When a molecule is conserved through evolution, strong immunogenicity does not develop, this occurs because: a. The molecule would have many differences at the primary chain level between species b. The molecule has no significant structural differences c. The epitopes on that molecule do not vary much between species d. The immune system would not recognize these epitopes as foreign e. At least two of the above are true. If I transplant a serum with hemoglobin from a different species into a human, it will produce less immunogenicity than if I transplant a myosin molecule from another species. a. TRUE b. False

When a molecule is conserved through evolution, strong immunogenicity does not develop, this occurs because: a. The molecule would have many differences at the primary chain level between species b. The molecule has no significant structural differences c. The epitopes on that molecule do not vary much between species d. The immune system would not recognize these epitopes as foreign e. At least two of the above are true. If I transplant a serum with hemoglobin from a different species into a human, it will produce less immunogenicity than if I transplant a myosin molecule from another species. a. TRUE b. False

Human Heredity: Principles and Issues (MindTap Course List)

11th Edition

ISBN:9781305251052

Author:Michael Cummings

Publisher:Michael Cummings

Chapter9: Gene Expression And Gene Regulation

Section: Chapter Questions

Problem 1CS: There have been recurring cases of mad-cow disease in the United Kingdom since the mid-1990s....

See similar textbooks

Similar questions

A. What are the 4 “Koch’s Postulates”? B. Are they still valid today? How would we use them to establish the relationship between a specific microbe and a disease? Can we substitute such things as finding antibodies or DNA traces for some steps? If so, why? C. How has microbiology contributed to the advancement in molecular biology?
Which of these happen(s) in the case of antigenic shift in influenza A?a. single mutations in hemagglutininb. recombination of RNA segments between bird and human strainsc. change from influenza A to influenza Bd. both a and b
a. You have a test tube containing 5 ml of a solution ofbacteriophages, and you would like to estimate thenumber of bacteriophages in the tube. Assuming thetube actually contains a total of 15 billion bacteriophages, design a serial dilution experiment thatwould allow you to estimate this number. Ideally,the final plaque-containing plates you count shouldcontain more than 10 and fewer than 1000 plaques.b. When you count bacteriophages by the serialdilution method as in part (a), you are assuming aplating efficiency of 100%; that is, the number ofplaques on the petri plate represents exactly thenumber of bacteriophages you mixed with the plating bacteria. Is there any way to test the possibilitythat only a certain percentage of bacteriophageparticles can form plaques (so that the plating efficiency would be less than 100%)? Why is it fair toassume that any plaques are initiated by one ratherthan multiple bacteriophage particles?
There have been recurring cases of mad-cow disease in the United Kingdom since the mid-1990s. Mad-cow disease is caused by a prion, an infectious particle that consists only of protein. In 1986, the media began reporting that cows all over England were dying from a mysterious disease. Initially, there was little interest in determining whether humans could be affected. For 10 years, the British government maintained that this unusual disease could not be transmitted to humans. However, in March 1996, the government did an about-face and announced that bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, can be transmitted to humans, where it is known as variant Creutzfeldt-Jakob disease (VCJD). As in cows, this disease eats away at the nervous system, destroying the brain and essentially turning it into a spongelike structure filled with holes. Victims experience dementia; confusion; loss of speech, sight, and hearing; convulsions; coma; and finally death. Prion diseases are always fatal, and there is no treatment. Precautionary measures taken in Britain to prevent this disease in humans may have begun too late. Many of the victims contracted it over a decade earlier, when the BSE epidemic began, and the incubation period is long (VCJD has an incubation period of 10 to 40 years). A recent study concluded that 1 in 2,000 people in Great Britain carry the abnormally folded protein that causes VCJD. In spite of these numbers, the death rate from VCJD remains low. It is not clear whether this means that the incubation period for the disease is much longer than previously thought, or whether they may never develop the disease. How can a prion replicate itself without genetic material?
The evolutionary tree of HIV-1 revels that a.) The SIV strain that infects gorillas is the same strain that infects chimpanzees. b.) HIV is a monophyletic clade that originated from the same strain of SIV. c.) All of the answers are correct d.) The virus jumped from chimpanzees to humans four times. E.) western chimpanzees are more closely related to gorillas than eastern chimpanzees.
Griffith's Experiments using Streptococcus pneumonia rough and smooth strains concluded which of the following? a) DNA is the Molecule of Inheritance b) Some, yet unknown, factor of inheritance could be transferred from one bacteria to another. c) Protein if the Molecule of Inheritance d) Heat treating bacteria has NO effect on its virulence
Among the most prevalent pathologies that afflicthuman beings is heart disease, which can have a severeimpact on quality of life and can even result in prematuredeath. While heart disease mostly afflicts those who areolder, 1% or 2% of people in their 30s, and even in their20s, suffer from this disease. Genetic and environmentalcomponents of this disease exist. What strategy mightyou use to choose families to participate in a GWAS ofheart disease–causing genes? Explain your reasoning.
Which of the following two organisms are most likely to have the highest degree of homology in their hemoglobin proteins? Select one: a. dogs, wolves b. wolves, chimps c. white pine, red pine d. bees, flowers e. dogs, cats
In the Western blot shown here, proteins were isolated from redblood cells and muscle cells from two different individuals. Oneindividual was unaffected, and the other suffered from a diseaseknown as thalassemia, which involves a defect in hemoglobin. Theblot was exposed to an antibody that recognizes β globin, whichis one of the polypeptides that constitute hemoglobin. Equal totalamounts of cellular proteins were added to each lane. Explain these results.
If you extract the DNA of the coliphage φ X174, you willfind that its composition is 25 percent A, 33 percent T,24 percent G, and 18 percent C. Does this compositionmake sense in regard to Chargaff’s rules? How wouldyou interpret this result? How might such a phage replicate its DNA
Explain why the effect of a genotype on a phenotypecannot always be determined without knowing whatthe environment is, and why the effect of a particularenvironment on a phenotype cannot always be determinedwithout knowing what the genotype is.
Describe the significance of sickle cell anemia to understanding evolution and the role of culture in adaptation, and explain why it’s a great example of a balanced polymorphism.