Cardiopulmonary Anatomy & Physiology
7th Edition
ISBN: 9781337794909
Author: Des Jardins, Terry.
Publisher: Cengage Learning,
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Question
Chapter 7, Problem 1CAQ4
Summary Introduction
To review:
The given blank space in the statement, “The increased blood pressure, heart rate, and respiratory rate seen in the emergency department were compensatory mechanisms activated to counteract the decreased arterial oxygenation. These mechanisms _________,” is to be filled-in.
Introduction:
Carbon monoxide is an odorless and colorless gas. The incomplete combustion of a carbon-containing fossil fuel leads to the production of this gas. It is also produced as a by-product of hemoglobin degradation inside the human body.
Please refer to Clinical Application Case-1 for further information.
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Cardiopulmonary Anatomy & Physiology
Ch. 7 - 1. During acute alveolar hypoventilation, the...Ch. 7 - The bulk of the CO2 produced in the cells is...Ch. 7 - 3. During acute alveolar hyperventilation, the...Ch. 7 - 4. In chronic hypoventilation, renal compensation...Ch. 7 - Prob. 5RQCh. 7 - Prob. 6RQCh. 7 - Metabolic alkalosis can develop from 1....Ch. 7 - Prob. 8RQCh. 7 - Prob. 9RQCh. 7 - Prob. 10RQ
Ch. 7 - Prob. 11RQCh. 7 - 12. Metabolic acidosis caused by fixed acids is...Ch. 7 - Prob. 13RQCh. 7 - Prob. 14RQCh. 7 - Prob. 15RQCh. 7 - Prob. 1CAQ1Ch. 7 - Prob. 1CAQ2Ch. 7 - Prob. 1CAQ3Ch. 7 - Prob. 1CAQ4Ch. 7 - Prob. 1CAQ5Ch. 7 - Case 1
6. The nomogram verified that the sole...Ch. 7 - Case 2 The fact that the initial pH (7.10) and...Ch. 7 - Prob. 2CAQ2Ch. 7 - Prob. 2CAQ3Ch. 7 - Case 2
4. If sodium bicarbonate had initially been...
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- Case 1 As a result of the severe left heart failure and increased pulmonary blood pressure in the case, fluid moved out of the pulmonary capillaries and into the extracapillary spaces. The pathologic process caused the thickness of the alveolar-capillary membrane to______arrow_forwardCase 1 The pathophysiologic process that developed in this case was corrected with______. During each breath, the patient's chest wall (caved inward_____________; moved outward __________) and then returned to normal ___________at the end of each expiration.arrow_forwardCase 1 2. As a result of the previously described condition, the patient's right lung__________, which in turn caused an acute (decreased ______; increased_____) lung compliance condition.arrow_forward
- Case 2 Because of the lung hyperinflation described in question 1, the patient was generating (small_________; large_____) pleural pressure changes with (little or no ________ moderate to large_________) volume changes.arrow_forwardCase 1 3. Why did the patient's PCWP, Cl, SVR, and urine output all improve after the administration of nitroprusside?arrow_forwardCase 2 The patient's fatigue and respiratory rate of 6 breaths/min further caused the ventilation- perfusion ratio to (______rise;________ fall).arrow_forward
- Case 2 As fluid accumulated in the patients alveoli, the diffusion of oxygen into the pulmonary capillaries decreased. This was verified by the __________________________________.arrow_forwardCase 1 When the patient was in the emergency department what pulmonary function measurement served as an important clinical indicator of the severity of the patient's asthma attack?arrow_forwardCase 1 6. The nomogram verified that the sole cause of the elevated pH was due to the _____________________________________________________________________________.arrow_forward
- Case 1 As the patients blood pressure decreased from the loss of blood, neural impulses transmitted from ________________ to the vasomotor and cardiac centers in the medulla (decreased ________; increased _______).arrow_forwardCase 2 1. This patient demonstrated both obstructive and restrictive lung disorders. During the first part of the case, which pulmonary function studies verified that the patient had an obstructive pulmonary disorder? _______________________ ________________________arrow_forward
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