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Elevated levels of NADH in the liver promote gluconeogenesis. I'm curious as to your thoughts on this claim.
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- Study Figure 19.18 and decide which of the following statements is false. Pyruvate dehydrogenase is inhibited by· NIADH. Pyruvate dehydrogenase is inhibited by AΤΡ. Citrate synthase is inhibited by NADH. Succinyl-CoA activates citrate synthase. Acetyl-CoA activates pyruvate carboxylase.Increased NADH concentrations in the liver encourage gluconeogenesis. What do you think about this assertion?Increased levels of NADH in the liver promote the process of gluconeogenesis. What do you make of this statement?
- In the liver, higher levels of NADH promote gluconeogenesis. I'm curious as to your thoughts on this claim.The production of glucose by the liver is facilitated by elevated amounts of the molecule NADH. What are your thoughts on the aforementioned assertion?Liver NADH increases gluconeogenesis. How do you interpret this?
- After several days of starvation, the capacity of the liver to metabolize acetyl-CoA via the citric acid cycle is greatly diminished. Explain.what effect would an increase in glucose typically have on the activity of glycogen synthasewhat is one physiological consequence that might occur if gluconeogenesis was the exact opposite of glycolysis? please help
