Campbell Biology in Focus, Books a la Carte Edition; Modified Mastering Biology with Pearson eText - ValuePack Access Card - for Campbell Biology in Focus (2nd Edition)
2nd Edition
ISBN: 9780134433769
Author: Lisa A. Urry, Michael L. Cain, Steven A. Wasserman
Publisher: PEARSON
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Question
Chapter 35.3, Problem 2CC
Summary Introduction
To determine:
The cells and functions that would be deficient, if child were born without a thymus. Determine the functions do cell-surface antigen receptors fulfill for memory B-cells.
Introduction:
The thymus is a gland which is responsible for the development of T-cells which are one of the important types of white blood cells and play an important role in the immune system.
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Explain the "antigen recognition by cells of adaptive immunity" in a simple way.
Relate it with Class I,II,III MHC molecules/genes.
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Describe the mechanisms for antigen processing and presentation via MHC class I and class
II molecules to T lymphocytes. How do these different pathways lead to appropriate defenses
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Class II MHC proteins display what kind of antigens? What class of T cell recognizes antigens bound to class II MHC? What types of cells display these proteins?
Chapter 35 Solutions
Campbell Biology in Focus, Books a la Carte Edition; Modified Mastering Biology with Pearson eText - ValuePack Access Card - for Campbell Biology in Focus (2nd Edition)
Ch. 35.1 - Pus is both a sign of infection and an indicator...Ch. 35.1 - MAKE CONNECTIONS How do the molecules that...Ch. 35.1 - Prob. 3CCCh. 35.2 - Prob. 1CCCh. 35.2 - Explain how memory cells strengthen the immune...Ch. 35.2 - WHAT IF? If both copies of a light-chain gene and...Ch. 35.3 - Prob. 1CCCh. 35.3 - Prob. 2CCCh. 35.3 - Prob. 3CCCh. 35 - Prob. 1TYU
Ch. 35 - Prob. 2TYUCh. 35 - Prob. 3TYUCh. 35 - Prob. 4TYUCh. 35 - Prob. 5TYUCh. 35 - Prob. 6TYUCh. 35 - Prob. 7TYUCh. 35 - DRAW IT Consider a pencil-shaped protein with two...Ch. 35 - MAKE CONNECTIONS Contrast clonal selection with...Ch. 35 - Prob. 10TYUCh. 35 - FOCUS ON EVOLUTION Describe one invertebrate...Ch. 35 - Prob. 12TYUCh. 35 - Prob. 13TYU
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- B lymphocytes, the precursors of plasma cells, are triggered to proliferate by the binding of multivalent antigens to receptors on their surfaces. The cell-surface receptors are transmembrane immunoglobulins. Univalent antigens, in contrast, do not activate B cells. (a) What do these findings reveal about the mechanism of B-cell activation? (b) How might antibodies be used to activate B cells?arrow_forwardAbout Antigen presenting cells, it is CORRECT to affirm that: a). Macrophages, B lymphocytes and dendritic cells are APCs. meaning they express MHC-ll. b). Only APCs present MHC-11. c). Macrophages,B lymphocytes and dendritic cells are phagocytes, but not APCs.d). All the non-APC cells of our body present MHC-1.arrow_forwardExplain why are B cells known as Antigen Presenting cells and what happens after antigen presentation?arrow_forward
- Sketch a B cell antigen receptor. Label the V and C regions of the light and heavy chains. Label the antigenbinding sites, disulfide bridges, and transmembrane region. Where are these features located relative to the V and C regions?arrow_forwardWhere are major histocompatibility complex-I (MHC-I) molecules located in the human body (cells)? Briefly describe how these surface markers present antigens to other cells. (Make sure to include where the antigen originates and what type of T-cell interacts with MHC-I antigen presentation.)arrow_forwardWhich type of MHC-class molecule is found on all nucleated cells and is used to communicate with cytotoxic T-lymphocytes? Which classes are displayed on APCs, and which class is used specifically to communicate with (a) helper T-lymphocytes and (b) cytotoxic T-lymphocytes?arrow_forward
- b) Briefly describe the stages in T cell development in the thymus and describe the mechanisms that lead to self- tolerance. Explain one example of any kind of defect that leads to failure of central tolerance and cause an autoimmune disease?arrow_forwardOutline the typical immunophenotypic features of peripheral B cells in: (i) X-linked Agammaglobulinaemia. (ii) CD40L (CD154) deficiency.arrow_forwardWould the primary response or the secondary response be more affected by a lack of memory B cells for a particular antigen?arrow_forward
- A flu-infected cell has down-regulated its MHC class I expression. The lymphocytes below comes to the site and interacts with the cell. How does the downregulation of MHC class I affect these ability of lymphocytes to kill the infected cell? a) influenza-specific T cell b) NK cell c) herpes-specific T cell Indicate whether the possibility increases, decreases or is unchanged for each cell.arrow_forwardExplain the typical immunophenotypic features of peripheral B cells in: (i) X-linked Agammaglobulinaemia (ii) CD40L (CD154) deficiency (iii) B-Chronic Lymphocytic Leukaemia. wwarrow_forwardExplain the typical immunophenotypic features of peripheral B cells in: (i) X-linked Agammaglobulinaemia (ii) CD40L (CD154) deficiency (iii) B-Chronic Lymphocytic Leukaemia.arrow_forward
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