Lab 3 Assignment Ishmeet

Question in photo

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Correct answers already provided! Please don't just tell me the answers bc I know them already. Help me with my own question. I get everything else in this problem other than the third option: Introduce the mutant human HD allele as a transgene into the mouse genome with transgene integration anywhere in the mouse genome. Why is the first question (left) okay with introducing mutant human HD allele and the second question (right) is not? I heard that introducing allele without using CRISPR-Cas9 is very rare and difficult. If so, how does it work in the first problem (Hungtinton's chorea)?

Transposons are useful as mutagens because they act asmolecular tags for genomic DNA sequences that can beidentified rapidly by inverse _______

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Please answer it asap.

i need the answer quickly

(E) identify the source of DNA material Questions 21-23 A culture of white-eyed fruit flies (Drosophila melanogaster) was maintained for many generations. Females from the stock white-eyed culture were crossed with red-eyed (wild-type) males. The F1 females were crossed with the white-eyed males from the original culture. The resulting phenotypes of the progeny are summarized below. Parental Generation Cross F1 Generation (at least 500 flies) 100% of females are red-eyed 100% of males are white-eyed White-eyed females x red-eyed males F1 Generation Cross F2 Generation (at least 500 flies) Fl red-eyed females x white-eyed males 50% of females are red-eyed and 50% are white- eyed 50% of males are red-eyed and 50% are white-eyed 21. The best explanation for the red-eyed F1 females is (A) mutation (B) culture contamination (C) dominance (D) multiple loci (E) sex-influenced traits 22. There are white-eyed females in the F2 generation because (A) white is a dominant allele (B) the white…

Could I get some assistance with this please.

Leah

The Meeting Sarah stared blankly at the blue paisley wallpaper. Her husband Mike sat by her side, bending and unbending a small paper clip. “Sarah and Michael, it’s good to meet you,” welcomed the genetic counselor, as she entered the room. “I apologize for being late, but I was just meeting with another couple. Let’s see, you’d like to have a child, but you’re concerned because of your family history of cystic fibrosis.” “Yes,” Sarah replied softly. “Mike and I met at a CF support group meeting a few years ago. He had a younger brother who died of cystic fibrosis, and I had a younger sister. We saw the painful lives they had—difficulty breathing, the constant respiratory infections. Although the treatments for CF are better now, we just don’t know if we can…” she trailed off. “I can certainly understand your concern,” the genetic counselor responded sympathetically. “That’s where I hope to help, by providing as much information and advice as I can. I’m glad that you came to see me…

please give answer asap

GQ4

Need help with parts a,b and c, please and thank you!

Why is karyotyping significant in understanding chromosomal abnormalities? Short essay only thanks please the main answer

patibility Mode] UE Layout References Mailings Review View A A Aa vA 前、一间刷 一| T AaBbCcDdE AaBE x A Av X, Normal No Spacing Headi co Edit and Save Start your free one month trial of Microsoft 365 or sign in to activate an existing subscription. 9. In human beings, the gene for red-green colorblindness (r) is sex-linked and recessive to its allele for normal vision (R), while the gene for freckles (F) is autosomal and dominant over its allele for nonfreckled (f). A nonfreckled, normal-visioned woman whose father was freckled and colorblind, marries a freckled, colorblind man whose mother was nonfreckled. a. What is the genotype of the woman's father? b. What is the probability that the couple's first child will be a non-freckled, normal visioned girl c. What is the probability that the first two children born to the couple will be freckled and colorblind girls? d. What is the probability that the first child born to the couple will either be a freckled, colorblind boy or a non-freckled,…

could you please help with part d on this problem thank you!

1_30*_SP23 - General Biology I (for majors)/1 of us page The anticodon sequence created from the following DNA: TACGGGGCTGAGATT F1 Select one: O a. Tyr-Gly-Ala-Glu-lle O b. AUGCCCCGACUCUAA c. UACGGGGCUGAGAUU O d. Met-Pro-Arg-Leu-STOP F2 # 80 F3 $ 000 000 F4 % F5 MacBook Air F6 & r F7 DII F8

please answer and explain thoroughly!!!

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GQ2

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Wa FILE PAGE 3 OF 3 HH HOME INSERT DESIGN PAGE LAYOUT 198 WORDS Q Type here to search KARYOTYPING ANALYSIS lab activity (1) - Microsoft Word REVIEW VIEW FOXIT READER PDF REFERENCES MAILINGS 3.Cut out the following chromosomes (if you can't have it printed, you may draw them but please make sure you get the exact size and shape of the chromosome ), arrange them in pairs, and compare with the normal human karyotype. You can scan your output (or take pictures) and submit the e copy (comment it below this post ) together with the discussion on the genetic disorder involved. Discuss your answer briefly. 30°C Haze □4) ? Microsoft account 7:49 PM 5/1/2022 95%

This is not a writing assignment. The information is given. I’m confused for this since there are 3 parts associated with the question.

genetics question for Icke P. Jenny, like her mother, is heterozygous for both X-linked traits. Her father suffered from bothhypophosphatemia and muscular dystrophy. Jenny’s fiancé, Mark has a mother suffering from musculardystrophy despite having normal bone conditions. His father has been blessed with strong bones andmuscles but suffered from amelogenesis imperfecta. a. Give the complete genotypes of the following individuals:  Jenny, Jenny's mother, Jenny's father, Mark, Mark's mother, Mark's father. b. Should Jenny and Mark eventually marry, what is the probability that they will have: a child with muscular dystrophy, a child with amelogenesis imperfecta, a son with hypophosphatemia, a daughter with muscular dystrophy, a child with the same genotype as Jenny, a child with the same genotype as Mark

Question in photo

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PLease help, double and triple check your answers, im using this to study, these questions are NOT graded they are PRACTICE problems

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Can I get an explanation for 1a, 1d, 1e, 2c, 2d and the calculations for the level of interference please.

40 Below is a pedigree of a family, some of whom have the autosomal dominant condition Huntington's disease. Affected individuals are indicated by a dark square or circle. The male in generation I (indicated by the arrow) is heterozygous for the Huntington's disease mutation. The following two questions relate to this pedigree. Generation II II If H represents the disease allele, and h the wild type allele, what is the genotype of the individual indicated by the *? Select one alternative: O hh O Hh O hH O HH