ExamsoftE mitochondriaQuestion #: 25Xconcentration of acetylcholine had no effect on enzyme inhibition. Further, analysis of the mixture indicted that noWhile developing new inhibitors we came up with new compound, a derivative of acetylcholine (neurotransfer)This new neurotransmitter showed inhibition of the enzyme A in presence of acetylcholine. In addition, increase inadditional products or intermediate were formed or seen. Based on this information this inhibitor is;A. Competitive inhibitorB. Non-competitive InhibitorC. Covalent InhibitorD. None of the above

Enzymes are high molecular weight protein that catalyse biochemical reactions. They contain a active…

Answered: E mitochondria Question #: 25 While…

Biochemistry

6th Edition

ISBN:9781305577206

Author:Reginald H. Garrett, Charles M. Grisham

Publisher:Reginald H. Garrett, Charles M. Grisham

Chapter19: The Tricarboxylic Acid Cycle

Section: Chapter Questions

Problem 19P

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If cardiac muscle cells were prevented from undergoing aerobic metabolism, they ultimately would ________. undergo glycolysis synthesize ATP stop contracting start contracting
Focus on the interaction of ATP with the kinase first. Would you say ATP binds on the surface of the kinase or in a cleft? What color are the phosphorus atoms? What color are the Mn2+ ions?Which part of ATP do the Mn2+ ions interact with?
Please ASAP. Thank you. KCNQ or Slow delayed rectifier channels : When is this channel mostly activated? What is the function of this channel? What happens if this channel is inactivated?
30. Runaway mitochondria 1 . Suppose that the mitochondria of a patient oxidize NADH irrespective of whether ADP is present. The P : O ratio for oxidative phosphorylation by these mitochondria is less than normal. Predict the likely symptoms of this disorder.
exaplin the intracellular mehcanims of smooth msucle via agonist such as Hitamine and acytycholine. link hisamine to muscarnic recptors link acetycholine to a neurotransmittors Give a diagram for each.
Please ASAP. Thank you. KATP currents: What controls the gating of this channel? What happens when the ATP levels decreases?
27. Working at cross-purposes . Write a balanced equation showing the effect of simultaneous activation of glycogen phosphorylase and glycogen synthase. Include the reactions catalyzed by phosphoglucomutase and UDP-glucose pyrophosphorylase.
Assessing the Effect of Active-Site Phosphorylation on Enzyme Activity (Integrates with Chapter 15.) The serine residue of isocitrate dehydroenase that is phosphorylated by protein kinase lies within the active site of the enzyme. This situation contrasts with most other examples of coa1ent modification by protein phosphorylation. where the phosphorylation occurs at a sate remote from the active site. What direct effect do you think such active-site phosphorylation might have on the catalytic activity of isocitrate dehydrogcn.ise? (Sec Barford, D., 1991. Molecular mechanisms for the control of enzymic activity by protein phosphorytation. Biochimica et Biophysica Acta 1133:55â€”62.)
BIOC 384 Structure and Function of Pyruvate Dehydrogenase Q10.2: What is the metabolic logic regarding stimulation of pyruvate dehydrogenase kinase (PDK) activity by ATP, whereas PDK activity is inhibited by ADP?
What biochemical alterations facilitate the switch to aerobic glycolysis?
Pls help ASAP, thank you! "Regulation of metabolic pathways most typically occurs at enzyme steps that are"
5.Myasthenia (muscle weakness)is characterized by damage in the transmission of neurotic impuise and decreased amount of acetylcholine due to autoimmune mechanisms. Prozerine is used for treatment of this disease.Specify the mechanism of this drug action. For this: 1)Write the reaction of acetylcholine degradation at neuromuscular junction. 2)What enzyme will be inhibited with prozerine? Name the class of this enzyme. 3)What is the mechanism of this enzyme inhibition?

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