Campbell Biology in Focus
3rd Edition
ISBN: 9780134710679
Author: Lisa A. Urry, Michael L. Cain, Steven A. Wasserman, Peter V. Minorsky, Rebecca Orr
Publisher: PEARSON
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Textbook Question
Chapter 31.1, Problem 2CC
WHAT IF? If a plant has the double mutation ctr and ein, what is its triple-response
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Chapter 31 Solutions
Campbell Biology in Focus
Ch. 31.1 - Prob. 1CCCh. 31.1 - WHAT IF? If a plant has the double mutation ctr...Ch. 31.1 - Prob. 3CCCh. 31.2 - If an enzyme in field-grown soybean leaves is most...Ch. 31.2 - WHAT IF? If a plant flowers in a controlled...Ch. 31.2 - MAKE CONNECTIONS Plants detect the quality of...Ch. 31.3 - Prob. 1CCCh. 31.3 - Prob. 2CCCh. 31.3 - Prob. 3CCCh. 31.4 - Prob. 1CC
Ch. 31.4 - Chewing insects mechanically damage plants and...Ch. 31.4 - Prob. 3CCCh. 31 - The hormone that helps plants respond to drought...Ch. 31 - Prob. 2TYUCh. 31 - Prob. 3TYUCh. 31 - Prob. 4TYUCh. 31 - Prob. 5TYUCh. 31 - Prob. 6TYUCh. 31 - DRAW IT Indicate the response of Arabidopsis to...Ch. 31 - Prob. 8TYUCh. 31 - FOCUS ON EVOLUTION As a general rule,...Ch. 31 - FOCUS ON INTERACTIONS In a short essay (100-150...Ch. 31 - Prob. 11TYU
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- Did your mutation actually disrupt binding? Please choose the best explanation based on what you calculated above. No, because the affinity for the mutant is higher than the affinity for the wild type. Yes, because the affinity for the mutant is lower than the affinity for the wild type. No, because the affinity for the mutant is lower than the affinity for the wild type. Yes, because the affinity for the mutant is higher than the affinity for the wild type.arrow_forwardWHAT IF? Suppose X-rays caused a sequence changein the TATA box of a particular gene’s promoter. Howwould that affect transcription of the gene? (SeeFigure 17.9.)arrow_forwardWHAT IF? What would happen if a mutation in the myoD gene resulted in the production of an alteredMyoD protein that could not activate the myoD gene?arrow_forward
- WHAT IF? Suggest possible functions of the genes whose sequenceor expression was altered as the experimental populations evolved in thelow-glucose environment.arrow_forwardCreate workplan step by step on regenerating a BT maize plant selecting using phosphinothricin herbicide. Mention uses of agrobacterium , somatic embryogenesis, and construction of engineered TDNA for your workplanarrow_forwardList and explain four loop holes as of March 2020, that do not require the USDA to regulate gene edited plants?arrow_forward
- WHAT IF? Since the results support a role for mouse FOXP2 invocalization, you might wonder whether the human FOXP2 protein is akey regulator of speech. If you were given the amino acid sequences ofwild-type and mutant human FOXP2 proteins and the wild-type chimpanzee FOXP2 protein, how would you investigate this question? What furtherclues could you obtain by comparing these sequences to that of the mouseFOXP2 protein?arrow_forwardWHAT IF? A certain mutation in E. coli changes the lacoperator so that the active repressor cannot bind. Howwould this affect the cell’s production of β-galactosidase?arrow_forwardWhat is mutation? Explain the significance of mutation in plant breeding. Give an example of a disease resistant variety of cultivated plant induced by mutation.arrow_forward
- What are the for phytoglycogen confirmation properties in maize can that can determine the extracted glycogen in the plants. Present it nicely.arrow_forwardLet’s suppose a researcher was interested in the effects of mutationson the expression of a protein-encoding gene for a proteinthat is 472 amino acids in length. This protein is expressed in leafcells of Arabidopsis thaliana. It has a molecular mass of approximately56,640 Da. Make a drawing that shows the expected resultsof a Western blot using proteins isolated from the leaf cells thatwere obtained from the following plants:Lane 1. A plant homozygous for a nonmutant geneLane 2. A plant homozygous for a deletion that removes the promoterfor this geneLane 3. A heterozygous plant in which one gene is nonmutant andthe other gene has a mutation that introduces an early stop codon atcodon 112Lane 4. A plant homozygous for a mutation that introduces anearly stop codon at codon 112Lane 5. A plant homozygous for a mutation that changes codon108 from a phenylalanine codon into a leucine codonarrow_forwardA. The genes CLV3, CLV1/2 and WUS are involved in maintaining the size of shoot apical meristem. Diagram the interactions among these gene products, using ‘—>’ for positive interactions (i.e. activation) and ‘ —|’ for negative interactions (i.e. repression). B. What phenotype would you expect for a double mutant clv3 wus? And why?arrow_forward
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