NADH boosts gluconeogenesis in the liver. So, what do you think about this assertion?
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NADH boosts gluconeogenesis in the liver. So, what do you think about this assertion?
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Solved in 3 steps
- Study Figure 19.18 and decide which of the following statements is false. Pyruvate dehydrogenase is inhibited by· NIADH. Pyruvate dehydrogenase is inhibited by AΤΡ. Citrate synthase is inhibited by NADH. Succinyl-CoA activates citrate synthase. Acetyl-CoA activates pyruvate carboxylase.Liver NADH increases gluconeogenesis. How do you interpret this?Increased levels of NADH in the liver promote the process of gluconeogenesis. What do you make of this statement?
- Can you explain the difference in the regulation of carbohydrate metabolism in liver and muscle?What is the effect of insulin on the committed step of glycolysis in the liver? Describe the mechanism by which insulin can affect the activity of this enzyme in the liver?Its principal function is to increase the concentration of glucose in the blood by speeding the glycogenolysis and gluconeogenesis in the liver.
- salvia does not contain amylase until babies are two months old. How could thu affect an infant's digestve requirements?Glucose can be made from oxaloacetate during gluconeogenesis, but if oxaloacetate concentrations are decreased,what other substance can be used to make glucose? How might this contribute to increased fat loss?Why does epinephrine stimulate glycolysis? I know why epinephrine stimulates glycogenolysis which is to release glucose stores. But epinephrine stimulating glycolysis seems counterintuitive as it would synthesize less glucose and more pyruvate?