Why can physostigmine, an AchE blocker, be used to treat myasthenia gravis?A. Physostigmine blocks nicotinic acetylcholine receptors.OB. Physostigmine prolongs the action of acetylcholine at the NMJ.OC. Physostigmine increases the amount of acetylcholine in synaptic vesicles.D. Physostigmine increases the reuptake of acetylcholine.O E. Physostigmine blocks K* channels.

Myasthenia gravis is an autoimmune disorder. In this disorder there is muscle weakness due to…

Answered: Why can physostigmine, an AchE blocker,…

Basic Clinical Lab Competencies for Respiratory Care: An Integrated Approach

5th Edition

ISBN:9781285244662

Author:White

Publisher:White

Chapter14: Introduction To Respiratory Care Pharmacology

Section: Chapter Questions

Problem 3SEPT

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Which statement about nicotinic acetylcholine receptors (nAChRs) is true? a. Each nAChR is composed of seven different protein subunits. b. They are ionotropic receptors and conduct Na+ and Ca2+ ions across the cell membrane causing depolarization and a fast excitatory response. c. They are ionotropic receptors and conduct K+ ions across the cell membrane, causing depolarization and a fast excitatory response. d. They are found in all areas of the nervous system except the brain.
Sympathetic stimulation to lung tissue A. Causes activation of beta2 receptors which contract smooth muscles of airways. B. Causes activation of beta1 receptors which relax smooth muscles of airways C. Causes activation of beta2 receptors which relax smooth muscles of airways. D. Causes activation of beta1 receptors which contract smooth muscles of airways
When do neural discharges in the brain have their strongest effect? a. When the discharge rate of individual nerve cell is highest b. When individual nerve cells fire in regular way c. When the coherence of discharges in groups of nerve cells is greatest d. When many nerve cells have burst activity
Give typing answer with explanation and conclusion to all parts The question: a. Explain why competitive nicotine antagonists can produce muscle relaxation. b. Which drug group can be given to restore muscle activity? Explain why the drug group can be used for this purpose.
Give an example of a drug that acts as an agonist for acetylcholine receptors and one that acts as an antagonist for these receptors.
Parkinson Disease A)Causes the loss of stimulation of excitatory motor signals. B)May be treated by levodopa. C)causes the loss of inhibition of excitatory motor signals. D)Results from the loss of dopaminergic neurons in the brain.
Nicotinic receptors are located on which of the following? a. plasma membranes of ganglionic neurons b. target cells that receive parasympathetic innervation c. blood vessels in skeletal muscles d. sweat glands
What would be the purpose of giving a drug that stimulates dopamine receptors to a stroke patient? A. To break up blood clots in the nervous system B. To increase collateral sprouting C. To combat diaschisis D. To stimulate regrowth of axons
Correct the following false statement: "Any cell that has the acetylcholine receptor will have the same response as any other cell that also has the acetylcholine receptor when exposed to acetylcholine". Explain why this statement is false.
What is the direct effect (prior to any reflex compensation) of administeringintravenously a selective beta 1 receptor agonist?A)Increased heart rate, stroke volume and total peripheral resistance.B)Increased total peripheral resistance and renin secretion.C)Increased heart rate and stroke volume.D) Increased total peripheral resistance and decreased renin secretion.E) Decreased renin secretion, total peripheral resistance and heart rate.
A mutation causes a switch in dopamine receptor G-protein-coupled mechanisms in the basal ganglia, causing D1 and D2 receptors to swap their G-protein subunits. If these animals began showing Parkinson-like neuronal degeneration, what would be the resulting motor function? Question 1 options: A) Hyperkinetic B) No change C) Hypokinetic
What are the synaptic effects of glycine? a. It directly stimulates glutamate receptors. b. It facilitates the effects of glutamate. c. It directly stimulates acetylcholine receptors. d. It facilitates the effects of acetylcholine.

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