Study Guide for Campbell Biology
Study Guide for Campbell Biology
11th Edition
ISBN: 9780134443775
Author: Lisa A. Urry, Michael L. Cain, Steven A. Wasserman, Peter V. Minorsky, Jane B. Reece, Martha R. Taylor, Michael A. Pollock
Publisher: PEARSON
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Chapter 48, Problem 12TYK

Which of the following statements concerning chemical synapses is not true?

  1. a. Synaptic terminals at the ends of branching axons contain synaptic vesicles, which enclose the neurotransmitter.
  2. b. The influx of sodium when an action potential reaches the presynaptic membrane causes synaptic vesicles to release their neurotransmitter into the cleft.
  3. c. The binding of neurotransmitter to receptors on the postsynaptic membrane usually opens (or sometimes closes) ligand-gated ion channels.
  4. d. Neurotransmitter is often rapidly degraded in the synaptic cleft.
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Which of the following is the role of axonal Calcium (Ca2+) channels in neuronal communication at the synapse? a) they inhibit the action potential causing it to stop b) Calcium channels have no role in this process c) by allowing calcium to exit the cell, they promote the propagation of action potential across the synapse by creating depolarization d) by blocking calcium entrance, they mediate the contraction of presynaptic axon to facilitate movement e) when they open, calcium enters the presynaptic terminal, promoting fusion of neurotransmitter vesicles with presynaptic membrane, and release of neurotransmitters to the synaptic cleft
What determines the effect of neurotransmitter release on the post-synaptic neurons?  a. the size of the action potential. b. the post-synaptic receptors c. the neurotransmitter contained in the synaptic vesicles. d. the concentration of calcium ions reached in presynaptic terminals. e. the size of the synaptic vesicles.
Charge from depolarization along the axon reaches the axon terminal, but there is a lack of neurotransmitter release. Which of the following would cause this? Group of answer choices A. Lack of Na+ in the synaptic cleft B. Lack of Ca2+ in the synaptic cleft C. Reduced amount of K+ at the axon terminal D. Lack of voltage-gated sodium channels in the axon terminal
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