Campbell Biology In Focus
2nd Edition
ISBN: 9780134203072
Author: Lisa A. Urry, Michael L. Cain, Steven A. Wasserman, Peter V. Minorsky, Jane B. Reece
Publisher: PEARSON
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Question
Chapter 35.3, Problem 2CC
Summary Introduction
To determine:
The cells and functions that would be deficient, if child were born without a thymus. Determine the functions do cell-surface antigen receptors fulfill for memory B-cells.
Introduction:
The thymus is a gland which is responsible for the development of T-cells which are one of the important types of white blood cells and play an important role in the immune system.
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Explain the "antigen recognition by cells of adaptive immunity" in a simple way.
Relate it with Class I,II,III MHC molecules/genes.
Thank you
NOTE: Bullet form
About Antigen presenting cells, it is CORRECT to affirm that:
a). Macrophages, B lymphocytes and dendritic cells are APCs. meaning they express MHC-ll. b). Only APCs present MHC-11. c). Macrophages,B lymphocytes and dendritic cells are phagocytes, but not APCs.d). All the non-APC cells of our body present MHC-1.
Describe the mechanisms for antigen processing and presentation via MHC class I and class
II molecules to T lymphocytes. How do these different pathways lead to appropriate defenses
against different types of microorganisms?
Chapter 35 Solutions
Campbell Biology In Focus
Ch. 35.1 - Pus is both a sign of infection and an indicator...Ch. 35.1 - MAKE CONNECTIONS How do the molecules that...Ch. 35.1 - Prob. 3CCCh. 35.2 - Prob. 1CCCh. 35.2 - Explain how memory cells strengthen the immune...Ch. 35.2 - WHAT IF? If both copies of a light-chain gene and...Ch. 35.3 - Prob. 1CCCh. 35.3 - Prob. 2CCCh. 35.3 - Prob. 3CCCh. 35 - Prob. 1TYU
Ch. 35 - Prob. 2TYUCh. 35 - Prob. 3TYUCh. 35 - Prob. 4TYUCh. 35 - Prob. 5TYUCh. 35 - Prob. 6TYUCh. 35 - Prob. 7TYUCh. 35 - DRAW IT Consider a pencil-shaped protein with two...Ch. 35 - MAKE CONNECTIONS Contrast clonal selection with...Ch. 35 - Prob. 10TYUCh. 35 - FOCUS ON EVOLUTION Describe one invertebrate...Ch. 35 - Prob. 12TYUCh. 35 - Prob. 13TYU
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Similar questions
- B lymphocytes, the precursors of plasma cells, are triggered to proliferate by the binding of multivalent antigens to receptors on their surfaces. The cellsurface receptors are transmembrane immunoglobulins. Univalent antigens, in contrast, do not activate B cells. (a) What do these findings reveal about the mechanism of B-cell activation? (b) How might antibodies be used to activate B cells?arrow_forwardClass II MHC proteins display what kind of antigens? What class of T cell recognizes antigens bound to class II MHC? What types of cells display these proteins?arrow_forwardSketch a B cell antigen receptor. Label the V and C regions of the light and heavy chains. Label the antigenbinding sites, disulfide bridges, and transmembrane region. Where are these features located relative to the V and C regions?arrow_forward
- Explain why are B cells known as Antigen Presenting cells and what happens after antigen presentation?arrow_forwardWhere are major histocompatibility complex-I (MHC-I) molecules located in the human body (cells)? Briefly describe how these surface markers present antigens to other cells. (Make sure to include where the antigen originates and what type of T-cell interacts with MHC-I antigen presentation.)arrow_forwardWhich type of MHC-class molecule is found on all nucleated cells and is used to communicate with cytotoxic T-lymphocytes? Which classes are displayed on APCs, and which class is used specifically to communicate with (a) helper T-lymphocytes and (b) cytotoxic T-lymphocytes?arrow_forward
- b) Briefly describe the stages in T cell development in the thymus and describe the mechanisms that lead to self- tolerance. Explain one example of any kind of defect that leads to failure of central tolerance and cause an autoimmune disease?arrow_forwardWould the primary response or the secondary response be more affected by a lack of memory B cells for a particular antigen?arrow_forwardA flu-infected cell has down-regulated its MHC class I expression. The lymphocytes below comes to the site and interacts with the cell. How does the downregulation of MHC class I affect these ability of lymphocytes to kill the infected cell? a) influenza-specific T cell b) NK cell c) herpes-specific T cell Indicate whether the possibility increases, decreases or is unchanged for each cell.arrow_forward
- Outline the typical immunophenotypic features of peripheral B cells in: (i) X-linked Agammaglobulinaemia. (ii) CD40L (CD154) deficiency.arrow_forwardExplain the typical immunophenotypic features of peripheral B cells in: (i) X-linked Agammaglobulinaemia (ii) CD40L (CD154) deficiency (iii) B-Chronic Lymphocytic Leukaemia. wwarrow_forwardExplain the typical immunophenotypic features of peripheral B cells in: (i) X-linked Agammaglobulinaemia (ii) CD40L (CD154) deficiency (iii) B-Chronic Lymphocytic Leukaemia.arrow_forward
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