CAMPBELL MASTERING BIOLOGY ACCESS>I<
18th Edition
ISBN: 9781323766286
Author: Pearson
Publisher: PEARSON
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Chapter 16.2, Problem 3CC
WHAT IF? If you were a doctor who wanted to use iPS cells to treat a patient with severe type 1 diabetes, what new technique would have to be developed?
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WHAT IF? If you measured ADH levels in patients with ADH receptormutations and in patients with aquaporin mutations, what would you expectto find, compared with wild-type subjects?
WHAT IF? Patients with diseases such as heart disease or Alzheimer’scould have their own skin cells reprogrammed to become iPS cells. Once procedures have been developed for converting iPS cells into heart or nervoussystem cells, the patients’ own iPS cells might be used to treat their disease.When organs are transplanted from a donor to a diseased recipient, the recipient’s immune system may reject the transplant, a dangerous condition. Wouldusing iPS cells be expected to carry the same risk? Why or why not? Giventhat these cells are actively dividing, undifferentiated cells, what risks mightthis procedure carry?
WHAT IF? If two cells have different scaffolding proteins,explain how they might behave differently in response tothe same signaling molecule.
Chapter 16 Solutions
CAMPBELL MASTERING BIOLOGY ACCESS>I<
Ch. 16.1 - Prob. 1CCCh. 16.1 - MAKE CONNECTIONS Explain how the signaling...Ch. 16.1 - How do fruit fly maternal effect genes determine...Ch. 16.2 - Deitys egg donor and surrogate mother were...Ch. 16.2 - WHAT IF? If you were a doctor who wanted to use...Ch. 16.3 - Prob. 1CCCh. 16.3 - Prob. 2CCCh. 16.3 - Prob. 3CCCh. 16 - Muscle cells differ from nerve cells mainly...Ch. 16 - Cell differentiation always involves A. the...
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- . WHAT IF? Suppose a cell’s membrane potential shiftsfrom -70 mV to -50 mV. What changes in the cell’spermeability to K+or Na+could cause such a shift?arrow_forwardCan I get help? Draw a schematic depicting how the cellular GTP:GDP concentration ratio facilitates the mechanism whereby GEFs induce G proteins to exchange GTP for GDP.arrow_forwardWHAT IF? If apoptosis occurred when it should not,what types of protein defects might be the cause? Whattypes could result in apoptosis not occurring when itshould?arrow_forward
- Fil in the blanks 1. A resting muscle fiber or neuron has an internal charge that is A. positive, B. negative, or C. neutral compared to the extracellular environment. This resting membrane charge is maintained by the A. calcium ion channels, B. sodium voltage gates, C. potassium voltage gates, or D. sodium/potassium pumps? . When a muscle fiber is at resting membrane potential there is a(an) "higher", "equal", OR "lower" concentration of sodium inside the cell compared to outside the cell and a(an) "higher", "lower", OR "equal" concentration of potassium inside the cell compared to outside.arrow_forwardCell signaling. The ER doctors gave a patient an injection of epinephrine to increase his heart rate and blood pressure. The heart rate and blood pressure went up but not enough. The second dose of the same drug seemed not to have an effect. What do you think may be a possible explanation for this result?arrow_forwardWHAT IF? Some membrane proteins diffuse faster in the plasma membrane when the cytoskeleton or the extracellular matrix is artificially removed than when cells are unperturbed. Explain why.arrow_forward
- As1 You are using a cell-culture model to screen for agonists that you wish to develop for the clinic. What is a risk that must be considered if the response you are measuring in order to compare your potential drugs is a long way from the ligand-binding event in the cellular cascade?arrow_forward1. What is the primary intracellular signaling factor activated during resistance exercise? What is an ultimate outcome of activating this factor? 2.What is the primary intracellular signaling factor activated during aerobic exercise? What is an ultimate outcome of activating this factor?arrow_forwardWHAT IF? Suppose the experiment had shown that class I mutants could grow only in MM supplementedby ornithine or arginine and that class II mutants could grow in MM supplemented by citrulline, ornithine,or arginine. What conclusions would the researchers have drawn from those results regarding the biochemicalpathway and the defect in class I and class II mutants?arrow_forward
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