EBK ANATOMY & PHYSIOLOGY: THE UNITY OF
7th Edition
ISBN: 8220102796409
Author: SALADIN
Publisher: YUZU
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Chapter 17, Problem 1TYC
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The steroid hormone produces great
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Explain how FRET could be used to monitor the association of Gαs and adenylyl cyclase following activation of the epinephrine receptor.
A hormone signals through a G protein-coupled receptor as shown in the diagram. After the production of IP3, which of these events will MOST quickly stop the transduction of the signal?
A. the hydrolysis of IP3
B. the hydrolysis of GTP
C. the hydrolysis of PIP2
D. the hydrolysis of the hormone
I believe the answer is (B) the hydrolysisof GTP, because the G protein becomes inactive after GTP is hydrolyzed to GDP. However, the solution provided by Bartleby is (C). Please explain. Thanks
A cellular response occurs when a signal transduction pathway is activated in animals. Explain two forms of cellular responses that may arise as a result of paracrine or endocrine signaling.
Chapter 17 Solutions
EBK ANATOMY & PHYSIOLOGY: THE UNITY OF
Ch. 17.1 - Prob. 1BYGOCh. 17.1 - Prob. 2BYGOCh. 17.1 - Prob. 3BYGOCh. 17.1 - Prob. 4BYGOCh. 17.1 - Prob. 5BYGOCh. 17.1 - Prob. 1AYLOCh. 17.1 - Prob. 2AYLOCh. 17.1 - Prob. 3AYLOCh. 17.1 - Prob. 4AYLOCh. 17.1 - Prob. 5AYLO
Ch. 17.2 - Prob. 6BYGOCh. 17.2 - Prob. 7BYGOCh. 17.2 - Prob. 8BYGOCh. 17.2 - Prob. 9BYGOCh. 17.2 - Prob. 10BYGOCh. 17.2 - Prob. 1AYLOCh. 17.2 - Prob. 2AYLOCh. 17.2 - Prob. 3AYLOCh. 17.2 - Prob. 4AYLOCh. 17.2 - Prob. 5AYLOCh. 17.2 - Prob. 6AYLOCh. 17.2 - Prob. 7AYLOCh. 17.2 - Prob. 8AYLOCh. 17.3 - Prob. 11BYGOCh. 17.3 - Prob. 12BYGOCh. 17.3 - Prob. 13BYGOCh. 17.3 - Prob. 14BYGOCh. 17.3 - Prob. 15BYGOCh. 17.3 - Prob. 16BYGOCh. 17.3 - Prob. 1AYLOCh. 17.3 - Prob. 2AYLOCh. 17.3 - Prob. 3AYLOCh. 17.3 - Prob. 4AYLOCh. 17.3 - Prob. 5AYLOCh. 17.3 - Prob. 6AYLOCh. 17.3 - Prob. 7AYLOCh. 17.3 - Prob. 8AYLOCh. 17.3 - Prob. 9AYLOCh. 17.3 - Prob. 10AYLOCh. 17.4 - Prob. 17BYGOCh. 17.4 - Prob. 18BYGOCh. 17.4 - Prob. 19BYGOCh. 17.4 - Prob. 20BYGOCh. 17.4 - Prob. 21BYGOCh. 17.4 - Prob. 1AYLOCh. 17.4 - Prob. 2AYLOCh. 17.4 - Prob. 3AYLOCh. 17.4 - Prob. 4AYLOCh. 17.4 - Prob. 5AYLOCh. 17.4 - Prob. 6AYLOCh. 17.4 - Prob. 7AYLOCh. 17.4 - Prob. 8AYLOCh. 17.4 - Prob. 9AYLOCh. 17.4 - Prob. 10AYLOCh. 17.4 - Prob. 11AYLOCh. 17.4 - Prob. 12AYLOCh. 17.5 - Prob. 22BYGOCh. 17.5 - Prob. 23BYGOCh. 17.5 - Prob. 24BYGOCh. 17.5 - Prob. 1AYLOCh. 17.5 - Prob. 2AYLOCh. 17.5 - Prob. 3AYLOCh. 17.6 - Prob. 25BYGOCh. 17.6 - Prob. 26BYGOCh. 17.6 - Prob. 27BYGOCh. 17.6 - Prob. 1AYLOCh. 17.6 - Prob. 2AYLOCh. 17.6 - Prob. 3AYLOCh. 17.6 - Prob. 4AYLOCh. 17.6 - Prob. 5AYLOCh. 17.7 - Prob. 28BYGOCh. 17.7 - Prob. 29BYGOCh. 17.7 - Prob. 30BYGOCh. 17.7 - Prob. 1AYLOCh. 17.7 - Prob. 2AYLOCh. 17.7 - Prob. 3AYLOCh. 17.7 - Prob. 4AYLOCh. 17.7 - Prob. 5AYLOCh. 17.7 - Prob. 6AYLOCh. 17.7 - Prob. 7AYLOCh. 17.7 - Prob. 8AYLOCh. 17 - CRH secretion would not raise the blood...Ch. 17 - Prob. 2TYRCh. 17 - Prob. 3TYRCh. 17 - Prob. 4TYRCh. 17 - Prob. 5TYRCh. 17 - Prob. 6TYRCh. 17 - Prob. 7TYRCh. 17 - Prob. 8TYRCh. 17 - Prob. 9TYRCh. 17 - Prostaglandins are derived from a. phospholipase....Ch. 17 - Prob. 11TYRCh. 17 - Prob. 12TYRCh. 17 - Growth hormone hypersecretion in adulthood causes...Ch. 17 - Prob. 14TYRCh. 17 - Prob. 15TYRCh. 17 - Prob. 16TYRCh. 17 - Target cells can reduce pituitary secretion by a...Ch. 17 - Prob. 18TYRCh. 17 - Prob. 19TYRCh. 17 - ______ is a process in which a cell increases its...Ch. 17 - adeno-Ch. 17 - Prob. 2BYMVCh. 17 - Prob. 3BYMVCh. 17 - Prob. 4BYMVCh. 17 - Prob. 5BYMVCh. 17 - Prob. 6BYMVCh. 17 - Prob. 7BYMVCh. 17 - Prob. 8BYMVCh. 17 - Prob. 9BYMVCh. 17 - Prob. 10BYMVCh. 17 - Prob. 1TFCh. 17 - Prob. 2TFCh. 17 - Prob. 3TFCh. 17 - Prob. 4TFCh. 17 - Prob. 5TFCh. 17 - Prob. 6TFCh. 17 - Prob. 7TFCh. 17 - A deficiency of dietary iodine would lead to...Ch. 17 - Prob. 9TFCh. 17 - Prob. 10TFCh. 17 - Prob. 1TYCCh. 17 - Suppose you were browsing in a health-food store...Ch. 17 - Prob. 3TYCCh. 17 - Prob. 4TYCCh. 17 - A young man is involved in a motorcycle accident...
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- Imagine a disease that involves reduced expression of receptor X. There is no concentration of full agonist that can restore receptor X signaling back to normal levels. Explain how an allosteric modulator may be used to help in this situation.arrow_forwardHuman growth hormone binds to a cell-surface membrane protein that is not a receptor tyrosine kinase. The intracellular domain of the receptor can bind other proteins inside the cell. Furthermore, studies indicate that the receptor is monomeric in the absence of hormone but dimerizes on hormone binding. Propose a possible mechanism for growth-hormone signaling.arrow_forwardDescribe two features of insulin signaling that affect glucose utilization. Aβ-adrenergic response can be modulated through the actions of a receptorkinase and arrestin because phosphorylation by the kinase desensitizes thereceptor. How might signaling by a tyrosine receptor kinase, such as theinsulin receptor, be modulated?arrow_forward
- Discuss the family of nuclear hormone receptors and explain how ligand binding to such a receptor can result in changes in gene expressionarrow_forwardInsulin is a hormone that regulates blood sugar by binding to its receptor, insulin receptor tyrosine kinase. How does insulin’s behavior differ from steroid hormone signaling, and what can you infer about its structure?arrow_forwardDescribe a mechanism by which a steroid hormone might act to increase intracellular levels of cyclic AMP. Match the words in the left column to the appropriate blanks in the sentences on the right. activate decrease inhibit repressed induced increase Reset Help Since steroid hormone-receptor complexes specific genes, a steroid hormone could in principle transcription of the gene for adenylate cyclase and, hence, steady-state level of this enzyme. Other possibilities could include synthesis of a phosphodiesterase inhibitor or synthesis of cAMP phosphodiesterase. the transcription of thearrow_forward
- A hormone signals through a G protein-coupled receptor as shown in the diagram. After the production of IP3, which of these events will MOST quickly stop the transduction of the signal? A. the hydrolysis of IP3 B. the hydrolysis of GTP C. the hydrolysis of PIP2 D. the hydrolysis of the hormonearrow_forwardA hormone signals through a G protein- coupled receptor as shown in the diagram. After the production of IP3, which of these events will MOST quickly stop the transduction of the signal? A. the hydrolysis of IP3 B. the hydrolysis of GTP C. the hydrolysis of PIP2 D. the hydrolysis of the hormonearrow_forwardOverexpression of this receptor in the cells of the adrenal gland causes Cushing's syndrome, a disease caused by the overproduction of cortisol. One strategy that scientists have employed to treat this disease is the use of small molecules that bind to, but do not activate, MC2R. This type of molecule is generically referred to as an antagonist. How can an antagonist bind to the same receptor as ACTH but not activate it? O a. The antagonist can bind covalently while ACTH binds non-covalently O b. The antagonist is only partially complimentary to the binding pocket of MC2R O The antagonist binds to ACTH and blocks it from binding to the receptor properly O d. The antagonist is the exact same structure as ACTH but since it is synthetic it doesn't work One cause of Cushing's syndrome is the inappropriate production of GPCRS in the adrenal gland that are not normally present in those cells. When these receptors are activated they result in the production of cortisol. Which statement about…arrow_forward
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