Biological Science (6th Edition)
6th Edition
ISBN: 9780321976499
Author: Scott Freeman, Kim Quillin, Lizabeth Allison, Michael Black, Emily Taylor, Greg Podgorski, Jeff Carmichael
Publisher: PEARSON
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Chapter 38, Problem 10TYPSS
MODEL Make a sketch of a simplified flower with all four organs, and indicate how the pattern of expression for just two genes (e.g., “D” and “E”) could, hypothetically, regulate the development of each organ. Consider that what’s important is whether a gene is on or off and that a structure can be specified when neither gene is expressed.
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Chapter 38 Solutions
Biological Science (6th Edition)
Ch. 38 - 1. What happens when double fertilization...Ch. 38 - 2. CAUTION In angiosperms, are sperm and eggs...Ch. 38 - Prob. 3TYKCh. 38 - Prob. 4TYKCh. 38 - 5. Why is the emergence of the radicle an...Ch. 38 - What are the advantages and disadvantages of...Ch. 38 - Prob. 7TYUCh. 38 - Prob. 8TYUCh. 38 - Prob. 9TYPSSCh. 38 - 10. MODEL Make a sketch of a simplified flower...
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- Suppose a flower had normal expression of genes A and Cand expression of gene B in all four whorls. Based on the ABChypothesis, what would be the structure of that flower, startingat the outermost whorl?(A) carpel-petal-petal-carpel(B) petal-petal-stamen-stamen(C) sepal-carpel-carpel-sepal(D) sepal-sepal-carpel-carpelarrow_forwardAnalyze the results of Browning and Gunning, andexplain the role of placental transfer tissues in the movement ofnutrients from mother plant to embryo.arrow_forwardCite briefly discuss some problems related to plant embryogenesis.arrow_forward
- What is a meristem? Explain the role of meristems in plant development.arrow_forwardTom studies the floral identity genes. He found a mutant only develop flowers with two whorls of sepals and two whorls of carpels. Which may class(s) of floral homeotic genes be affected based on the ABC model? ______________. (A) A class genes (B) B class genes (C) C class genes (D) E class genesarrow_forwardWhich of the curves shown in the figure best represents the developmental potential of cells as they progress through development? Briefly justify your answer.arrow_forward
- Give an example of how gene expression affects flowering time orflower appearancearrow_forwardHow would plant development change if the functions of the genes SHOOTMERISTEMLESS (STM) and MONOPTEROUS (MP) were reversed? a. The embryo–suspensor axis would be reversed. b. The embryo–suspensor axis would be duplicated. c. The root–shoot axis would be reversed. d. The root–shoot axis would be duplicated.arrow_forwardCan you solve all the parts to this question please a) What is the role of the LEAFY gene in floral development? (b) What is the phenotype of the leafy mutant? (c) Most of the ABC program genes are directly regulated by the transcription factor LEAFY (LFY). Still, LFY is expressed broadly throughout the floral meristem when the discrete A, B, and C domains are established. Discuss how this single broadly expressed transcription factor can generate distinct domains of ABC gene expression. Feel free to use diagrams.arrow_forward
- Monoecious plants such as corn have either staminate or carpelate flowers. Knowing what you do about the molecular mechanisms of floral development, which of the following might explain the development of single-sex flowers? a. Expression of B-type genes in the presumptive carpel whorl will generate staminate flowers. b. Loss of A-type genes in the presumptive petal whorl will allow C-type and B-type genes to produce stamens instead of petals in that whorl. c. Restricting B-type gene expression to the presumptive petal whorl will generate carpelate flowers. d. All of the choices are correct.arrow_forwardPlants and animals usually battle each other; describe why and how this occurs. Once animal-pollinated flowers evolved, a new type of plant–animal interaction was possible; describe it. How is it possible for a mutation in aplant to be beneficial for both the plant and for an animal?arrow_forward(a) What is the mutant that helps us to understand the adaxial identity of leaf development and the laminar outgrowth? (b) What is the mutant phenotype? (c) Which kind of protein does this gene encode?arrow_forward
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