Now, examine the figure below. Highly Variable Plasticity, strong Genotype-by- Environment Interaction No Plasticity Plasticity Trait Trait Trait
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Phenotypic plasticity refers to the mechanism of change in the phenotype of the organism with respect to the environment.
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- Hi! Can you give an explanation for each sample identity in paragraph form (for the rationale)? Thank you.Explain why the effect of a genotype on a phenotypecannot always be determined without knowing whatthe environment is, and why the effect of a particularenvironment on a phenotype cannot always be determinedwithout knowing what the genotype is.In a sex linked recessive disease, the frequency of the causative allele is 0.4. What is the genotype frequency among males? In addition, what is the genotype frequency of heterozygous female population?
- Among the most prevalent pathologies that afflicthuman beings is heart disease, which can have a severeimpact on quality of life and can even result in prematuredeath. While heart disease mostly afflicts those who areolder, 1% or 2% of people in their 30s, and even in their20s, suffer from this disease. Genetic and environmentalcomponents of this disease exist. What strategy mightyou use to choose families to participate in a GWAS ofheart disease–causing genes? Explain your reasoning.Where's the 5 examples?5.) A man and a woman living in a tropical area where malaria is prevalent and health care is not accessible have seven children during their lifetime. The genotypes of their children are: ss, Ss, SS, ss, Ss, Ss, and SS. What must the genotypes of both parents be? Your answer should include a Punnett square to illustrate your work, and list all the genotypes and phenotypes.
- 900 bacteria had two recessive alleles out of a total of every 10,000 bacteria. Using the Hardy-Weinberg equation(s), determine the frequency of the recessive and dominant alleles.APOE Allelic frequency Control EOAD LOAD Vascular disease e4 0.07 0.28 0.35 0.12 e3 0.88 0.70 0.63 0.87 What group is used as a control here? Based on this data, do you think APOE e4 allele increases one’s chance to develop EOAD or LOAD? Defend your answer. Your answer has to refer to the specific data in the table. Suppose that you want to improve this case control study by controlling one more variable that is a known risk factor for Alzheimer’s disease, how will you design your groups? Specify the risk factor you want to control and how you plan to control it.The following are three possible combinations of DNAcharacteristics that may be found in an individual’sgenetic profile. Using the product rule, rank each ofthese combinations of DNA characteristics from mostcommon to least common. The number after each characteristic indicates its percentage distribution in thepopulation.a. FGA 24,24 (3.6%), TH01 6,8 (8.1%), and D16S53911,12 (8.9%)b. vWA 14,19 (6.2%), D21S11 30,30 (3.9%), andD13S317 12,12 (8.5%)c. CSF1PO 9,10 (11.2%), D18S51 14,17 (2.8%), andD8S1179 17,18 (6.7%)
- The frequency of a recessive disease is 9%. What is the frequency of the allele that causes the disease? What is the frequency of the dominant allele? What is the frequency of the carriers (heterozygotes) of the disease?How do I calculate the ST50 from my raw data to give me time at which 50% sedation has occurred graphically of all of my replicates? Using excel do I use linear graphs? scatterplot? what kind of trendline? with what setting? Raw data includes time from 0-24 hours + # of flies sedated at different intervals. Enin a village where hte proportion of individuals who are susceptible to malaria (genotype hbA/hbA) is 0.53 and hte population is asssumed to be at hardy weingberg equalibrium what is the expected freqency of the HbS alle? hint what is the genotype of people with sickle cell disease and how is that genotype represneted in the hardy weinberg equation