Biology - Study Guide
8th Edition
ISBN: 9780321501561
Author: Martha R. Taylor
Publisher: PEARSON EDUCATION (COLLEGE)
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Textbook Question
Chapter 48, Problem 18TYK
If the binding of a neurotransmitter to its receptor opens Cl− channels, what would be the effect on the postsynaptic cell? (Cl− is in higher concentration outside the cell.)
- a. It would hyperpolarize, producing an IPSP.
- b. It would depolarize and form an EPSP but probably not generate an action potential.
- c. It would initiate a signal transduction pathway as part of modulated signaling.
- d. Its membrane potential would not change because neither sodium nor potassium channels opened.
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Chapter 48 Solutions
Biology - Study Guide
Ch. 48 - Prob. 1IQCh. 48 - a. What is the principal cation inside the cell?...Ch. 48 - The following diagram shows the changes in...Ch. 48 - Prob. 4IQCh. 48 - Prob. 5IQCh. 48 - Prob. 6IQCh. 48 - Prob. 7IQCh. 48 - Develop a flowchart or diagram or write a...Ch. 48 - Prob. 1TYKCh. 48 - Prob. 2TYK
Ch. 48 - During a neurons resting state a. there are more...Ch. 48 - Which of the following contribute(s) to the...Ch. 48 - Prob. 5TYKCh. 48 - Prob. 6TYKCh. 48 - Prob. 7TYKCh. 48 - After the rapid depolarization of an action...Ch. 48 - Nodes of Ranvier are a. gaps where Schwann cells...Ch. 48 - Prob. 10TYKCh. 48 - Signal transmission is faster in myelinated axons...Ch. 48 - Which of the following statements concerning...Ch. 48 - Prob. 13TYKCh. 48 - Prob. 14TYKCh. 48 - Prob. 15TYKCh. 48 - Prob. 16TYKCh. 48 - Prob. 17TYKCh. 48 - If the binding of a neurotransmitter to its...
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- GABA is an example of an inhibitory neurotransmitter. Drugs that increase the level of GABA are used to treat diseases such as epilepsy where the signal transmission needs to be impaired. The effect of the inhibitory neurotransmitter on the postsynaptic membrane is to: Select one: a. decrease the threshold level b. hyperpolarize the postsynaptic membrane c. depolarize the postsynaptic membrane d. maintain the resting potentialarrow_forwardCharge from depolarization along the axon reaches the axon terminal, but there is a lack of neurotransmitter release. Which of the following would cause this? Group of answer choices A. Lack of Na+ in the synaptic cleft B. Lack of Ca2+ in the synaptic cleft C. Reduced amount of K+ at the axon terminal D. Lack of voltage-gated sodium channels in the axon terminalarrow_forwardThe difference between the channels along the axon and the channels in the axon terminal is that? a) axon channels are voltage-gated sodium channels and axon terminal channels are voltage-gated calcium channels b) there is no difference c) axon channels are voltage-gated sodium channels and axon terminal channels are ligand- gated sodium channelsarrow_forward
- In the hippocampus, when glutatmate is released into the synaptic cleft, but the postsynaptic cell is NOT depolarized, there is only a small postsynaptic response. Why is this the case? A. Ca++ ions block the NMDA ion channel B. Mg++ ions block the NMDA ion channel C. Glutamate is an inhibitory amino acid neurotransmitter D. There are no NMDA receptors on the postsynaptic cellarrow_forwardYou learned in this chapter that Na+/K+ active transport pumps in the plasma membrane of the axons are responsible for creating the imbalance between Na+ and K+ inside and outside of the neuron that produces the resting membrane potential. In early research studying the role of ions and the involvement of active transport of ions in neural signaling, investigators used the giant axon of a squid as a model. The diameter of a giant axon is far greater than that of a mammalian axon, which enabled researchers to isolate it easily and use it in in vitro experiments. In one early experiment, researchers investigated the active transport of Na+ out of the axon in response to the presence of cyanide. Experimentally they hooked up a section of axon to a syringe, immersed the axon in artificial seawater, introduced radioactive 22Na (as 22NaCl) into the axon, and then quantified the transport of 22Na out through the axons plasma membrane. The rate of 22Na transport out of the axon was determined by measuring the radioactivity released into the fluid sur- rounding the axon over a period of time. The Figure shows the results of the experiment. What is the effect of cyanide on Na1 transport out of the squid axon? How do the data show the effect? Source: P. C. Caldwell et al. 1960. The effects of injecting energy-rich phosphate compounds on the active transport of ions in the giant axons of Loligo. The Journal of Physiology 152:561590. Cengage Learning 2017arrow_forwardIn an experiment you place a neuron and its synaptic contacts into a medium containing no calcium ions. You stimulate the neuron causing an action potential to propagate down the axon into the axon terminal of the neuron. 3.) What is the most likely result of this experimental manipulation? A. Prolong the refractory period of the action potential B. Decrease the time required to move sodium ions out of the axon terminal C. Prevent neurotransmitter from binding to receptors on postsynaptic cells D. Enhance the voltage changes associated with the action potential E. Prevent release of neurotransmitter into the synapsearrow_forward
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