Which is NOT a reason why the fruit flies make goad model organistrs? simple food requirements they're found atl over the workd O only a 2 week life cycle O can keep milions in a roon The wild type phenotype is always what is dominant. True False What would we use to anesthetize our flies if we were vworking in the real lab? Propofol O Xenon FlyNap Alfentanil
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- Which statements are true? Explain why or why not.1 In the small intestine, stem cells in the cryptsdivide asymmetrically to maintain the population of cellsthat make up the villi; after each division, one daughterremains a stem cell and the other begins to divide rapidlyto produce differentiated progeny.2 Stem cells, being stem cells, are by definition thesame in all tissues.3 Every tissue that can be renewed is renewed froma tissue-specific population of stem cells.4 Disturbance of the balance in the activities ofosteoblasts and osteoclasts in favor of osteoclasts cangive rise to the condition known as osteoporosis, the brit-tle-bone syndrome of the elderly.During the study of your genetics course, you are learning the different developmental stages of a unique Tsetse fly, named defsis-timen, and you discover differences in their developmental pattern i.e. unusual and normal...... Rest of the question is in picture Answer part b and c And in part c its unlinked genes not unlinedWhen Thomas Hunt Morgan crossed his red-eyed F1 generation flies to each other, the F2 generation includedboth red- and white-eyed flies. Remarkably, all the white-eyed flies were male. What was the explanation forthis result? a. The gene involved is on the X chromosomeb. The gene involved is on the Y chromosomec. The gene involved is on an autosome, but only in malesd. Other male-specific factors influence eye color in fruit fliese. None of these is correct
- A zookeeper has collected a male and a female lizard that look likethey belong to the same species. They mate with each other andproduce phenotypically normal offspring. However, the offspringare sterile. Suggest one or more explanations for their sterility.Two adjacent cells in the nematode worm nor-mally differentiate into an anchor cell (AC) and a ventraluterine precursor (VU) cell, but which of the two becomesthe AC and which becomes the VU cell is completely ran-dom: the cells have an equal chance of adopting eitherfate, but they always adopt different fates. Mutations ofLin12 alter these fates. In hyperactive Lin12 mutants, bothcells become VU cells, while in inactive Lin12 mutants,both cells become ACs. Thus, Lin12 is central to the deci-sion-making process. In genetic mosaics in which oneprecursor cell has the hyperactive Lin12 and the other pre-cursor has the inactive Lin12, the cell with the hyperactiveLin12 always becomes the VU cell and the cell with inac-tive Lin12 always becomes the AC. Assuming that one cellsends a signal and the other cell receives it, explain howthese results suggest that Lin12 encodes a protein requiredto receive the signal. Offer a suggestion for how the fatesof these two precursor cells are normally…genes relies on the existence of ovoD, a dominant female sterile mutation of the ovo gene, whichis located near the middle of the acrocentricDrosophila X chromosome. Females that are ovoD/ovo+ are sterile; ovoD-containing germ-line cellscannot produce eggs.a. Mutations in gene X are recessive lethals, so homozygotes for these mutations do not develop intoadults. Explain how researchers could use the ovoDmutation in a mitotic recombination experiment todetermine (i) whether or not females might supplythe RNA or protein product of gene X to the eggsthey make in their ovaries, and (ii) whether thismaternally supplied product is needed for properdevelopment of their progeny. Where in the genome would gene X need to be located for thisapproach to work?b. The ovoD mutant gene has been cloned, so genomicDNA for this mutant gene is available. How couldyou use this cloned DNA to determine whether any embryonic lethal mutation located anywhere in thegenome was an allele of a maternal effect…
- Enhanced Spatial Learning in Mice With an Autism Mutation Autism is a neurobiological disorder with symptoms that include impaired social interactions and stereotyped patterns of behavior. Around 10 percent of autistic people have an extraordinary skill or talent such as greatly enhanced memory. Mutations in neuroligin 3, an adhesion protein that connects brain cells to one another, have been associated with autism. One mutation changes amino acid 451 from arginine to cysteine. In 2007, Katsuhiko Tabuchi and his colleagues genetically modified mice to carry the same arginine-to-cysteine substitution in their neuroligin 3. Mice with the mutation had impaired social behavior. To test spatial learning ability, the mice were placed in a water maze: a deep pool of warm water in which a platform is submerged a few millimeters below the surface. The platform is not visible to swimming mice. Mice do not particularly enjoy swimming, so they locate a hidden platform as fast as they can. When tested again, they can remember its location by checking visual cues around the edge of the pool. How quickly they remember the platforms location is a measure of spatial learning ability (FIGURE 15.18). FIGURE 15.18 spatial learning ability in mica mutation in neuroligin 3 (R451C), compared with unmodified (wild-type) mica. 1. In the first, how many days did it take unmodified mice to learn to find the location of the hidden platform within 10 seconds?IPSCs are nearly identical to human embryonic stem cells in terms of gene expression, but there may be other ways in which they are not equivalent. For example, the telomeres of IPSCs often vary in length, with many IPSCs cells having telomeres shorter than those of embryonic. How might shortened telomeres affect the life-span of IPSCs or of differentiated cells derived from them?Occasionally more than one sperm penetrates and enters a human secondary oocyte. The result is formation of a multiple spindles and disruption of the first mitotic division. Explain why the presence of multiple sperm has this lethal disruptive effect.
- Cattle in industrial dairy farms are typically given rBST, or recombinant bovine somatotropin. This animal hormone increases a cow's milk production, but there is concern that such hormones may have unforeseen effects on milk-drinking humans. There is no similar concern about plant hormones in the plant foods we eat. Why not?Cat fur coat color genetics is interesting. Orange fur is dominant (''B'') to black fur (''b'') and piebald, which is white spotting, is dominant (''S'') to non-white-spotted (''s''). Both of those genes are located on the X chromosome. In addition, in cats, somatic cells exhibit X-inactivation randomly, which explains why there are tortoiseshell cats (when heterozygous, have orange and black spots) as well as calico cats (orange and black but also with white spots). An orange and white spotted male is mated with a tortoiseshell female. State the genotype of the male and the female.Cat fur coat color genetics is interesting. Orange fur is dominant (''B'') to black fur (''b'') and piebald, which is white spotting, is dominant (''S'') to non-white-spotted (''s''). Both of those genes are located on the X chromosome. In addition, in cats, somatic cells exhibit X-inactivation randomly, which explains why there are tortoiseshell cats (when heterozygous, have orange and black spots) as well as calico cats (orange and black but also with white spots). An orange and white spotted male is mated with a tortoiseshell female. Draw a dihybrid Punnet square to show the possible offspring: