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What do you mean by Use of Dexamethasone in the context of COVID 19? Please add diagrams or figures supporting your answer. Please briefly explain at your own easy words. I will rate you positive if you do so.   Please don't use AI for answering this question.

Turnover of RNA and protein helps to control gene expression by influencing the concentration of these intracellular molecules at certain times * True false

Vo) 4G+ 10:57 C LTE1 ll 81% Aishwarya 3 minutes ago Explain what MRNA is and rRNA is.

Define C-value paradox

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Please show complete explanation/justification/solutions clearly

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"Schematic outline of melanogenesis. UVR stimulates the expression of POMC by keratinocytes. The peptide produced is the precursor of the hormone a-MSH, that binds to MC1R in melanocytes. This union leads to an increase in cellular cyclic adenosine monophosphate (cAMP) that in turn leads to increased levels of MITF expression, which upregulates the transcription of TYR, TYRP1 and TYRP2, producing brown-black eumelanin. In the absence of a-MSH, the antagonist ASIP binds to MC1R, and phaeomelanin is synthetized instead. Melanosomes are then transferred to keratinocytes through the dendrites via a shedding vesicle system."   Briefly discuss how these several aspects of the molecular and cellular biology of melanin production and melanosome transfer contribute to determine skin color. How would different mutations in the hormonal response to ultraviolet light, the melanin production signal transduction pathways, and melanosome transfer influence skin color, hair color, and eye color? Did…

Q38. The ΔF508 mutation of the CFTR gene is the cause of most cases of cystic fibrosis (CF). One focus of research into a cure for CF is "gene therapy" — inserting normal copies of the CFTR gene into the cells of CF patients. Which of the following areas of CF research is most likely to improve symptoms for CF patients?   A. Inserting extra copies of genes that code for the proteosome. B. Studying the chaperone proteins of the endoplasmic reticulum (ER). C. Improving the lung function of people who are carriers of the CF allele (Cc). D. Stabilizing the plasma membrane were the ΔF508 form of the protein accumulates.

Transduction: Describe the process of generalized transduction as a type of horizontal transfer. In your response be sure to address the following:Outline the steps needed in transduction including an explanation of any terms (i.e. transducing particle).

expand this into 1000 words. the context is Identification of Differentially Expressed Genes in Breast Cancer Using RNA-Seq "In investigating the impact of TONSL overexpression on cellular physiology and gene regulation, differential expression analysis emerges as a pivotal analytical approach. By comparing gene expression profiles between primary cells and TONSL-overexpressing cells, researchers can identify genes whose expression levels are significantly altered. This analysis not only sheds light on the direct targets of TONSL but also unveils broader regulatory networks and pathways influenced by TONSL overexpression. Moreover, by pinpointing key genes involved, researchers can discern potential molecular mechanisms underlying the observed phenotypic changes associated with TONSL overexpression, offering valuable insights into TONSL's role in cellular homeostasis and function. Furthermore, functional enrichment analysis stands as a complementary approach to understanding the…

Please help me with this question. More than one answer may be correct. THe graph relating to the information is included below. The figure shows the number of cells that have clusters of IRE1 molecules after those cells are treated with various levels of thapsigargin (Tg), a chemical that can induce ER stress. IRE1 can form these clusters when ER stress is induced and this clustering can cause activation of RNAse activity in IRE1. In this experiment, normal IRE1 was used (IRE1α) that can bind to Sec61, along with a modified version of IRE1 that binds to Sec61 more weakly than normal IRE1 (wIRE1α), and another modified version that binds to Sec61 more strongly than normal IRE1 (sIRE1α). From this figure you can conclude that: Question 18 options: IRE1 binding to Sec61 promotes the formation of IRE1 clumps IRE1 binding to Sec61 prevents the formation of IRE1 clumps co-translational translocation is a key process the golgi aparatus is heavily involved in the unfolded protein response The…

The maximum light absorption for the DNS assay:595nm240nm295nm540n

Throughout downstream processing, various analytical methods must be used to evaluate the effectiveness of unit operations. Provide a method or methods (if more than one is required) for each scenario. Assess total protein in rice flour (solid material). Visualize antibody fragments (heavy chain and light chain) and compare molecular weights. Quick determination of the concentration of a purified sample of monoclonal antibody. Determine the concentration of monoclonal antibody in clarified extract. Study aggregation of a protein product without disruption of tertiary protein structure. Characterize phenolics (secondary metabolites and impurities produced by green plant tissue) by identifying phenolic species and relative concentrations. Detect protein variants that differ by a substitution of two amino acids. Quantify the level of endotoxin in the final purified product. Quantify bacterial contamination in your final formulated product.

Q: on Transcription process Briefly describe the differences in the transcription and translation processes as they occur in prokaryotic cells and eukaryotic cells. Thank you very much for your help.

What is dosage compensation? Give its importance.  explain please

coded for oxyte Based on your pases would h oxytocin?

Q.Consider this problem: You are working in the lab to study the pattern of paralysis and  candidate genes involved in this process. You revealed that homozygous mutation in the SLB gene is the main cause of paralysis. The following sequence of SLB gene at the beginning of the  translated region is found in individuals without paralysis. 5‟-GTA GCA TTT AAG CTT CAG  TCC AAG - 3‟ (Met Thr Phe Glu Ile Gln Ser Arg). This sequence is however changed to the  following sequence in affected individuals 5‟- GTA GCA TTT AAG CTT TAG TCC AAG - 3‟  (Met Thr Phe Glu Ile STOP). What mutation you will identify from this observation? Explain with reason. After identifying mutation if there is any, enlist all the possible repair mechanisms  that can be helpful to repair the current situation. Elaborate your answer with that why you think  that your suggested repair pathway should be used and how it can be effective for repairing in  the current scenario?

13 Match each of the QPCR samples (i-v) with the correct amplification plot (A-E) and determine the Ct value for each of the amplification curves. Number of template copies in the QPCR reaction: Amplification plot (A-E): Ct Value 1200 1000 i. 1X 106 ii. 1X 105 iii. 1X 108 iv. 1X 107 V. NTC (No template control) 800 600 400 200 A C 0 5 10 15 20 25 30 Cycles vi. Create a standard curve graph of template copies vs Ct with the data in the table above. Log of Starting Quantity of DNA vrs C+ vii. 26 24 22221 20 18 16 14 12 כי 10 8 6 4 2 0 5 5.5 6 6.5 7 7.5 8 Log Starting Quantity of DNA (Template copies) If a patient sample has a Ct of 20, interpolate the number of template copies at the start of the PCR reaction using the standard curve you created above?

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. Please explain the role of Ubiquitin Ligase Activity in the regulation of apoptotic cell death.

16b this was not graded and will not be graded

Answer in step by step with explanation. Don't use Ai and chatgpt. Answer in all options.

Link: Lack of RAC1 in macrophages protects against atherosclerosis - PMC (nih.gov) Could somone explain exactly what this means below? NOT HW just trying to get a better understanding on what this experiment is about.  To produce mice that are deficient for RAC1 in macrophages, female C57BL/6 mice homozygously expressing the floxed Rac1 gene (Rac1fl/fl) [6] were crossbred with male mice homozygously expressing Cre under the monocyte-specific lysozyme M promoter (LC) [12]. Mice were genotyped for Rac1 deficiency as previously described

Retinoblastoma: The Hits Just Keep Coming case study Part III – The Second Hit Questions A second hit might occur through epigenetic alterations. In the promoter of RB1, there is a CpG island. Knowing this, how might you predict that a cell could epigenetically inactivate RB1 transcription?   A second hit might also occur through loss of heterozygosity (LOH). An example of how LOH may occur by reciprocal crossing over during mitosis is diagrammed in Figure 1 (next page). Discuss and interpret this model with your group. Write a brief explanation of (a) what LOH means and (b) how LOH by mitotic reciprocal crossing over can give rise to a cell lineage with functional loss of the wild-type copy of a tumor suppressor gene.     . One of the ways that we know what the RB protein does in cells is that its inactivation is a common priority of tumor-initiating viruses. What advantage would a virus gain by inactivating RB function in its host cell? One example of a DNA virus (a virus that…