Principles of Biology
2nd Edition
ISBN: 9781259875120
Author: Robert Brooker, Eric P. Widmaier Dr., Linda Graham Dr. Ph.D., Peter Stiling Dr. Ph.D.
Publisher: McGraw-Hill Education
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Chapter 35.2, Problem 1BC
Summary Introduction
To analyze:
The source from where a person can learn about voltage-gated Ca2+ channels and their role in cell-to-cell communication in animals.
Introduction:
The vertebral skeletal muscle cells generate and propagate action potentials in response to stimulus. It increases the concentration of cytosolic calcium ions that triggers the contraction of a muscle fiber.
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At which point in the graph do voltage-gated potassium channels open? (Base your answer to this question on the graph below depicting an action potential.)
A
B
C
D
E
Choose two false statements and explain why?
Toxin causes voltage-gated sodium channels to open at a lower voltage
toxin slows down the inactivation of sodium channels
toxin causes ligand-gated sodium channels to open
toxin causes voltage-gated potassium channels to
open faster
Experimenters injected bark scorpion venom into mouse neurons and measured how many action potentials were generated after the venom was introduced.
Is it possible that the venom could be affecting the activity of a voltage-gated potassium channel?
What effect could the venom have on a voltage-gated potassium channel to produce this result?
Chapter 35 Solutions
Principles of Biology
Ch. 35.1 - Prob. 1CCCh. 35.1 - Prob. 1TYKCh. 35.1 - Prob. 2TYKCh. 35.2 - Prob. 1CCCh. 35.2 - Rigor mortis is the condition that occurs shortly...Ch. 35.2 - Prob. 1BCCh. 35.2 - Prob. 3CCCh. 35.2 - Prob. 2BCCh. 35.2 - Prob. 1TYKCh. 35.2 - Prob. 2TYK
Ch. 35.3 - Prob. 1TYKCh. 35.4 - Prob. 1TYKCh. 35.4 - Prob. 2TYKCh. 35 - Prob. 1TYCh. 35 - Which, if any, of the following is not a function...Ch. 35 - Prob. 3TYCh. 35 - Prob. 4TYCh. 35 - Prob. 5TYCh. 35 - During cross-bridge cycling, binds to _________,....Ch. 35 - Prob. 7TYCh. 35 - Prob. 8TYCh. 35 - Prob. 9TYCh. 35 - Prob. 10TYCh. 35 - Prob. 1CCQCh. 35 - Prob. 2CCQCh. 35 - Prob. 3CCQCh. 35 - Discuss the structure and function of the two...Ch. 35 - Prob. 2CBQ
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- part 1: What is the membrane topology of typical voltage-gated ion channels and how are they sensitive to voltage? Part 2: Explain the differences in the relative selectivity of voltage-gated Na and K channels.arrow_forwardWhat proteins are responsible for the decrease of membrane potential in section 3? opening of voltage gated sodium channels opening of voltage gated potassium channels potassium leak channels opening of ligand gated sodium channels Na+/ K+/atpasearrow_forwardDendrotoxins, produced by the mamba snakes (Dendroaspis), are inhibitors of the voltage-gated K+ channels. What phase of the action potential would this toxin affect? How would it affect ion permeability during this phase? How would ion movement be affected?arrow_forward
- You learned in this chapter that Na+/K+ active transport pumps in the plasma membrane of the axons are responsible for creating the imbalance between Na+ and K+ inside and outside of the neuron that produces the resting membrane potential. In early research studying the role of ions and the involvement of active transport of ions in neural signaling, investigators used the giant axon of a squid as a model. The diameter of a giant axon is far greater than that of a mammalian axon, which enabled researchers to isolate it easily and use it in in vitro experiments. In one early experiment, researchers investigated the active transport of Na+ out of the axon in response to the presence of cyanide. Experimentally they hooked up a section of axon to a syringe, immersed the axon in artificial seawater, introduced radioactive 22Na (as 22NaCl) into the axon, and then quantified the transport of 22Na out through the axons plasma membrane. The rate of 22Na transport out of the axon was determined by measuring the radioactivity released into the fluid sur- rounding the axon over a period of time. The Figure shows the results of the experiment. What is the effect of cyanide on Na1 transport out of the squid axon? How do the data show the effect? Source: P. C. Caldwell et al. 1960. The effects of injecting energy-rich phosphate compounds on the active transport of ions in the giant axons of Loligo. The Journal of Physiology 152:561590. Cengage Learning 2017arrow_forwardWatch this video (http://openstaxcollege.org/l/neurotrans) to learn about the release of a neurotransmitter. The action potential reaches the end of the axon, called the axon terminal, and a chemical signal is released to tell the target cell to do something, either initiate a new action potential, or to suppress that activity. In a very short space, the electrical signal of the action potential is changed into the chemical signal of a neurotransmitter, and then back to electrical changes in the target cell membrane. What is the importance of voltage-gated calcium channels in the release of neurotransmitters?arrow_forwardWhat membrane protein of the neuron is responsible for briefly increasing the Na+ permeability of the membrane during the rising phase of the nerve impulse? a. the sodium-potassium pump b. the voltage gated potassium channel c. the voltage gated sodium channel d. the stimulus gated sodium channel e. the ligand gated ion channelarrow_forward
- When neurotransmitters contact a muscle cell's receptor proteins, those receptors change their shape and let ions go through the membrane. The receptors are an example of: A. voltage sensitive channels B. tension-sensitive channels C. ligand-activated channelsarrow_forwardAmplification of a chemical signal occurs when? a. phosphorylation of proteins by protein kinase receptors is balanced by the immediate removal of phosphate groups by phosphatases b. a receptor in the plasma membrane activates several “first messenger” molecules while a signal molecule is bound to it c. a cAMP molecule activates one protein kinase molecule before being converted to AMP d. a receptor activates a single first messenger and a single second messengerarrow_forwardA neuronal precursor in a fly embryo expresses which molecule to signal neighboring cells not to become neurons (Choices are shh, delta, noggin, notch)arrow_forward
- Give me an example of a voltage gated ion in action in the body.arrow_forwardWhat would happen to an insect if you treated an insect with a chemical that inhibits acetylcholinesterase? 1) K+ channels get stuck open and the neuron stays perminantly hyperpolarized 2) Acetylcholine would continue to activiate Ca++ channels until death by exhaustion 3) Acetylcholine would continue to activiate Na+ channels until death by exhaustionarrow_forwardAcetylcholine is an excitatory neurotransmitter that is responsible for initiating muscle movement. Consider the scenario where a mouse was bitten by a snake that injected venom into their circulatory system. The venom has an inhibitory effect on the reuptake of acetylcholine from the synaptic cleft. Explain how the mouse’s body would respond to the venom as well as the venoms effect at the cellular level.arrow_forward
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