Explain the NF-KB pathway and the interaction between it and COX and interleukins in the inflammatory repsonse process in regards to adenocarcinoma tumours colon cancer.
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Explain the NF-KB pathway and the interaction between it and COX and interleukins in the inflammatory repsonse process in regards to adenocarcinoma tumours colon cancer.
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- Explain the overexpression of a protein that binds to the Fas ligand contributes to the survival of the lung and colon tumor cells.Explain the signifi cance of the observation that peptides such as fMet-Leu-Phe “activate” the phagocytotic (particle-engulfi ng) functions of mammalian leukocytes (white blood cells).compare and explain which genes from these from the CIN,MSI,CIMP pathways have the strongest links to colorectul cancer and which genes have the weakest. Give full explaions with thr Gene examples.
- During inflammaion, host tissue may be damaged owing to the release of toxic oxygen derivatives produced by activated phagocytes. List some of hese toxic species and explain what cellular/biochemical mechanisms limit these damaging off-target effects.reveiw the evidence for and againt the importance of inflammatory response genes of the CIN, MSI, CIMP pathways that are involved in regards to adenocarcinoma tumours colorectal cancer (colon) Provide detailed analysis with examples.THE CHROMOSOMAL INSTABILITY PATHWAYS IN COLON CANCER Provide exmaples of genes give evidence
- Explain the term hematopoietic growth factors (HGFs)?Describe the mutational event that produces the MYC oncogene in Burkitt’s lymphoma. Why does the particular mechanism for generating oncogenic MYC result in a lymphoma rather than another type of cancer? Describe another mechanism for generating oncogenic MYC.Describe the binding site for superantigens on T cells andAPCs. How does this relate to the activation of a much largerpopulation of T cells than normal upon exposure tosuperantigens?
- State whether the level of each of the following protein in a cancer cell is higher or lower than normal and how is that beneficial to the cancer cell? a. ARF protein b. MDM2 c. caspases d. Fas receptor e. TNF-αNeoplastic cells typically evade immunosurveillance. Explain how this occurs and describe how targeting tumour-mediated immune suppression has led to novel cancer immunotherapeuticsExplain the activation of the protein kinase, c-raf, in response to growth factor receptor activation
