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we read that in tumor cells Rb protein is hyperphosphorylated. In response to that, will p53 level increase or decrease? Why or why not?
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- In tumor cells Rb protein is hyperphosphorylated. In response to that, will p53 level increase or decrease? Why or why not? new answer pleasep53 is a gene / protein often associated with cancer. Why? What does p53 do? What kind of gene is it? Is it associated more with any one particular type of cancer or all cancers? Tell me more about p53, but please do not exceed one typed page.Which is a common cause for the production of oncogenes? a. defective ion channel proteins b. defects in DNA polymerase c. viral infection d. dephosphorylation of cyclin-dependent protein kinases e)exposure to infrared radiation Please answer asap and type your answer and do not copy from anywhere please answer asap
- What is your stand regarding STEM Cell research/medications/treatments? Limit your answer to 15 sentencesWhat is a stem cell? 2. How are embryonic stem cells di#erent from other types of stem cells? 3. Given their function, how do you think embryonic stem cells are used as a treatment for damaged cells? Please answer them in complete sentences, it’s not gradedIf our DNA contains all the information about who we are, how can something that does not involve changing the gene sequence cause changes in phenotypes? Please answer this immediately. Thank you!
- Which could increase your risk for developing cancer? A. continuously active p53B. continuously active pRbC. all of the aboveD. none of the abovePlease answer fast Gain-of-function mutations could contribute to cancer progression if the mutant gene is, a) p53 b) Rb c) cdks d) Wee1 2. Loss-of-function mutations could contribute to cancer progression if the mutant gene is, a) Rb b) Ras-GEF c) cdc25 d) cdks Help please! and explain if possibleMy question is two fold. What are some diseases that cause and/or are caused by apoptosis? Also if apoptosis is the most common form of programmed cell death in animals, what other forms of programmed cell death are there? Thank you.
- (x-linked genes q5) Please answer this question for mePlease answer fast and all question other wise I will give downvote. QUESTION 1 Why is cancer a disease of old age ? A. Older DNA is more easily damaged B. There are fewer stem cells to repair tissue in older people C. DNA mutations accumulate with age QUESTION 2 The frequency of this molecular change in cancer cells makes it readily detectable by liquid biopsy. A. Point mutations B. Gene amplification C. DNA methylation D. PSA antigen QUESTION 3 Can surgery (with conventional chemotherapy if necessary) ever cure cancer? A. Yes, but typically only when cancer is detected at an early stage B. No, it simply prolongs life, usually by a few months C. Yes, even advanced cancer can frequently be cured by chemotherapy D. Only when it is combined with a targeted therapyYou are in charge of a new gene therapy clinic. Two cases have been referred to you for review and possible therapy. Case 1. A mutation in the promoter of a proto-oncogene causes the gene to make too much of its normal product, a receptor protein that promotes cell division. The uncontrolled cell division has caused cancer. Case 2. A mutation in an exon of a tumor-suppressor gene makes this gene nonfunctional. The product of this gene normally suppresses cell division. The mutant gene cannot suppress cell division, and this has led to cancer. What treatment options can you suggest for each case?
