Prescott's Microbiology
10th Edition
ISBN: 9781259281594
Author: Joanne Willey, Linda Sherwood Adjunt Professor Lecturer, Christopher J. Woolverton Professor
Publisher: McGraw-Hill Education
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Chapter 39, Problem 4CHI
While many Vibrio cholerae strains are found in aquatic environments, only a small fraction of these cause human disease. When in aquatic ecosystems, V. cholerae is frequently found attached to the exoskeleton of zooplankton, which is made of chitin. Indeed, V. cholerae produces an extracellular chitinase, so zooplanktonassociated growth presumably provides a good source of organic carbon and nitrogen. Growth on zooplankton and in the human gut shares the requirement for attachment proteins. Remarkably, the same protein, called GbpA, has been shown to bind to both the N-acetylglucosamine (GlcNAc) of zooplankton chitin and to epithelial cells. The glycoproteins and lipids on the surface of epithelial cells are commonly modified with GlcNAc. How would you show whether or not GbpA binds specifically to epithelial GlnNAc? How would you determine if GbpA is needed for virulence? How would you test the hypothesis that GbpA is produced by pathogenic strains but not, or to a lesser extent, by nonpathogenic strains of V. cholerae?
Read the original paper: Kirn, T. J., et al. 2005. A colonization factor links Vibrio cholerae environmental survival and human infection. Nature 438:863.
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Chapter 39 Solutions
Prescott's Microbiology
Ch. 39.1 - What organisms serve as the environmental...Ch. 39.1 - How is the M protein also thought to be involved...Ch. 39.1 - Prob. 3MICh. 39.1 - Why do you think chlamydiae differentiate into...Ch. 39.1 - Compare and contrast the means by which people...Ch. 39.1 - Prob. 1.3RIACh. 39.1 - What are the three major causes of meningitis? Why...Ch. 39.1 - Prob. 2.2RIACh. 39.1 - What is the reason for the complex antibiotic...Ch. 39.1 - How do multidrug-resistant strains of tuberculosis...
Ch. 39.1 - Prob. 3.1RIACh. 39.1 - What is the mechanism by which PTx kills host...Ch. 39.1 - Prob. 3.3RIACh. 39.1 - Describe the streptococcal exotoxins and how they...Ch. 39.1 - Prob. 4.2RIACh. 39.2 - What are the three stages of Lyme disease? What...Ch. 39.2 - Prob. 1RIACh. 39.2 - Prob. 2RIACh. 39.2 - Prob. 3RIACh. 39.2 - Prob. 4RIACh. 39.2 - How does transovarian passage occur?Ch. 39.3 - What are the chief differences between tuberculoid...Ch. 39.3 - How does H. pylori increase the local pH in its...Ch. 39.3 - Prob. 3MICh. 39.3 - Prob. 4MICh. 39.3 - Prob. 5MICh. 39.3 - Prob. 1.1RIACh. 39.3 - Why do you think the slow growth rate of M. leprae...Ch. 39.3 - Prob. 2.1RIACh. 39.3 - Prob. 2.2RIACh. 39.3 - Prob. 2.3RIACh. 39.3 - Prob. 2.4RIACh. 39.3 - Prob. 2.5RIACh. 39.3 - Prob. 3.1RIACh. 39.3 - Prob. 3.2RIACh. 39.3 - What is the difference between S. aureus and MRSA?Ch. 39.3 - Prob. 3.4RIACh. 39.3 - Prob. 3.5RIACh. 39.3 - Prob. 3.6RIACh. 39.4 - How do the neurological effects of botulinum toxin...Ch. 39.4 - To which E. coli class does the strain 0157:H7...Ch. 39.4 - How can Clostridium botulinum cause disease even...Ch. 39.4 - Compare the gastroenteritis caused by Vibrio...Ch. 39.4 - Prob. 2.1RIACh. 39.4 - Compare toxigenic E. coli disease with invasive...Ch. 39.4 - Prob. 2.3RIACh. 39.4 - Prob. 3.1RIACh. 39.4 - Prob. 3.2RIACh. 39.4 - Prob. 3.3RIACh. 39.5 - Prob. 1MICh. 39.5 - Prob. 1RIACh. 39.5 - Prob. 2RIACh. 39.5 - How is ornithosis transmitted?Ch. 39.5 - Prob. 4RIACh. 39.6 - What cells and tissues make up the pseudomembrane?Ch. 39.6 - Prob. 2MICh. 39.6 - Prob. 1RIACh. 39.6 - In both C. difficileassociated disease and...Ch. 39.6 - Prob. 3RIACh. 39 - Describe a typhoid carrier. How does one become a...Ch. 39 - Many consider cholera as the most severe form of...Ch. 39 - Compare the three stages of syphilis and Lyme...Ch. 39 - While many Vibrio cholerae strains are found in...Ch. 39 - The spirochete Borrelia burgdorferi causes Lyme...
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