1. How is UV radiation a good type of control mechanism against microbial growth? Please explain what happens to the microbe and effects this control causes.

Biology: The Dynamic Science (MindTap Course List)
4th Edition
ISBN:9781305389892
Author:Peter J. Russell, Paul E. Hertz, Beverly McMillan
Publisher:Peter J. Russell, Paul E. Hertz, Beverly McMillan
Chapter26: Prokaryotes: Bacteria And Archaea
Section: Chapter Questions
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1. How is UV radiation a good type of control mechanism against microbial growth? Please explain what happens to the microbe and effects this control causes.

2. Suppose you do the Kirby-Bauer test on a hypothetical Staphylococcus species with penicillin and tetracycline. You record diameters of 20mm for tetracycline and 24mm for penicillin. Which antibiotic is most effective against this bacterium and why? Please explain and interpret these results.

3. Please provide the scientific name of your microbe that was used in the UV experiment (i.e. S. aureus). Compare your plates and interpret/analyze your results. Please discuss your findings and any patterns you were able to gather.

4. After performing the “Effects of Antiseptics & Disinfectants” lab which agent(s) showed potential to control S. marcescens growth? P. aeruginosa? Please explain why you believe these agent(s) work.

5. What purpose does water serve in the “Effects of Antiseptics & Disinfectants” lab? What did you learn about the use of soap in this same lab?

6. Please provide your results from the Kirby-Bauer Antibiotic susceptibility test.

Antibiotic

Inhibition Zone of (microbe 1) in mm

Sensitivity Result of (microbe 1)

Inhibition Zone of (microbe 2) in mm

Sensitivity Result of (microbe 2)

Gentamicin

 

 

 

 

Penicillin

 

 

 

 

SXT

 

 

 

 

Vancomycin

 

 

 

 

Tetracycline

 

 

 

 

Erythromycin

 

 

 

 

(please insert the name of your microbe for microbe 1 & microbe 2)

7. Based upon your findings which antibiotic would you recommend using for each microbe & explain why.

8. Some antibiotics show no zone of inhibition against the microbe. How is this possible? Explain how the microbe is able to combat against the antibiotic with proper reasoning.

 
 
 
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