How do nonsteroidal antiinflammatory drugs (NSAIDS), such as aspirin reduce inflammation ? O They inhibit the conversion of prostagladins into molecules that cause inflammation O They block the formation of prostaglandins. O They inhibit the release of prostaglandins O They inhibit the action of prostaglandins. O They inhibit the activation of prostaglandins.

How do nonsteroidal antiinflammatory drugs (NSAIDS), such as aspirin reduce inflammation ? O They inhibit the conversion of prostagladins into molecules that cause inflammation O They block the formation of prostaglandins. O They inhibit the release of prostaglandins O They inhibit the action of prostaglandins. O They inhibit the activation of prostaglandins.

Ebk:Nutrition & Diet Therapy

10th Edition

ISBN:9780357391747

Author:DEBRUYNE

Publisher:DEBRUYNE

Chapter24: Energy- And Protein- Modified Diets For Metabolic And Respiratory Stress

Section: Chapter Questions

Problem 3SC

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A.Where are Epinephrine and Norepinephrine made? (cells, organ) B.What organ/or cells secrete them? (may differ from site of formation) C.What stimulates their secretion? Explain negative feedback control of secretion. D.Where do they work (target) E.What do they regulate: physiological outcome?
Which of the following statementsabout hormones and nerve impulses isaccurate?O A. Hormones and nerve impulses bothcarry messages throughout the body, buthormones work more slowly.O B. Hormones and nerve impulses bothcarry messages in the body, but hormonesonly work on cells very near where thehormones are made.O C. Hormones and nerve impulses bothcarry messages in the body using electricalsignals.O D. Hormones and nerve impulses bothcarry messages throughout the body, buthormones work more quickly
Down-regulationa. produces a decrease in the number of receptors in the target cells.b. produces an increase in target cells’ sensitivity to a hormone.c. is found in target cells that respond to hormones that are maintainedat constant levels.d. occurs partly because of an increase in receptor synthesis by thetarget cell.e. All of these are correct.
which of the following happens when a therapeutic synthetic homone is introduced intno the body to treat patients natural hormone insufficiency?(select all that apply) A. The target cell responds the same way as when the hormone is natural B. the synthetic hormone has no effect on the target tissues C. the amount of natural hormone produced by the patients body declines D. the synthetic hormone is destroyed before it can have an effect on the target tissue.
Women who have been blind since birth almost never have breast cancer. A high level of one specific hormone is though to contribute to their low cancer rate. Which hormone is it and why are levels of this hormone unusually high in the blind?
A hormone differs from a neurotransmitter in thata. hormones act extracellularly, whereas neurotransmitters act withinthe cell that synthesized them.b. hormones are released only by neurons, whereas neurotransmittersare released by many different types of cells.c. hormones cause only fast responses (seconds or less) to stimuli,whereas neurotransmitters cause slow responses (minutes to hours)to stimuli.d. hormones affect only epithelial cells, whereas neurotransmittersaffect only muscle cells.e. hormones are released into the bloodstream and can activate manycells in many parts of the body, whereas neurotransmitters arereleased by neurons and affect adjacent cells.
You are designing neurotransmitters and hormones. 1. Which should have a higher affinity for their receptors? 2. Which should act more rapidly?
Given these events:(1) The α subunit of a G protein interacts with Ca2+channels.(2) Calcium ions diffuse into the cell.(3) The α subunit of a G protein is activated.Choose the arrangement that lists the events in the order they occurafter a hormone binds to a receptor on a smooth muscle cell.a. 1,2,3 c. 2,1,3 e. 3,2,1b. 1,3,2 d. 3,1,2
Endorphins are substance P receptors. fit into opiate receptors. are natural analogues of cannabinoids. counter the effects of opiates. increase the production of substance P. None of these responses is correct. 0
How does the binding of a hormone to a receptor activate a G-protein? a. It replaces bound GDP with GTP. b. It’s magic. c. It replaces bound GTP for GDP. d. A and B. e. None of the above.
The drug Celebrex selectively inhibits PTGS2 while aspirin and other NSAID’s inhibit both PTGS1 and PTGS2 in the same way. Why do you think researchers wanted to discover a selective inhibitor to PTGS2?
Suppose that the circulating concentration of hor-mone is 10–10 M and the Kd for binding to its receptor is 10–8M. What fraction of the receptors will have hormone bound?If a meaningful physiological response occurs when 50% ofthe receptors have bound a hormone molecule, how muchwill the concentration of hormone have to rise to elicit aresponse? The fraction of receptors (R) bound to hormone(H) to form a receptor–hormone complex (R–H) is [R–H]/([R] + [R–H]) = [R–H]/[R]TOT = [H]/([H] + Kd).