In the past, it was believed that lactic acid accumulated in the muscles of tracking athletes as a result of intense exertion. For this reason, coaches would have athletes slowly jog to move more oxygen into the muscles so lactic acid would be oxidized and there would be less soreness the next day. Based on current research, what is the most likely consequence of lactate production in the athlete’s performance? What is more likely to be the cause of muscle soreness?

In the past, it was believed that lactic acid accumulated in the muscles of tracking athletes as a result of intense exertion. For this reason, coaches would have athletes slowly jog to move more oxygen into the muscles so lactic acid would be oxidized and there would be less soreness the next day. Based on current research, what is the most likely consequence of lactate production in the athlete’s performance? What is more likely to be the cause of muscle soreness?

Human Physiology: From Cells to Systems (MindTap Course List)

9th Edition

ISBN:9781285866932

Author:Lauralee Sherwood

Publisher:Lauralee Sherwood

Chapter2: Cell Physiology

Section: Chapter Questions

Problem 4TAHL: Why do you think a person is able to perform anaerobic exercise (such as lifting and holding a heavy...

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Why do you think a person is able to perform anaerobic exercise (such as lifting and holding a heavy weight) only briefly but can sustain aerobic exercise (such as walking or swimming) for long periods? (Hint: Muscles have limited energy stores.)
ATP for muscle contraction can be provided by ______. a. aerobic respiration b. lactate fermentation c. transfer of a phosphate from creatine phosphate d. all of the above
The filaments of skeletal muscle are moved by free energy derived from the hydrolysis of ATP. During sustained muscle contraction ATP levels remain fairly constant, while levels of creatine phosphate fall. Explain why this is so. Under what circumstances is an oxygen debt incurred during muscle contraction?
Under what conditions will lactic acid accumulate in skeletal muscle? A. when citric acid enzymes are depleted B. when ATP is depleted C. when O2 is depleted D. when NADH is depleted
Which of the following is NOT a stock form of the key molecules for energy generation in muscle? A. Creatine phosphate B. Hemoglobin C. Glycogen D. Myoglobin
What would be the ATP yield per molecule of glucose in the muscle if glycogen were the source of the glucose?
Muscle fatigue occurs because of.. A relative deficit of ATP Excessive accumulation of lactic acid Ionic imbalances Depletion of nutient stores All of the above
Which of the following is the correct summary of the anaerobic breakdown of glucose in the skeletal muscle? Glucose + ADP + Pi ⟶ lactate + ATP Glucose + 2ADP + 2Pi ⟶ 2 lactate + 2ATP Glucose + 2ADP + 2Pi + 2NAD+ ⟶ 2 pyruvate + 2ATP + 2NADH + 2H+ Glucose + ADP + Pi ⟶ pyruvate + ATP Glucose + 2ADP + 2Pi ⟶ 2 pyruvate + 2ATP
Since ancient times it has been observed that certain game birds, such as grouse, quail, and pheasants, are easily fatigued. The Greek historian Xenophon wrote: “The bustards . . . can be caught if one is quick in starting them up, for they will fly only a short distance, like partridges, and soon tire; and their flesh is delicious.” The flight muscles of game birds rely almost entirely on the use of glucose 1-phosphate for energy, in the form of ATP . The glucose 1-phosphate is formed by the breakdown of stored muscle glycogen, catalyzed by the enzyme glycogen phosphorylase. The rate of ATP production is limited by the rate at which glycogen can be broken down. During a “panic flight,” the game bird’s rate ofglycogen breakdown is quite high, approximately 120 mmol/min of glucose 1-phosphate produced per gram of fresh tissue. Given that the flight muscles usually contain about 0.35% glycogen by weight, calculate how long a game bird can fly. (Assume the average molecular weight of a…
Impairment of intracellular calcium homeostasis in muscle fibers lead to chronic, severe muscle dysfunction. Impaired Calcium ATPase activity can lead to abnormal, persistent elevation of intracellular calcium concentrations. Explain how persistently high intracellular calcium concentration would affect cross-bridge formation, and muscle contractions, and muscle tension? (Mention the effects on the steps involved cross bridge formation, and muscle contraction/relaxation cycle, and overall muscle tension).
If you were able to control fatigue in the muscle cell experimentally such that you only exposed the muscle to high levels of Pi and no other metabolites, what would you observe? A decline in force without much change in velocity A decline in velocity without much change in force A muscle operating below Lo A decline in both force and velocity An increased affinity of Ca2+ for troponin
An athlete participates in aerobic exercise three times a week. One of the changes is an increased ability to deliver oxygen to her skeletal muscles. Over time she notices that she can continue the exercise with greater intensity and duration. The reason for this change is that there is a(n) a. greater response from phosphate transfer. b. greater production of ATP from glycolysis and less from aerobic cellular respiration. c. greater production of ATP from aerobic cellular respiration. d. increased production of lactate.