Suppose that a time machine allows you to go back in time (and to travel to Australia), and you have collected rabbits and Myxoma from the years 1955 and 1980. Once you are back in the twenty-first century, you infect both sets of rabbits with both sets of Myxoma viruses. The most likely result will be (1) that the rabbits from 1955 injected with the 1955 virus will have a survival rate than rabbits from 1980 injected with the 1980 virus. survival rate than those same rabbits injected with the 1980 virus; and (2) that the rabbits from 1955 injected with the 1980 virus will have a O A. lower; lower O B. lower; higher C. higher; lower D. higher; higher E. the survival rates will be the same in experiments (1) and (2)

Suppose that a time machine allows you to go back in time (and to travel to Australia), and you have collected rabbits and Myxoma from the years 1955 and 1980. Once you are back in the twenty-first century, you infect both sets of rabbits with both sets of Myxoma viruses. The most likely result will be (1) that the rabbits from 1955 injected with the 1955 virus will have a survival rate than rabbits from 1980 injected with the 1980 virus. survival rate than those same rabbits injected with the 1980 virus; and (2) that the rabbits from 1955 injected with the 1980 virus will have a O A. lower; lower O B. lower; higher C. higher; lower D. higher; higher E. the survival rates will be the same in experiments (1) and (2)

Biology Today and Tomorrow without Physiology (MindTap Course List)

5th Edition

ISBN:9781305117396

Author:Cecie Starr, Christine Evers, Lisa Starr

Publisher:Cecie Starr, Christine Evers, Lisa Starr

Chapter10: Biotechnology

Section: Chapter Questions

Problem 2CT

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In 1918, an influenza pandemic that originated with avian flu killed 50 million people. Researchers isolated samples of that virus from bodies of infected people preserved in Alaskan permafrost since 1918. From the samples, they sequenced the viral genome, then reconstructed the virus. The reconstructed virus is 39,000 times more infectious than modern influenza strains, and 100 percent lethal in mice. Understanding how this virus works can help us defend ourselves against other deadly influenza strains that arise. For example, discovering what makes it so infectious would help researchers design more effective vaccines. Critics of the research are concerned: If the virus escapes the containment facilities (even though it has not done so yet), it might cause another pandemic. Worse, terrorists could use the published DNA sequence and methods to make the virus for horrific purposes. Do you think this research makes us more or less safe?
Why do scientists worry more about new strains of viruses like influenza and coronviruses as potential causes of deadly human pandemics, but they never talk about the next big Herpesvirus strain deadly pandemic? Which of the statements below helps partially answer that? Influenza and CoV are RNA viruses which results in having higher mutation rates -- they can more easily jump between host species. Herpesviruses are dsDNA viruses and usually very host specific. Only humans get Herpesviruses, while animals can get Influenza viruses and Coronaviruses. dsDNA viruses aren't very transmissible and even when they are, they're not deadly. We already have effective vaccines for Herpes. Influenza and CoV have more genes, more complex gene expression, and can undergo genetic recombination or reassortment, while Herpesviruses are more limited.
two directly trasmitted viral diseases have the same basic reproduction number (R0 - R not) even though their dynamics are different. Which of the following differences between the two diseases could lead to the same basic reproduction number? a. one disease has a higher transmission rate and a longer recovery period than the other b. one disease has a lower transmission rate and a longer recovery period than the other c. one disease has a lower transmission rate and a shorter recovery period than the other d. for one disease, the recovert period is long; for the other, no one recovers
There have been recurring cases of mad-cow disease in the United Kingdom since the mid-1990s. Mad-cow disease is caused by a prion, an infectious particle that consists only of protein. In 1986, the media began reporting that cows all over England were dying from a mysterious disease. Initially, there was little interest in determining whether humans could be affected. For 10 years, the British government maintained that this unusual disease could not be transmitted to humans. However, in March 1996, the government did an about-face and announced that bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, can be transmitted to humans, where it is known as variant Creutzfeldt-Jakob disease (vCJD). As in cows, this disease eats away at the nervous system, destroying the brain and essentially turning it into a spongelike structure filled with holes. Victims experience dementia; confusion; loss of speech, sight, and hearing; convulsions; coma; and finally death. Prion diseases are always fatal, and there is no treatment. Precautionary measures taken in Britain to prevent this disease in humans may have begun too late. Many of the victims contracted it over a decade earlier, when the BSE epidemic began, and the incubation period is long (vCJD has an incubation period of 10 to 40 years). A recent study concluded that 1 in 2,000 people in Great Britain carry the abnormally folded protein that causes vCJD. In spite of these numbers, the death rate from vCJD remains low. It is not clear whether this means that the incubation period for the disease is much longer than previously thought, or whether they may never develop the disease. If you were traveling in Europe, would you eat beef? Give sound reasons why or why not.
There have been recurring cases of mad-cow disease in the United Kingdom since the mid-1990s. Mad-cow disease is caused by a prion, an infectious particle that consists only of protein. In 1986, the media began reporting that cows all over England were dying from a mysterious disease. Initially, there was little interest in determining whether humans could be affected. For 10 years, the British government maintained that this unusual disease could not be transmitted to humans. However, in March 1996, the government did an about-face and announced that bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, can be transmitted to humans, where it is known as variant Creutzfeldt-Jakob disease (vCJD). As in cows, this disease eats away at the nervous system, destroying the brain and essentially turning it into a spongelike structure filled with holes. Victims experience dementia; confusion; loss of speech, sight, and hearing; convulsions; coma; and finally death. Prion diseases are always fatal, and there is no treatment. Precautionary measures taken in Britain to prevent this disease in humans may have begun too late. Many of the victims contracted it over a decade earlier, when the BSE epidemic began, and the incubation period is long (vCJD has an incubation period of 10 to 40 years). A recent study concluded that 1 in 2,000 people in Great Britain carry the abnormally folded protein that causes vCJD. In spite of these numbers, the death rate from vCJD remains low. It is not clear whether this means that the incubation period for the disease is much longer than previously thought, or whether they may never develop the disease. What measures have been taken to stop BSE?
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In 2006, the US Food and Drug Administration (FDA) approved the use of bacteriophage preparation that can be used on ready-to-eat meat and poultry products to kill Listeria monocytogenes, a bacterium that causes listeriosis. Some people may have concerns about the use of phages in foods. From what you have learned about bacteriophages and viruses, why do you think we do not have to be concerned about the presence of bacteriophages in food?
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