Study Guide for Campbell Biology
11th Edition
ISBN: 9780134443775
Author: Lisa A. Urry, Michael L. Cain, Steven A. Wasserman, Peter V. Minorsky, Jane B. Reece, Martha R. Taylor, Michael A. Pollock
Publisher: PEARSON
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Textbook Question
Chapter 48, Problem 9TYK
Nodes of Ranvier are
- a. gaps where Schwann cells abut and action potentials are generated.
- b. neurotransmitter-containing vesicles located in the synaptic terminals.
- c. the parts of neurons where action potentials are initiated.
- d. clusters of receptor proteins located on the postsynaptic membrane.
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Students have asked these similar questions
What determines the effect of neurotransmitter release on the post-synaptic neurons?
a. the size of the action potential.
b. the post-synaptic receptors
c. the neurotransmitter contained in the synaptic vesicles.
d. the concentration of calcium ions reached in presynaptic terminals.
e. the size of the synaptic vesicles.
Post synaptic neurons membrane potentials are less negative than resting membrane potentials when this occur
A. Creates a new set point for the resting membrane potential
B. Has a lower propensity to produce an action potential
C. Reacts by generating action potential
D. Higher chance of reaching the potential threshold
A patch-clamp device is used to
a.
Study the properties of individual neurotransmitters
b.
Study the properties of individual membrane channels
c.
Measure the strength of an electrochemical gradient
d.
Infuse different kinds of ions into an axon
Chapter 48 Solutions
Study Guide for Campbell Biology
Ch. 48 - Prob. 1IQCh. 48 - a. What is the principal cation inside the cell?...Ch. 48 - The following diagram shows the changes in...Ch. 48 - Prob. 4IQCh. 48 - Prob. 5IQCh. 48 - Prob. 6IQCh. 48 - Prob. 7IQCh. 48 - Develop a flowchart or diagram or write a...Ch. 48 - Prob. 1TYKCh. 48 - Prob. 2TYK
Ch. 48 - During a neurons resting state a. there are more...Ch. 48 - Which of the following contribute(s) to the...Ch. 48 - Prob. 5TYKCh. 48 - Prob. 6TYKCh. 48 - Prob. 7TYKCh. 48 - After the rapid depolarization of an action...Ch. 48 - Nodes of Ranvier are a. gaps where Schwann cells...Ch. 48 - Prob. 10TYKCh. 48 - Signal transmission is faster in myelinated axons...Ch. 48 - Which of the following statements concerning...Ch. 48 - Prob. 13TYKCh. 48 - Prob. 14TYKCh. 48 - Prob. 15TYKCh. 48 - Prob. 16TYKCh. 48 - Prob. 17TYKCh. 48 - If the binding of a neurotransmitter to its...
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- How do neurotransmitters directly affect the postsynaptic neuron? A)They cause a change in the electrical potential, exciting the neuron and causing it to fire b)They cause a change in the electrical potential, either exciting or inhibiting the next neuron. C)They cause a change in the electrical potential, inhibiting the neuron and stopping it from fi d)They connect with the appropriate receptor on the postsynaptic neuronarrow_forwardWhich of these is least likely to trigger an action potential in a post-synaptic cell? A)EPSPs and IPSPs arrive simultaneously at multiple synapses. B)The other choices are equally likely to trigger an action potential. C)Single EPSPs arrive simultaneously at multiple synapses. D)Multiple EPSPs arrive rapidly at a single synapse.arrow_forwardNerve membrane hyperpolarization after an action potential a)Is the movement of membrane potential voltage below normal resting potential voltage. b) This Is mostly due to the slow-closing of K+ channels. c)Makes it more difficult to evoke another action potential. d) This Is mostly due to the slow-closing of Na+ channels.arrow_forward
- What membrane protein of the neuron is responsible for briefly increasing the Na+ permeability of the membrane during the rising phase of the nerve impulse? a. the sodium-potassium pump b. the voltage gated potassium channel c. the voltage gated sodium channel d. the stimulus gated sodium channel e. the ligand gated ion channelarrow_forwardIf you increase the amount of neurotransmitter in the synaptic cleft, how would you expect the post-synaptic potential (PSP) to change? a) The PSP would be stronger b) The PSP would change from depolarizing to hyperpolarizing c) The PSP would not changearrow_forwardThe extracellular sodium [Na+]0 is reduced in the saline bath. Following another current injection in a neuron, the membrane potential changes were recorded. a) Why has the membrane potential changed following the Na+ reduction? b) Why has the current injection produced no action potentials? c) How might you experimentally rescue action potential generation?arrow_forward
- There is a type of toxin found in the liver of the puffer fish that prevents the opening of voltage-gated Na+ channels in neurons. The effect of this toxin is to: Group of answer choices A. prevents the initiation of the action potential B. depolarizes the membrane potential and maintains it depolarized C. prolongs the return of the membrane potential to the resting level D. increases the duration of the action potentialarrow_forwardWhich of the following is the role of axonal Calcium (Ca2+) channels in neuronal communication at the synapse? a) they inhibit the action potential causing it to stop b) Calcium channels have no role in this process c) by allowing calcium to exit the cell, they promote the propagation of action potential across the synapse by creating depolarization d) by blocking calcium entrance, they mediate the contraction of presynaptic axon to facilitate movement e) when they open, calcium enters the presynaptic terminal, promoting fusion of neurotransmitter vesicles with presynaptic membrane, and release of neurotransmitters to the synaptic cleftarrow_forwardA common feature of action potentials is that they(A) cause the membrane to hyperpolarize and then depolarize.(B) can undergo temporal and spatial summation.(C) are triggered by a depolarization that reaches threshold.(D) move at the same speed along all axons.arrow_forward
- Charge from depolarization along the axon reaches the axon terminal, but there is a lack of neurotransmitter release. Which of the following would cause this? Group of answer choices A. Lack of Na+ in the synaptic cleft B. Lack of Ca2+ in the synaptic cleft C. Reduced amount of K+ at the axon terminal D. Lack of voltage-gated sodium channels in the axon terminalarrow_forwardWhich one of these is an example of an excitatory postynaptic potential in a typical neuron? a) A voltage change from -70mV to -70.5mV b) A voltage change from +35mV to 0MV c) A voltage change from 0mV to +0.35mV d) A voltage change from -69.5mV to -70mVarrow_forwardWhen summation of multiple graded synaptic potentials occurs which of the following would "neutralize/cancel out" an EPSP in dendrites? A. opening a voltage gated sodium channel B. opening a voltage gated calcium channel C. openning a ligand gated calcium channel D. closing a ligand gated potassium channel E. opening a ligand gated chloride channelarrow_forward
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