BIOCHEM-ACHIEVE(FIRST DAY DISCOUNTED)
9th Edition
ISBN: 2818000069358
Author: BERG
Publisher: MAC HIGHER
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Chapter 21, Problem 40P
Interpretation Introduction
Interpretation:
Other UDP-carbohydrate encountered in the study of biochemistry should be discussed.
Concept introduction:
UDP- carbohydrates fall under
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Students have asked these similar questions
A sample of glycogen from a patient with liver disease is incubated with orthophosphate, phosphorylase, the transferase, and the debranching enzyme ( α -1,6- glucosidase). The ratio of glucose 1- phosphate to glucose formed in this mixture is 100. What is the most likely enzymatic deficiency in this patient?
NH,
This compound is directly involved in:
он
O=P-0-CH,
H
но
OmP-0-
O oxidizing an alcohol in the conversion of sucrose to fructose.
O the synthesis of malonyl coenzyme A.
reducing a ketone in gluconeogenesis.
reducing a C=C double bond in fatty-acid synthesis.
In class, I mentioned that fructose is metabolized differently in the liver compared to glucose.
Refer to the figure shown below to calculate the number ofATPs you would expect from the metabolism of
fructose in the liver. Show your work!
Fructokinase
Fructose
Fructose-1-P
АТР
ADP
Aldolase B
Dihydroxy-
acetone
phosphate
Glyceraldehyde
АТР
Triose kinase
Triose
phosphate
isomerase
ADP 4
- Glyceraldehyde-3-P
Glycolysis
Руruvate
Acetyl-CoA
Fatty acids and
triglycerides
Chapter 21 Solutions
BIOCHEM-ACHIEVE(FIRST DAY DISCOUNTED)
Ch. 21 - Prob. 1PCh. 21 - Prob. 2PCh. 21 - Prob. 3PCh. 21 - Prob. 4PCh. 21 - Prob. 5PCh. 21 - Prob. 6PCh. 21 - Prob. 7PCh. 21 - Prob. 8PCh. 21 - Prob. 9PCh. 21 - Prob. 10P
Ch. 21 - Prob. 11PCh. 21 - Prob. 12PCh. 21 - Prob. 13PCh. 21 - Prob. 14PCh. 21 - Prob. 15PCh. 21 - Prob. 16PCh. 21 - Prob. 17PCh. 21 - Prob. 18PCh. 21 - Prob. 19PCh. 21 - Prob. 20PCh. 21 - Prob. 21PCh. 21 - Prob. 22PCh. 21 - Prob. 23PCh. 21 - Prob. 24PCh. 21 - Prob. 25PCh. 21 - Prob. 26PCh. 21 - Prob. 27PCh. 21 - Prob. 28PCh. 21 - Prob. 29PCh. 21 - Prob. 30PCh. 21 - Prob. 31PCh. 21 - Prob. 32PCh. 21 - Prob. 33PCh. 21 - Prob. 34PCh. 21 - Prob. 35PCh. 21 - Prob. 36PCh. 21 - Prob. 37PCh. 21 - Prob. 38PCh. 21 - Prob. 39PCh. 21 - Prob. 40PCh. 21 - Prob. 41PCh. 21 - Prob. 42PCh. 21 - Prob. 43PCh. 21 - Prob. 44PCh. 21 - Prob. 45PCh. 21 - Prob. 46P
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- Which of the following is NOT part of the multi-enzyme complex of fatty acid synthase? O enoyl-ACP reductase O acetyl CoA carboxylase O 3-hydroxybutyrylACP-dehydratese O B-ketoacyl-ACP reductasearrow_forwardYears ago there was interest in using uncouplers such as dinitrophenol as weight control agents. Presumably, fat could be oxidized without concomi- tant ATP synthesis for re-formation of fat or carbohydrate. Why was this a bad (i.e., fatal) idea?arrow_forwardCan the carbons in ketones be metabolized and wind up in a glucose monosaccharide? If yes, using merely the information presented in class, outline a possible metabolic route for these carbons. If no, explain why not.arrow_forward
- The complete oxidation of glucose 6-phosphate derived from free glucose yields 30 molecules ATP, whereas the complete oxidation of glucose 6- phosphate derived from glycogen yields 31 molecules of ATP. Account for this difference.arrow_forwardFor saturated fatty acids, these reactions can continue as you have drawn until all the carbons of the fatty acid have been oxidized to acetyl-CoA. What is problematic about an unsaturated fatty acid, such as C16:cis-9?arrow_forwardA sample of glycogen from a patient with liver disease is incubated with Pi, normal glycogen phosphorylase, and normal debranching enzyme. The ratio of glucose 1-phophate to glucose in the reaction mixture is 100. What is the patient’s most likely enzymatic deficiency?arrow_forward
- Triacylglycerols in adipose tissue do not enter the bloodstream as triacyglycerolsexplainarrow_forwardFatty acids are oxidized by acyl COA dehydrogenase. Branched fatty acids, containing a methyl group at beta carbons cannot be oxidized by this enzyme. How does the oxidation of such branched fatty acids take place? Explain with all set of reactions and enzymes involved.. give necessary detail.. avoid cpy rightingarrow_forwardWhat reaction precedes decarboxylation in the conversion of glucose to ribose in the pentose phosphate pathway? Formation of a C-2 alpha carbonyl Dehydrogenation of C-2 to form a lactone Oxidation of the C-2 ketone to carboxylic acid Oxidation of the C-1 aldehyde to carboxylic acidarrow_forward
- Can you please give me more expaination for the question below? I know the equision of Meachile, but I would like to learn what is your theory to draw the graph, I mean why Glycogen is refered as [s] and V max = Product formed glycogen? An experiment was performed to determine the effects of an inhibitor on the breakdown of glycogen by an enzyme. In an accompanying experiment, the inhibitor was added to the glycogen-enzyme suspension and reacted using the same experimental conditions. The data obtained from these experiments is tabulated below. Glycogen (mM) Product Formed Glycogen only (mM/min) Product Formed Glycogen and Inhibitor (mM/min) 0.5 22.6 15.9 1.0 32.1 24.9 1.5 38.1 28.2 2.0 41.2 35.1 2.5 44.7 40.0 3.0 48.5 43.3arrow_forwardWith fatty infiltration of the liver, the synthesis of phospholipids is disturbed. What is the biochemical basis of the development of such a condition? What are its possible nests? Justify the answer.arrow_forwardWe have stated that fatty acids cannot yield a net gain in carbohydrates in organisms that lack a glyoxalate pathway. Why can odd-chain fatty acids be thought to break this rule to a small extent?arrow_forward
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