Suppose a patient has correctly diagnosed GALT (galactose-1-phosphate uridylyltransferase) deficiency. Would this patient have lower steady state levels of NADPH in the presence of normal steady state glucose levels? Why is NADPH an important byproduct of the pentose phosphate pathway anyways?
Suppose a patient has correctly diagnosed GALT (galactose-1-phosphate uridylyltransferase) deficiency. Would this patient have lower steady state levels of NADPH in the presence of normal steady state glucose levels? Why is NADPH an important byproduct of the pentose phosphate pathway anyways?
Biochemistry
6th Edition
ISBN:9781305577206
Author:Reginald H. Garrett, Charles M. Grisham
Publisher:Reginald H. Garrett, Charles M. Grisham
Chapter19: The Tricarboxylic Acid Cycle
Section: Chapter Questions
Problem 21P: Complete oxidation of a 16-carbon fatty acid can yield 129 molecules of ATP Study Figure 19.2 and...
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Suppose a patient has correctly diagnosed GALT (galactose-1-phosphate uridylyltransferase) deficiency. Would this patient have lower steady state levels of NADPH in the presence of normal steady state glucose levels? Why is NADPH an important byproduct of the pentose phosphate pathway anyways?
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