Given these parents-note, they are NOT quite the same as each other!: mom = Aa Bb Cc Dd dad = Aa Bb cc Dd NOTE that the A gene acts incompletely dominantly. a) What chance does their baby having the "strongest phenotype" in A, being recessive in b, recessive in c, and dominant in D? Give one fraction. Show work. Cc. Now what is the chance of having that same b) phenotype? Now assume mom was CC instead

Given these parents-note, they are NOT quite the same as each other!: mom = Aa Bb Cc Dd dad = Aa Bb cc Dd NOTE that the A gene acts incompletely dominantly. a) What chance does their baby having the "strongest phenotype" in A, being recessive in b, recessive in c, and dominant in D? Give one fraction. Show work. Cc. Now what is the chance of having that same b) phenotype? Now assume mom was CC instead

Human Heredity: Principles and Issues (MindTap Course List)

11th Edition

ISBN:9781305251052

Author:Michael Cummings

Publisher:Michael Cummings

Chapter5: The Inheritance Of Complex Traits

Section: Chapter Questions

Problem 5QP: As it turned out, one of the tallest Potsdam Guards had an unquenchable attraction to short women....

See similar textbooks

Similar questions

As it turned out, one of the tallest Potsdam Guards had an unquenchable attraction to short women. During his tenure as guard, he had numerous clandestine affairs. In each case, children resulted. Subsequently, some of the childrenwho had no way of knowing that they were relatedmarried and had children of their own. Assume that two pairs of genes determine height. The genotype of the 7-foot-tall Potsdam Guard was A9A9B9B9, and the genotype of all of his 5-foot clandestine lovers was AABB. An A9 or B9 allele in the offspring each adds 6 inches to the base height of 5 feet conferred by the AABB genotype. a. What were the genotypes and phenotypes of all the F1 children? b. Diagram the cross between the F1 offspring, and give all possible genotypes and phenotypes of the F2 progeny
For a recessive condition, two normal heterozygous individuals have children. What is the likelihood of their children being affected by this condition? What is the likelihood of their children being carriers without the condition? What is the likelihood of their asymptomatic children being carriers? Suppose that an individual with the condition has children with a heterozygous individual, what is the likelihood of their children being carriers?
How does a positive ASO test for sickle-cell anemia determine that an individual is homozygous recessive for the mutation that causes sickle-cell anemia?
This pedigree traces the inheritance of a trait in humans. Based on this pedigree, is the allele for this disease dominant or recessive? Is it sex-linked? Explain. What genotypes are most probable for the individuals labeled 4, 7, and 10?
Phenylketonuria (PKU) is a disease that results from a recessive gene.Suppose that two unaffected parents produce a child with PKU. a. What is the probability that a sperm from the father will contain the PKU allele?b. What is the probability that an egg from the mother will contain the PKU allele?c. What is the probability that their next child will have PKU?d. What is the probability that their next child will be heterozygous for the PKU gene?
Phenylketonuria (PKU) is a disease that results from a recessive gene. Suppose that two unaffected parents produce a child with PKU. a. What is the probability that a sperm from the father will contain the PKU allele? b. What is the probability that an egg from the mother will contain the PKU allele? c. What is the probability that their next child will have PKU? d. What is the probability that their next child will be heterozygous for the PKU gene?
In humans, the genetic disease cystic fibrosis is caused by a recessive allele (a). The normal (healthy) allele is dominant (A). What is the genotype of someone who has cystic fibrosis? What are the two different genotypes that a healthy person could have? If two people were both heterozygous for the cystic fibrosis gene, what fraction of their children would be likely to have this disease? Hint: Draw a Punnett square to figure it out.
In a paternity suit, a woman with type O blood claims that a man with type AB blood is the father of her type O baby. List the genetypes of the mom, man, and baby. Can the woman prove he is the father or can he is not? Why or why not? If the accused man was type A insyead of type AB, what would be his possible genotypes? Could the woman prove he is the father? Can the man prove he is not the father? Why or why not?
Duchenne muscular dystrophy is sex linked and usuallyaffects only males. Victims of the disease become progressively weaker, starting early in life.a. What is the probability that a woman whose brotherhas Duchenne’s disease will have an affected child?b. If your mother’s brother (your uncle) had Duchenne’sdisease, what is the probability that you have receivedthe allele?c. If your father’s brother had the disease, what is theprobability that you have received the allele?
What the grandparents' genotypes are? Why doesn’t the father (II-1) have the disease breast cancer? What is the formal name for an individual having the gene but not showing the trait? For this particular family, what is the recombination rate between the D17S74 marker and the breast cancer gene?
In rabbits grey hair is dominant to white & black eyes are dominant to red. -Cross 2 parents who are both heterozygous for both traits. 1st Question to answer ---- How many of the offspring could have grey hair & black eyes? 2nd Question to answer ---- How many of the offspring could have grey hair & red eyes? 3rd Question to answer ---- How many of the offspring could have white hair & black eyes? 4th Question to answer ---- How many of the offspring could have white hair & red eyes?
Which of the following inheritance patterns was observed, for the T2 bacteriophages of Escherichia coli, in the experiments performed by Alfred Hershey and Martha Chase? A. neither 35S-labeled proteins nor 32P-labeled DNA molecules were passed on to viral progeny B. 35S-labeled DNA molecules were passed on to viral progeny, but not 32P-labeled proteins C. both 32P-labeled DNA molecules and 35S-labeled proteins were passed on to viral progeny D. 32P-labeled DNA molecules were passed on to viral progeny, but not 35S-labeled proteins E. both 35S-labeled DNA molecules and 32P-labeled proteins were passed on to viral progeny