Energy production pathway is targeted by drugs in the malignent cancerous cells to control an x cancer type. Use your speculation and tell targeting and destroying which one energy producing agent of the oxidative phosphorylation will be most effected in blocking most of the energy production and why?

Energy production pathway is targeted by drugs in the malignent cancerous cells to control an x cancer type. Use your speculation and tell targeting and destroying which one energy producing agent of the oxidative phosphorylation will be most effected in blocking most of the energy production and why?

Human Physiology: From Cells to Systems (MindTap Course List)

9th Edition

ISBN:9781285866932

Author:Lauralee Sherwood

Publisher:Lauralee Sherwood

Chapter2: Cell Physiology

Section: Chapter Questions

Problem 2TAHL

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Energy production pathway is targeted by drugs in the malignant (cancerous) cell to control an X cancer type. Use your speculation and tell targeting and destroying which one energy producing agent of the oxidative phosphorylation will be the most effective in blocking most of the energy production and why?
Energu production pathway is tartgeted bu drugs in melignant cells to control x cancer type. Use your sepculation and tell targeting and destroyin which one energy producing agent of oxydative phosphorylation will be most effective in blocking of the energy production and why?
Energy production pathway in targeted by drugs in the malignant (cancerous) cells to control an X cancer type. Use your speculation and tell targeting and destroying which one energy-producing agent of the oxidative phosphorylation will be most effective in blocking most of the energy and why?
Name what is asked: a. enzyme that is inhibited in the glycolytic pathway whenever the cell has ample ATP and is well supplied by other fuels such as fatty acidsb. resulting molecule(s) during the investment phase of glycolysisc. glycolytic substrate which is the precursor of the pentose phosphate pathway
Place the steps controling phosphofructokinase activity during glycolysis after ATP depletion. as: 1. 2. 3. 4. 5. 6. AMP binds to phosphofructokinase at an allosteric site. AMP levels rise. Phosphofructokinase changes from inactive to active 3D conformation. ATP levels in the cell rise. The rate-determining step of glycolysis proceeds at a lower activation energy. High level of metabolites from the citric acid cycle and oxidative phosophorylation cause feedback inhibition.
The enzyme Phosphofructokinase (PFK) is a key regulator of glycolysis. A) Does ATP bind the PFK allosteric siteor the active site? B) Does high amount of ATP in the cell activate or inhibit PFK? Why? C)What do you suppose happens to glucose if glycolysis is inhibited in the cell?
Glycolysis is often split up into three phases: 1. Energy Investment 2. Sugar Splitting and 3. Energy Payoff. During the Energy Investment Phase, 2 ATP are "spent" to generate Fructose 1,6 biphosphate and 2 ADP. What has happened to the terminal (third) phosphates in the 'spent" ATPs? A. They are lost in the cytoplasm B. They are transferred to the protein kinases C. They are transferred, one by one, to the glucose molecule by kinases D. They are lost as heat E. All of the above
In 2012, an Illinois man was killed by cyanide poisoning after he won a million dollars in the lottery. Cyanide is a lethal poison because it interferes with the electron transport chain in mitochondria. What effect would cyanide have on cellular respiration? (a) Glycolysis, the Krebs cycle, and oxidative phosphorylation would all be inhibited. (b) The Krebs cycle would be inhibited, but oxidative phosphorylation would not. (c) Oxidative phosphorylation would be inhibited. (d) Glycolysis, the Krebs cycle, and oxidative phosphorylation would all be stimulated.
a. Calculate how many carbons from an original glucose molecule will enter into the TCA cycle? (please provide your answer in number like 1, 2, 3) b. Based on the question you answered above what happens to the carbons that do not enter the cycle (write what it converts into)?
Which of the following statements about the malate-aspartate shuttle is false? a) 2.5 ATPs are formed per cytoplasmic NADH shuttled. b) Transamination is involved. c) Cytoplasmic oxaloacetate is converted to malate. d) Mitochondrial malate is converted to oxaloacetate. e) Antiport transport is not involved.
What effect will increasing the number of c subunits in ATP synthase have on the P:O ratio? A. It will increase for both NADH and FADH2. B. It will increase for NADH and decrease for FADH2. C. It will decrease for NADH and increase for FADH2. D. It will decrease for both NADH and FADH2. E. It will remain unchanged for both NADH and FADH2.
Kinases catalyze the transfer of a phosphate group from a phosphate donor such as adenosine triphosphate (ATP) to a substrate. A well‑known kinase is hexokinase. Hexokinase catalyzes the first step of the glycolysis cycle, and converts glucose to glucose‑6‑phosphate. The reaction of glucose with ATP is shown. The enzyme‑bound base is abbreviated as :B−, and ATP is abbreviated as a diphosphate bonded to adenosine monophosphate (AMP).