The Ventricles Of The Heart

Early doctors were researching arrhythmia in heart beat as a result of unknown abnormal neuro-cardio mechanisms of the heart, one of theories was that SA and AV nodes were interfering with each other’s bio-electrical impulses another theory was that the right side of the septum was hypersensitive to electrical impulse, all were more else on the right track because we know now that SVT is a result of a faulty electrical connections of the heart.

Heart failure can be attributed to either right sided, left or both. Left-sided heart failure is of two types, systolic failure and diastolic failure. Systolic failure is the when the left ventricle loses its ability to contract normally. The heart cannot pump with enough force to push enough blood into circulation. Diastolic failure is when the left ventricle loses its ability to relax normally. Which results in the heart not being able to fill with blood during the resting period. Both result in a decrease in cardiac output. (AHA, 2012). A decrease in the cardiac output into the systemic circulation causes blood to accumulate in the left ventricle, left atrium, and pulmonary circulation. This increase

Based on the external observation, the left side of the heart appeared bigger than the right side. When looking at the heart internally, the right ventricle pumps the blood to the lungs, and the left ventricle to the rest of the body. Therefore the left ventricle needs to be stronger and bigger, than the right ventricle, as it has a larger role in the functioning the heart.

1. The pulmonary circuit is supplied by which ‘side” of the heart? The systemic circuit? The right atrium

How does the structure of cardiac myocytes and intercalated disks follow the function of cardiac muscle tissue

R E V I E W S H E E T 30 Anatomy of the Heart

Heart failure, HF, is a result of one’s heart inefficiently pumping blood out to the body (Lewis, Dirksen, Heitkemper and Bucher, 2014, p.766). A healthy heart will pump blood out of the left and right ventricles rhythmically and simultaneously, creating an even flow of blood from the heart to the pulmonary arteries and the aorta (Lewis et al., 2014, p.769). Someone with heart failure has a ventricular dysfunction in either one or both ventricles; the ventricles are not filling or contracting properly. The failure of one ventricle to properly function leads to an overcompensation of the opposite ventricle as well as a disruption in normal blood flow that leads

Most heart diagrams show the left atrium and ventricle on the right side of the diagram. Imagine the heart in the body of a person facing you. The left side of their heart is on their left, but since you are facing them, it is on your right.

This experiment is mainly focus on the cell development performance in different birth weight groups. The cardiac muscle hypotrophy and muscle weakness direct affect muscle function and cause disease. With the cellular study, it could better explain how the glucose pathways affect cardiac energy use and phenotype development. If the left ventricular hypotrophy phenotype has directly connect with glucose intake, then with this study, LBW ventricular muscle can be more sensitive to the signal pathway

The cardiac muscle cells can be affected by heart disease through the inability to maintain its homeostasis through the calcium pump. If there is excess calcium within the heart it results in the in ability of the heart muscle to relax. As a result, the ADP/ATP ratio increases, phosphocreatine decreases, and energy stores are depleted. Furthermore, this imbalance affects the energy needed to relax the left ventricle. Secondly, diastolic heart failure can occur from changes in preload, afterload, renin-angiotension-aldosterone system, and the sympathetic nervous system. These changes affect the fibrillar collagen within the extracellular matrix that affects the ability of the ventricular to relax. These changes along with those compensatory mechanisms results in continuous resistance that results in increased in left ventricular end-diastolic pressure (McCance & Huether, 2010).

In the valvular disease the regurgitation of blood back to the ventricles occurs when the valves fail to close tightly and this will result in ventricular overload and increased muscle stretching. This increases the heart muscles need for oxygen and energy resulting in the cardiac muscles to contract harder (Karch, 2013). The failure of the left ventricle to pump efficiently will lead to pulmonary vessel congestion and in severe cases, pulmonary edema whereas the inefficient pumping of right ventricle will lead to liver congestion and peripheral edema (edema of the legs and feet). The cardiovascular system works as a closed system and therefore, if one-sided failure left untreated, will eventually lead to failure of both sides (Karch, 2013). The American College of Cardiology (ACC)/ American Heart Association (AHA) has incorporated a classification system of heart failure that include four stages. This staging system (stage A to stage D) recognizes that there are established risk factors and structural abnormalities that are characteristics of the four stages of heart failure.

of atria and ventricle. Impulses not being transmitted from atria to the ventricle; no whole number relationship between atrial and ventricular contractions was demonstrated.

6. Automaticity – ability of heart to beat spontaneously and repetitively without external neurohormonal control. The heart is capable of beating outside the body, given proper laboratory conditions. Automaticity is evidently linked to fluid and electrolyte balance rather than to nervous system control.

reports the amount of force exerted by the blood into the arteries during ventricular contraction.

While contraction in skeletal muscle is triggered by motor neurons under central control, certain cardiac muscle variants exhibit autorhythmicity. This means that that they are capable of producing their own depolarizing electrical potential. The cardiomyocytes that are capable of producing their own electrical potentials are found in what is referred to as the electrical condition system of the heart. This system is comprised of specializes cardiomyocytes that are autorhythmic and are able to conduct electrical potentials rapidly. These specialized structures include the sinoatrial node, atrioventricular node and bundle, and Purkinje fibers.