al humbers of the different types of plaques (c+ m+,c¯ m¯,c+ m¯,c¯m+) should she expect to see? | ake 31. The geneticist carries out the same experiment CONNECT described in Problem 30, but this time she mixes phages with genotypes c+ m¯ and c¯ m+. Are the mutations in coupling or repulsion (see Chapter 7)? What results are expected from this cross? were *32. A geneticist isolates two bacteriophage r mutants (ſ13 and r, ) that cause rapid lysis. He carries out the following crosses and counts the number of plaques listed here: Genotype of parental phage Progeny Number of plaques ht r13 xh l13 A ht i3 1. minio e no shoh nattalg yadri104 110 ing メ4 rororiobuic cojourea 2. INT 217 h* rz* l2-AR-IT er 0 Total 12 9. - | 98. 81 180 Total a. Calculate the recombination frequencies between r, and h and between r3 and h. bi ns b. Draw all possible linkage maps for these three genes.

al humbers of the different types of plaques (c+ m+,c¯ m¯,c+ m¯,c¯m+) should she expect to see? | ake 31. The geneticist carries out the same experiment CONNECT described in Problem 30, but this time she mixes phages with genotypes c+ m¯ and c¯ m+. Are the mutations in coupling or repulsion (see Chapter 7)? What results are expected from this cross? were 32. A geneticist isolates two bacteriophage r mutants (ſ13 and r, ) that cause rapid lysis. He carries out the following crosses and counts the number of plaques listed here: Genotype of parental phage Progeny Number of plaques ht r13 xh l13 A ht i3 1. minio e no shoh nattalg yadri104 110 ing メ4 rororiobuic cojourea 2. INT 217 h rz* l2-AR-IT er 0 Total 12 9. - | 98. 81 180 Total a. Calculate the recombination frequencies between r, and h and between r3 and h. bi ns b. Draw all possible linkage maps for these three genes.

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Description: number 32 al humbers of the differenttypes of plaques (c+ m+,c¯ m¯,c+ m¯,c¯m+) shouldshe expect to see?|ake31. The geneticist carries out the same experimentCONNECT described in Problem 30, but this time she mixes phageswithgenotypes c+ m¯ and c¯ m+.

Human Heredity: Principles and Issues (MindTap Course List)

11th Edition

ISBN:9781305251052

Author:Michael Cummings

Publisher:Michael Cummings

Chapter11: Genome Alterations: Mutation And Epigenetics

Section: Chapter Questions

Problem 16QP: Familial retinoblastoma, a rare autosomal dominant defect, arose in a large family that had no prior...

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In an in situ hybridization experiment, a certain clonebound to only the X chromosome in a boy with no diseasesymptoms. However, in a boy with Duchenne musculardystrophy (X-linked recessive disease), it bound to theX chromosome and to an autosome. Explain. Could thisclone be useful in isolating the gene for Duchenne muscular dystrophy?
. The production of pigment in the outer layer of seedsof corn requires each of the three independently assorting genes A, C, and R to be represented by at leastone dominant allele, as specified in Problem 64. Thedominant allele Pr of a fourth independently assortinggene is required to convert the biochemical precursorinto a purple pigment, and its recessive allele pr makesthe pigment red. Plants that do not produce pigmenthave yellow seeds. Consider a cross of a strain of genotype A/A ; C/C ; R/R ; pr/pr with a strain of genotypea/a ; c/c ; r/r ; Pr/Pr.a. What are the phenotypes of the parents?b. What will be the phenotype of the F1?c. What phenotypes, and in what proportions, willappear in the progeny of a selfed F1?d. What progeny proportions do you predict from thetestcross of an F1?
. In a particular kind of ornamental flower, the wildtype flower color is deep purple, and the plants aretrue-breeding. In one true-breeding mutant stock, theflowers have a reduced pigmentation, resulting in alavender color. In a different true-breeding mutantstock, the flowers have no pigmentation and are thuswhite. When a lavender-flowered plant from the firstmutant stock was crossed to a white-flowered plantfrom the second mutant stock, all the F1 plants hadpurple flowers. The F1 plants were then allowed toself-fertilize to produce an F2 generation. The 277 F2plants were 157 purple : 71 white : 49 lavender. a. Explain how flower color is inherited. Is this traitcontrolled by the alleles of a single gene?b. What kinds of progeny would be produced if lavender F2 plants were allowed to self-fertilize?
Consider two maize plants:a. Genotype C/cm ; Ac/Ac+, where cm is an unstableallele caused by a Ds insertionb. Genotype C/cm, where cm is an unstable allele causedby Ac insertionWhat phenotypes would be produced and in whatproportions when (1) each plant is crossed with a basepair-substitution mutant c/c and (2) the plant in part a iscrossed with the plant in part b? Assume that Ac and care unlinked, that the chromosome-breakage frequencyis negligible, and that mutant c /C is Ac+.
One issue with interrupted-mating experimentssuch as that in Problem 19 is that gene order may beambiguous if the genes are close together. Anothershortcoming is that such experiments do not provide accurate map distances. The reason is that researchers select for the first Hfr marker transferred intothe recipient, but the recovery of F− exconjugantswith a later Hfr marker is complex, depending bothon transfer of the marker into the cell and on crossovers that transfer the marker into the recipientchromosome.To make more accurate maps, bacterial geneticistsoften do Hfr × F− crosses in a different way: Theyselect for exconjugants that contain a late Hfr marker,and then screen for the presence of the earlier markers.This method ensures that all of the markers haveentered the F− cell, so relative gene distances arenow accounted for solely by crossover frequencies.Furthermore, gene order is clarified by considering thecrossovers responsible for each class of exconjugants.As an example,…
The gene controlling ABO blood type and the gene underlying nail-patella syndrome are said to show linkage. What does that mean in terms of their relative locations in the genome? What does it mean in terms of how the two traits are inherited with respect to each other?
Familial retinoblastoma, a rare autosomal dominant defect, arose in a large family that had no prior history of the disease. Consider the following pedigree (the darkly colored symbols represent affected individuals): a. Circle the individual(s) in which the mutation most likely occurred. b. Is the person who is the source of the mutation affected by retinoblastoma? Justify your answer. c. Assuming that the mutant allele is fully penetrant, what is the chance that an affected individual will have an affected child?
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