is the number of moles of ATP produced by 5 moles of glucose in Krebs cycle is the number of moles of CO2 produced by 4 moles of glucose in Krebs cycle is the number of moles of GTP produced by 5 moles of glucose in Krebs cycle is the classification of the enzyme that converts oxalosuccinate to alpha-ketoglutarate is the classification of the enzyme that converts dihydroxyacetone phosphate to glyceraldehyde-3-phosphate
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- dtermoine numberof ATPS GENERATED FROM COMPLETE oxidation of fructose-6-phosphate isocitrate stearidonic acid [18 carbons triangle 6,9,12,15] indicate where everything comes from ex ATPS FROM GLYCOLYSIS , NADH FROM TCA ETC LIST ALL1. The enzyme that catalyzes the transformation of C2H2 + H2 to C2H4 is likely to be classified as2. The enzyme that catalyzes the conversion of glucose + ATP to glucose-6-phosphate + ADP is likely to be classified asa. Isomeraseb. Ligasec. Transferased. Lyasee. Hydrolasef. OxidoreductaseHow many ATP may be produced from 1 mole of the following compounds on complete oxidation using the glycerol-3-phosphate shuttle? COMPOUNDSa.) Mannoseb.) Dihydroxyacetone phosphatec.) Citrated.) Malatee.) Succinate
- Which of the following glycolytic enzymes neither produces nor consumes ATP? A. phosphofructokinaseB. triose phosphate isomeraseC. pyruvate kinaseD. phosphoglycerate kinaseE. hexokinaseLeigh syndrome is characterized by psychomotor regression: that is, the progressive loss of mental andmovement abilities. Patients also suffer from lacticacidosis, a condition in which mitochondrial respiration is deficient, so their tissues metabolize glucoseanaerobically, leading to the buildup of lactate. Somepatients with Leigh syndrome have a mutation in themitochondrial gene MT-CO3, which encodes a subunit of the electron transport complex cytochromec oxidase. Other patients diagnosed with Leigh syndrome have a loss-of-function mutation in the nucleargene SURF1, which encodes a factor needed for theassembly of this same enzyme complex.a. How can the same symptoms result from mutationsin a mitochondrial gene and from mutations in anuclear gene?Regarding phosphofructokinase, which of the following statements is true: a. Low [ATP] stimulates the enzyme, but fructose-2,6-bisphosphate inhibits. b. High [ATP] stimulates the enzyme, but fructose-2,6-bisphosphate inhibits. c. The enzyme is more active at low [ATP] than at high, and fructose-2,6-bisphosphate activates the enzyme. d. High [ATP] stimulates the enzyme, and fructose-2,6-bisphosphate activates.
- If decanoic acid (10 : 0) undergoes complete β-oxidation in mitochondria, how many net ATP is yielded? (NADH = 2.5 ATP; FADH2 = 1.5 ATP; Acetyl CoA = 10 ATP) Answer in number onlyDefine the following terms:a. amphibolic pathwayb. anaplerotic reactionc. glyoxylate cycled. reduction potentiale. conjugate redox pairCan you please match the option that is the best fit for each word? Biotin Bicarbonate and ATP Oxaloacetate Malate transport Cori Cycle a. Reactants for adding carboxy group to biotin b. Mechanism for "moving" NADH equivalents from mitochondrial matrix to cytosol c. 4 carbon acid converted to 3 carbon, phosphoenolpyruvate with release of CO2 d. A mechanism to convert lactate produced in muscle to glucose in the liver, which is then transported into circulation e. Cofactor for pyruvate carboxylase
- ADP ATP NADP NADPH FAD FADH2 Oxidation Reduction chemiosmosis acetyl coenzyme >give the meaning of the acronyms >define eachWhat do you think would be the effect on oxidative phosphorylation of the following mutations of F1 or F0 ATPase subunits? Be as specific as possible. A. Subunit c Asp61 to Leu B. Subunit c Asp61 to Asn C. Subunit a Asn214 to Asp D. Subunit a Ser206 to AlaDescribe the function implied by the name of each of thefollowing enzymes:a. Citrate decarboxylaseb. Adenosine diphosphate phosphorylasec. Oxalate reductased. Nitrite oxidasee. cis-trans Isomerase