prion, an infectious particle that consists only of protein. In 1986, the media began reporting that cows all over England were dying from a mysterious disease. Initially, there was little interest in determining whether humans could be affected. For 10 years, the British government maintained that this unusual disease could not be transmitted to humans. However, in March 1996, the government did an about-face and announced that bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, can be transmitted to humans, where it is known as variant Creutzfeldt-Jakob disease (vCJD). As in cows, this disease eats away at the nervous system, destroying the brain and essentially turning it into a spongelike structure filled with holes. Victims experience dementia; confusion; loss of speech, sight, and hearing; convulsions; coma; and finally death. Prion diseases are always fatal, and there is no treatment. Precautionary measures taken in Britain to prevent this disease in humans may have begun too late. Many of the victims contracted it over a decade earlier, when the BSE epidemic began, and the incubation period is long (vCJD has an incubation period of 10 to 40 years). A recent study concluded that 1 in 2,000 people in Great Britain carry the abnormally folded protein that causes vCJD. In spite of these numbers, the death rate from vCJD remains low. It is not clear whether this means that the incubation period for the disease is much longer than previously thought, or whether they may never develop the disease. If you were traveling in Europe, would you eat beef? Give sound reasons why or why not.
- There have been recurring cases of mad-cow disease in the United Kingdom since the mid-1990s. Mad-cow disease is caused by a prion, an infectious particle that consists only of protein. In 1986, the media began reporting that cows all over England were dying from a mysterious disease. Initially, there was little interest in determining whether humans could be affected. For 10 years, the British government maintained that this unusual disease could not be transmitted to humans. However, in March 1996, the government did an about-face and announced that bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, can be transmitted to humans, where it is known as variant Creutzfeldt-Jakob disease (vCJD). As in cows, this disease eats away at the nervous system, destroying the brain and essentially turning it into a spongelike structure filled with holes. Victims experience dementia; confusion; loss of speech, sight, and hearing; convulsions; coma; and finally death. Prion diseases are always fatal, and there is no treatment. Precautionary measures taken in Britain to prevent this disease in humans may have begun too late. Many of the victims contracted it over a decade earlier, when the BSE epidemic began, and the incubation period is long (vCJD has an incubation period of 10 to 40 years). A recent study concluded that 1 in 2,000 people in Great Britain carry the abnormally folded protein that causes vCJD. In spite of these numbers, the death rate from vCJD remains low. It is not clear whether this means that the incubation period for the disease is much longer than previously thought, or whether they may never develop the disease. What measures have been taken to stop BSE?Figure 21.10 Which of the following statements is false? In the lytic cycle, new phages are produced and released into the environment. In the lysogenic cycle, phage DNA is incorporated into the host genome. An environmental stressor can cause the phage to initiate the lysogenic cycle. Cell lysis only occurs in the lytic cycle.Which of these statements is true? An antibiotic is any substance produced by a organism that is antagonistic to the growth of prokaryotes An antibiotic is any substance produced by a prokaryote that is antagonistic to the growth of other viruses An antibiotic is any substance produced by a prokaryote that is antagonistic to the growth of eukaryotic cells An antibiotic is any substance produced by a prokaryote that prevents growth of the same prokaryote.
- Which of the following events occurs in the lytic cycle of bacteriophage T4 infection but NOT in the lysogenic cycle? a.both digestion of host DNA and viral proteins synthesis b.attachment c.viral protein synthesis d.digestion of host DNA e.entryDuring viral infection, attachment is usually specific to a particular celltype becausea. the virus is attracted to the appropriate host cells by proteinssecreted into the extracellular fluid.b. the virus recognizes and binds to specific molecules in thecytoplasm of the host cell.c. the virus recognizes and binds to specific molecules on the surfaceof the host cell.d. the host cell produces channel proteins that provide passageways forviruses to enter the cytoplasm.e. the virus releases specific proteins that make holes in the membranelarge enough for the virus to enter.1. Transformation refers to .................................. Group of answer choices a.vertical transfer of genetic material b.transfer of genetic material via sex pili c.DNA fragment transferred between live F + donor and recipient cells d.using a virus to transfer DNA fragment e.the uptake of “naked” DNA fragment by competent cells 2. Identify the MISMATCH pair from the following Group of answer choices a.shake tube method :::: for cultivation of aerobic bacteria's b.glycerol uptake :::: facilitated diffusion c. gonorrheae pathogen ::::: prefers low oxygen d.SAB media :::: high salt and sugar with low pH e.defined media :::: simple media f.micronutrients :::: acts as cofactors in bacterial growth
- Gonorrhea is a sexually transmissible disease (STD) caused by the bacterium Neiserria gonorrhoeae. In the 1940s, penicillin was introduced as an antibiotic to treat gonorrhea. In particular, the drug inhibits the formation of new cell walls in dividing bacterial cells, making new cells vulnerable to the human body's defences. However, a new strain of Neiserria gonorrhoeae was found that is capable of breaking down penicillin, making this strain resistant. The emergence of this new strain has been associated with instances of non-strict compliance of certain patients on the use of penicillin as well as the improper administration of the drug. Based on the information given, formulate a strategy that could help control this new strain.1. Lytic phages lyse their host cells, whereas temperate phages either lyse their host or integrate their DNA into the host cell’s genome. Prophage DNA often codes for gene products that confer new properties on the host. Filamentous single-stranded DNA phages are extruded from the host cells without killing the cell. How can a productive phage infection not kill a host cell? 2. Peptidoglycan is a molecule unique to bacteria that provides strength to the cell wall. The Gram-positive cell wall is composed of a relatively thick layer of peptidoglycan as well as teichoic acids. Gram-negative cell walls have a thin layer of peptidoglycan and a lipopolysaccharide-containing outer membrane. Penicillin and lysozyme interfere with the structural integrity of peptidoglycan. What is the significance of lipid A?Match the terms 1Genome surrounded by a protein coat 2Functional viral particle capable to infect a new host cell 3Infectious protein 4Infectious agent that needs helper virus for infection 5T4 enzyme that creates holes in the E. coli plasma membrane 6When a temperate phage stimulates their bacterial host tostart producing a toxin (e.g. cholera, shiga) 7Virus that infects bacteria 8Hand N spikes 9Virus without an envelope 10Most common and best studies virulent phage of E. coli 11Protein coat that surrounds viral genome 12:true or false : some viruses can cause cancer [ Choose ] lambda True pathogenic stimulation False Nucleocapsid lysosome Naked virus induction lysozyme Flu viruses tobacco mosaic virus lysogeny capsid Lysogenic conversion Holin Prion satellite bacteriophage (or just phage) T4 Virion Streptococcus