Prescott's Microbiology
10th Edition
ISBN: 9781259281594
Author: Joanne Willey, Linda Sherwood Adjunt Professor Lecturer, Christopher J. Woolverton Professor
Publisher: McGraw-Hill Education
expand_more
expand_more
format_list_bulleted
Textbook Question
Chapter 18.5, Problem 1MI
Figure 18.12 Metabolic Pathways and Transport Systems of Treponema pallidum. This depicts T. pallidum
Based on this genomic reconstruction, can you determine if T. pallidum has a respiratory or fermentative metabolism?
Expert Solution & Answer
Trending nowThis is a popular solution!
Students have asked these similar questions
Yarrowia lipolytica(Yl) is a non-conventional yeast that diverged from baker’s yeast, Saccharomyces cerevisiae(Sc), early in evolutionary history. The regulation of glycolytic enzymes in Yl differs from that of Sc. The figures below show how PFK activity is affected by fructose-2,6-bisphosphate and phosphoenolpyruvate. Based on these data, which molecule is the major regulator of PFK activity in each species?
Yarrowia lipolytica(Yl) is a non-conventional yeast that diverged from baker’s yeast, Saccharomyces cerevisiae(Sc), early in evolutionary history. The regulation of glycolytic enzymes in Yl differs from that of Sc. The data below pertain to regulation of phosphofructokinase 1 (PFK). Data for Yl is shown with black circles and data for Sc is shown with white circles.
Pfk for both species is a homo-octomer (eight identical subunits). Given the data below, which species is more likely to be susceptible to allosteric modification? Briefly explain.
Why are there reversible steps in the glycolytic pathway? Explain why they all be could not be irreversible?
Chapter 18 Solutions
Prescott's Microbiology
Ch. 18.1 - MICRO INQUIRY What is the function of the 3-OH...Ch. 18.1 - MICRO INQUIRY Why is it important that identical...Ch. 18.2 - MICRO INQUIRY Which step (or steps) in this...Ch. 18.2 - Retrieve, Infer, Apply Why is the Sanger technique...Ch. 18.2 - Retrieve, Infer, Apply Explain the difference...Ch. 18.2 - Retrieve, Infer, Apply Why does reversible chain...Ch. 18.2 - Prob. 4RIACh. 18.2 - Retrieve, Infer, Apply Suggest a medical and an...Ch. 18.3 - Retrieve, Infer, Apply What is a phylotype?Ch. 18.3 - Retrieve, Infer, Apply NGS techniques are...
Ch. 18.3 - Retrieve, Infer, Apply Examine figure 18.8. How...Ch. 18.4 - Prob. 1MICh. 18.5 - Figure 18.12 Metabolic Pathways and Transport...Ch. 18.5 - Prob. 2MICh. 18.5 - Prob. 3MICh. 18.5 - Prob. 1.1RIACh. 18.5 - Retrieve, Infer, Apply How might the following...Ch. 18.5 - Retrieve, Infer, Apply What specific limitations...Ch. 18.5 - Retrieve, Infer, Apply Compare and contrast...Ch. 18.5 - Retrieve, Infer, Apply Why does two-dimensional...Ch. 18.5 - Retrieve, Infer, Apply What is the difference...Ch. 18.5 - Retrieve, Infer, Apply Describe a ChIP-Seq...Ch. 18.7 - Prob. 1MICh. 18.7 - Retrieve, Infer, Apply Cite an infectious disease...Ch. 18.7 - Prob. 2RIACh. 18.7 - Prob. 3RIACh. 18 - Propose an experiment that can be done easily with...Ch. 18 - Prob. 2CHICh. 18 - You are developing a new vaccine for a pathogen....Ch. 18 - Prob. 4CHICh. 18 - Prob. 5CHICh. 18 - Prob. 6CHI
Knowledge Booster
Learn more about
Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- The pathway for cystiene synthesis in E.coli involves five precursor compounds, compound A-E. The requirements for each of the autotrophs are summarized in the table below. Use the data, define the biosynthetic pathway. Draw the pathway for cysteine synthesis. Fill in the blanks with A-E compounds as appropriate and then label the arrows with the auxotrophs C1-C8 that are unable to complete that step. There are may be more than one quxotroph that is defective at a particular step. that is - on C4 in Darrow_forwardReferring to figure 5 and 8 which substrate molecule serves as the phosphate donor during substrate-level phosphorylation in step 10 of glycosis and the succinyl CoA-succinate step of the Krebs Cycle?arrow_forwardWhich type of carbohydrate will be the most effective to increase cellular respiration rate in the yeast? Why?arrow_forward
- Trypanosoma brucei is a parasitic protozoan that causes sleeping sickness in humans, Transmitted by the tsetse fly, sleeping sickness is a fatal disease characterized by fever, anemia, inflammation, lethargy, headache, and convulsions. When trypanosomes are present in the human bloodstream, they depend on glycolysis entirely for energy generation. The first seven glycolytic enzymes in these organisms are localized in peroxisome-like organelles called glycosomes, which are only regulated weakly by allosteric regulator molecules. Glycosomes take up glucose and export glycerate-3-phosphate. There are two pools of ADP and ATP (cytoplasmic and glycosomal), and the glycosomal membrane is impermeable to both nucleotides as well as most other glycolytic intermediates. If the glycosomal membrane is compromised, the concentration of phosphorylated glycolytic intermediates rises and the cells die. Explain.arrow_forwardWhat kind of enzymatic process is represented in this diagramarrow_forwardPompe disease is due to a deficiency in lysosomal α-1,4-glucosidase, also called acid maltase. Lysosomes function as recycling centers in the cell and normally degrade excess muscle glycogen into glucose for the energy-converting reactions of the glycolytic pathway. Pompe disease was described in 1932 by Dutch pathologist Johann Pompe, who recognized that lysosomes in the affected patient's accumulated large amounts of glycogen. However, Henri Hers—the same Belgian pathologist who described the symptoms of liver glycogen phosphorylase deficiency (Hers disease)—discovered in 1965 that Pompe disease was due to a lysosomal α-1,4-glucosidase deficiency. Enzyme deficiency Disease name Organ Disease symptoms Lysosomal α-1,4- glucosidase Pompe All organs Heart failure in infantile form; muscle defects in juvenile form Answer these questions: What protein/enzyme does the mutation effects? Symptoms and, if you can, how are the symptoms…arrow_forward
- Penicillin is an irreversible inhibitor of the transpeptidases, enzymes that cross-link components of the bacterial cell wall. The cell wall is thus fragile and the bacteria die. Penicillin and its derivatives are structural analogs of the natural substrates of these enzymes. Why doesn't penicillin normally kill humans, unless a severe allergic reaction (anaphylaxis) develops? How does penicillin inhibit transpeptidase?arrow_forwardWhat if the drug inhibited the linking of NAG directly to NAM in peptidoglycan? Would you expect it cause mortality in Strepte pneumonie? Please explain your answer. asap pleasearrow_forwardWhen making bread with common yeast, the reaction starts as an aerobic process and then becomes an anaerobic process after the oxygen is spent. A baker has created a new strain of yeast which contains no cytochrome c gene and, thus, no cytochrome c protein. This will affect what the yeast strain can do to obtain energy. Use the passage to answer the question. Since the air bubbles in bread are generated by yeast, how will the spaces in this bread compare to those in a loaf made using normal yeast? A. more bubbles B. differently-shaped bubbles C. smaller bubbles D. bubbles more on the interior of the loafarrow_forward
- In the presence of oxygen, the mitochondrion in yeast is used for aerobic respiration,however, under anaerobic conditions,the yeast mitochondria have been found to have other function. Identify and briefly discuss four of these functionsarrow_forwardGive the 3 major pathways that eventually become entry points of molecules into the Krebs Cycle. What are the molecules from these respective pathways are integrated into the Krebs Cycle?arrow_forwardA genetic disease called glucose-6-phosphate dehydrogenase deficiency is inherited in a manner similar to that of sickle-cell anemia except that it occurs most frequently in males. The defective enzyme cannot keep erythrocytes supplied with sufficient amounts of the antioxidant molecule NADPH NADPH protects cell membranes and other cellular structures from oxidation. Describe in general terms the inheritance pattern of this molecular disease. Why do you think that the antimalarial drug primaquine, which stimulates peroxide formation, results in devastating cases of hemolytic anemia in carriers of the defective gene? Does it surprise you that this genetic anomaly is commonly found in African and Mediterranean populations?arrow_forward
arrow_back_ios
SEE MORE QUESTIONS
arrow_forward_ios
Recommended textbooks for you
- Biology Today and Tomorrow without Physiology (Mi...BiologyISBN:9781305117396Author:Cecie Starr, Christine Evers, Lisa StarrPublisher:Cengage Learning
Biology Today and Tomorrow without Physiology (Mi...
Biology
ISBN:9781305117396
Author:Cecie Starr, Christine Evers, Lisa Starr
Publisher:Cengage Learning
Parasites: Protozoa (classification, structure, life cycle); Author: ATP;https://www.youtube.com/watch?v=V4iSB0_7opM;License: Standard youtube license