Loose Leaf For Anatomy & Physiology: An Integrative Approach
3rd Edition
ISBN: 9781260162493
Author: McKinley Dr., Michael; O'Loughlin, Valerie; Bidle, Theresa
Publisher: McGraw-Hill Education
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Question
Chapter 15.6, Problem 19WDL
Summary Introduction
To determine:
The possibility for the stimulation of adrenergic receptors to result in either vasodilation or vasoconstriction of the selected vessels of the blood.
Concept introduction:
There are cholinergic as well as adrenergic receptors. The signalling molecules like hormones or neurotransmitters are known as ligands as they get bound to the receptors. The ligands class, which bind to the receptors (adrenergic) in neurons is known as the monoamines or biogenic
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Students have asked these similar questions
release of --------- onto skeletal muscle blood vessles result in ......
1. Acetylcholine, vasoconstriction
2.norepinephrine,vasoconstriction
3.Acetylcholine, vasolidation
4,norepinephrine, vasolifation
1.can you tell me why do we choose sympathetic instead of para symapthetic. make it short and clear.
2.If we say sympathetic why do we choose pregangolic with ACH ? how can I know?
3. explain how is alpha beta thing relate. How do you choose vasoconstriction vs vasolidation?
please teach me a trick. make it short and claer please. I get so confused
Activation of α-adrenergic receptors in vascular smooth muscle stimulates (vasodilation or vasoconstriction). Give an example of an organ system where this occurs.
Why is it possible for acetylcholine to actually cause an increase in stroke volume, even though increased parasympathetic activity causes an overall decrease in cardiac output?
Chapter 15 Solutions
Loose Leaf For Anatomy & Physiology: An Integrative Approach
Ch. 15.1 - LEARNING OBJECTIVE
1. List the similarities and...Ch. 15.1 - What criterion is used to organize the nervous...Ch. 15.1 - Prob. 2LOCh. 15.1 - Prob. 3LOCh. 15.1 - Prob. 2WDLCh. 15.1 - Prob. 4LOCh. 15.1 - What CNS structure is the integration and command...Ch. 15.2 - Prob. 5LOCh. 15.2 - Prob. 4WDLCh. 15.2 - Prob. 6LO
Ch. 15.2 - Describe the general anatomic differences in the...Ch. 15.2 - Prob. 7LOCh. 15.2 - Prob. 6WDLCh. 15.3 - Prob. 8LOCh. 15.3 - Prob. 1WDTCh. 15.3 - Which four cranial nerves have a parasympathetic...Ch. 15.3 - Prob. 9LOCh. 15.3 - What organs are innervated by the pelvic...Ch. 15.4 - Prob. 10LOCh. 15.4 - Prob. 11LOCh. 15.4 - Prob. 12LOCh. 15.4 - Prob. 13LOCh. 15.4 - Prob. 9WDLCh. 15.4 - Prob. 10WDLCh. 15.4 - Prob. 14LOCh. 15.4 - Prob. 15LOCh. 15.4 - Prob. 2WDTCh. 15.4 - Prob. 11WDLCh. 15.4 - In what ways does the adrenal medulla pathway help...Ch. 15.5 - Prob. 16LOCh. 15.5 - Prob. 17LOCh. 15.5 - Prob. 13WDLCh. 15.5 - Prob. 14WDLCh. 15.6 - Prob. 18LOCh. 15.6 - Prob. 15WDLCh. 15.6 - Prob. 19LOCh. 15.6 - Prob. 3WDTCh. 15.6 - Prob. 16WDLCh. 15.6 - Prob. 17WDLCh. 15.6 - Prob. 20LOCh. 15.6 - Prob. 21LOCh. 15.6 - Prob. 18WDLCh. 15.6 - Prob. 19WDLCh. 15.7 - LEARNING OBJECTIVE
22. Discuss the nature of...Ch. 15.7 - Prob. 20WDLCh. 15.7 - Prob. 23LOCh. 15.7 - LEARNING OBJECTIVE
24. Describe the antagonistic...Ch. 15.7 - Prob. 21WDLCh. 15.7 - Prob. 22WDLCh. 15.7 - Prob. 25LOCh. 15.7 - What are the body structures innervated by the...Ch. 15.8 - Prob. 26LOCh. 15.8 - 27. Describe some major examples of autonomic...Ch. 15.8 - Prob. 24WDLCh. 15 - A splanchnic nerve in the sympathetic division of...Ch. 15 - Some parasympathetic preganglionic neuron cell...Ch. 15 - Prob. 3DYBCh. 15 - Prob. 4DYBCh. 15 - Sympathetic division preganglionic axons travel to...Ch. 15 - Prob. 6DYBCh. 15 - Prob. 7DYBCh. 15 - A sympathetic postganglionic axon is a. long and...Ch. 15 - Prob. 9DYBCh. 15 - Prob. 10DYBCh. 15 - Prob. 11DYBCh. 15 - For the following ganglia, identify the location...Ch. 15 - Prob. 13DYBCh. 15 - Prob. 14DYBCh. 15 - Prob. 15DYBCh. 15 - Prob. 16DYBCh. 15 - Prob. 17DYBCh. 15 - What may occur with the mass activation of the...Ch. 15 - Prob. 19DYBCh. 15 - Prob. 20DYBCh. 15 - Prob. 1CALCh. 15 - Arlene was in a heightened state of alertness well...Ch. 15 - George has hypertension (high blood pressure). His...Ch. 15 - Prob. 4CALCh. 15 - Prob. 5CALCh. 15 - Prob. 1CSLCh. 15 - When you were younger, your parents may have told...Ch. 15 - Prob. 3CSL
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Similar questions
- Activation of β2-adrenergic receptors in vascular smooth muscle stimulates vasodilation / vasoconstriction. Give an example of organs where this occurs.arrow_forwardWhat physiological factors influence cerebral blood flow? How can this be changed during stress conditions? What are the major compensatory responses to acute blood loss? List the most important hormones involved. What is a major difference between chronic and acute blood loss? Give some examples.arrow_forwardRoberto is a 78-year-old male who has recently been diagnosed with Class III Congestive Heart Failure (CHF). His physician told him that his left ventricle has become stiff and cannot relax. Although there is no current treatment that can “fix” Roberto’s condition, he is prescribed two medications: an ACE inhibitor and beta blockers. Which of these statements is most likely true of Roberto’s current symptoms? He doesn’t experience any symptoms at all at rest or during exercise. He has no symptoms at all during rest, but experiences palpitations, fatigues, and shortness of breath during mild exercise. He experiences fatigue, shortness of breath, and palpitations at rest and is not able to participate in any physical activity.arrow_forward
- What is the cause of reflex tachycardia?arrow_forwardwhat drives the enhanced sympathetic outflow to most other arterioles, the heart, and the veins and the decreased parasympathetic outflow to the heart?arrow_forwardFollowing a large increase in sympathetic output from the vasomotor center, what tissues will get less blood (increased resistance, reduced flow), and which ones will continue to get plenty of flow? How is sympathetic control of skeletal muscle arterioles different from other vascular beds?arrow_forward
- What effect does sympathetic stimulation have on stroke volume if thevenous return remains constant? Dilation of the coronary blood vesselsoccurs in response to an increased heart rate and stroke volume. Explainthe functional advantage of that effect.arrow_forwardDiscuss the concept of termination of neurotransmitter action by comparing the mechanisms by which acetylcholine and nitric oxide's actions are terminated. (a) Name the three primary mediators of purinergic receptors. (b) Which one of these mediators is sometimes used to treat supraventricular tachycardia? (c) Explain why the drug in (b) is considered safer than verapamil in the treatment of supraventricular tachycardia?arrow_forwardMr. Wilson, age 85, lives in a care facility, where he is not very mobileand is often lethargic. One day an aide noticed that he was sitting inhis usual chair but appeared to be unconscious. She took his pulse,which was 140 beats/minute and then took his blood pressure, whichwas 190/130. Which of the following conditions is (are) consistent withthese observations?a. strokeb. activation of the CNS ischemic responsec. heart attackd. shocke. Both a and b are correct.arrow_forward
- A beta 2-adrenergic agent was prescribed for further use because it has less cardiostimulatory (beta1) effect. Based on your knowledge of beta1 and beta2 receptors, why is this a good suggestion?arrow_forwardIdentify the most commonly used antihypertensives that can produce orthostatic hypotension. Which of the drugs affecting the sympathetic nervous system can also cause bradycardia? Describe the most significant adverse effects of direct acting vasodilators. Compare their effects to phosphodiesterase inhibitors.arrow_forwardEpinephrine is released during vigorous exercise. How would epinephrine cause vasoconstriction in the digestive organs but vasodilation in the heart?arrow_forward
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