[ Select ] H. What does the absence of glucose do to the bacterial cell v it inhibits the trp operon it inhibits the lac operon I. You now add tryptophan to the cell. What would happen in accelerates the lac operon operon? the trp operon is inhibited it accelerates the trp operon it has no effect I What kind of regulation does lactose provide to the repre

[ Select ] H. What does the absence of glucose do to the bacterial cell v it inhibits the trp operon it inhibits the lac operon I. You now add tryptophan to the cell. What would happen in accelerates the lac operon operon? the trp operon is inhibited it accelerates the trp operon it has no effect I What kind of regulation does lactose provide to the repre

Biology (MindTap Course List)

11th Edition

ISBN:9781337392938

Author:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Publisher:Eldra Solomon, Charles Martin, Diana W. Martin, Linda R. Berg

Chapter14: Gene Regulation

Section: Chapter Questions

Problem 13TYU: INTERPRET DATA Develop a simple hypothesis that would explain the behavior of each of the following...

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Plz answer ASAP. I will thumb up You are studying the regulation of the lactose operon in Escherichia coli, by measuring expression of the lacZ gene (i.e production of beta-galactosidase).(a) You identify several loss-of-function mutations in which lacZ is never expressed, in the presence and absence of glucose and lactose. What components of the lac operon could be mutated to produce this phenotype? List all possibilities.
. In an effort to determine the location of an operator sitefor a negatively regulated gene, you have made a seriesof deletions within the regulatory region. The extent ofeach deletion is shown by the line underneath the sequence, and the resulting expression from the operon(i = inducible; c = constitutive; − = no expression) isalso indicated.... GGAT C T T AGCCGGCTAACATGATAAATATAA......C C T AGAATCGGCCGA TTGTA C T A TTT ATAT T ...1 i2 –3 c4 –5 ca. What can you conclude from these data about thelocation of the operator site?b. Why do you think deletions 2 and 4 show no expression of the gene?
Create a metabolism cocept map using these terms RepressorInducer RiboswitchRepression InductionPositive regulationActivatorOperator Heat shocklac operontrp operon Catabolite RepressionHeat shocksigma-32 Negative regulationTwo-component regulationSensor Kinase Autoinducing Peptide (AIP)Response regulatorSigma factorsFeedback inhibitionProtein StabilityQuorum SensingRNA regulationAntisense RNAHomoserine Lactone (AHL)
1e regulation of the lac operon by cAMP-CAP complex is an example of positive regulation. what in general does it mean to say that a gene or operon is subject to positive regulation?
INTERPRET DATA Develop a simple hypothesis that would explain the behavior of each of the following types of mutants in E. coli. Mutant a: The map position of this mutation is in the trp operon. The mutant cells are constitutive; that is, they produce all the enzymes coded for by the trp operon, even if large amounts of tryptophan are present in the growth medium. Mutant b: The map position of this mutation is in the trp operon. The mutant cells do not produce any enzymes coded for by the trp operon under any conditions. Mutant c: The map position of this mutation is some distance from the trp operon. The mutant cells are constitutive; that is, they produce all the enzymes coded for by the trp operon, even if the growth medium contains large amounts of tryptophan.
The a/a operon is an inducible operon that controls the production of the sugar arabinose. When arabinose is present in a bacterium it binds to the protein AraC, and the complex binds to the initiator site to promote transcription. In this scenario, AraC is a(n). activator inducer repressor operator
For the E. coli lac operon, when lactose is present: a. and glucose is absent, cAMP binds and activates catabolicactivator protein (CAP). b. and glucose is absent, the level of cAMP decreases. c. activated CAP binds the repressor protein to remove it from the operator gene. d. the cell prefers lactose over glucose. e. RNA polymerase cannot bind to the promoter.
The activator (V protein) that is present in the bacterial cell is normally phosphorylated, but only binds to the A site in the operon when it is not phosphorylated. What operon-independent mechanism can regulate the binding of (and also ultimately regulates the removal of) protein V to the A site? Describe this binding/removal mechanism in detail – useappropriateterminologythatwasdiscussedinlecture. Aspartofyouransweraboutthisactivatorproteinbindingto (and then detaching from) the A site of the operon, be sure to address what are the results of this type of control in terms of function of the operon.
Attenuator control of the Trp operon what is the correct option from the choices below? involves a second repressor protein. would also work in eukaryotic cells. requires a short leader peptide gene. is independent of the Trp concentration. regulates transcriptional initiation of the operon.
Metabolic control is crucial part of survival for all bacteria. The lactose operon is located on the bacteria chromosome and assists in maintaining homeostasis. (l) Describe how an operon regulates the expression of genes. (m) Feedback plays an important part in life helping to maintain homeostasis. Explain how both negative and positive feedback help maintain homeostasis
. Recall that the trp operon has a special leader sequence (trpL) between the operator and the structural genes that offers attenuation as a mechanism for regulation of gene expression. (A) Draw a diagram of a trpL region of the operon when tryptophan is abundant in the cell.Label the following features: the DNA, 5’ and 3’ polarity of the RNA, the regions 1, 2, 3,and 4 and poly-U of the RNA, the pair of Trp codons (UGG), the ribosome, and RNA-Pol,along with any stem-loop structure that would form under these conditions (B) In the above example, will the rest of the trp operon genes be expressed? Briefly describe your reasoning why or why not (C) The trp codons in region 1 of the trpL gene have mutated to cysteines (UGG to UGC). What will be the effect on attenuation gene regulation of the trp operon? Brieflyexplain your reasoning.
Please discuss positive regulation with respect to lac operon.