Biology - Study Guide
8th Edition
ISBN: 9780321501561
Author: Martha R. Taylor
Publisher: PEARSON EDUCATION (COLLEGE)
expand_more
expand_more
format_list_bulleted
Textbook Question
Chapter 11, Problem 9IQ
- a. What can one conclude from the fact that the proteins involved in mitochondrial apoptosis in mammals are similar to the Ced-3, Ced-4 and Ced-9 proteins of nematodes?
- b. Give some examples of programmed cell death in humans.
Expert Solution & Answer
Want to see the full answer?
Check out a sample textbook solutionStudents have asked these similar questions
Describe an experiment that shows if early mouse embryo inner cell mass cells containing mitochondrial defects die through necrosis of apoptosis.
Discuss the complete cell cycle in a human cell, mitosis and meiosis, and the regulatory components (i.e. the proteins associated with cellular checkpoints) of the cell cycle.
Tumor growth results when the cell cycle checkpoints are ignored. Give an example of how tumor growth could result from either a loss-of-function or a gain-of-function mutation.
Compare and contrast the genetic defects that can lead to disruption of a tumor suppressor gene compared to the mechanisms for activation of oncogenes.
Chapter 11 Solutions
Biology - Study Guide
Ch. 11 - How do plant hormones (often called plant growth...Ch. 11 - Prob. 2IQCh. 11 - Label the parts in the following diagram of an...Ch. 11 - Prob. 4IQCh. 11 - a. What does a protein kinase do? b. What is a...Ch. 11 - Prob. 6IQCh. 11 - Prob. 7IQCh. 11 - Prob. 8IQCh. 11 - a. What can one conclude from the fact that the...Ch. 11 - Prob. 1SYK
Ch. 11 - Briefly describe the three stages of cell...Ch. 11 - Some signaling pathways alter a proteins activity;...Ch. 11 - Prob. 4SYKCh. 11 - Prob. 1TYKCh. 11 - Steroid hormones a. bind to cell-surface...Ch. 11 - Prob. 3TYKCh. 11 - Prob. 4TYKCh. 11 - Which of the following is a key difference between...Ch. 11 - Many human diseases (including bacterial...Ch. 11 - Prob. 7TYKCh. 11 - If a protein is activated by the addition of a...Ch. 11 - Prob. 9TYKCh. 11 - Prob. 10TYKCh. 11 - From studying the effects of epinephrine on liver...Ch. 11 - You are a Ca2+ ion in a typical animal cell. Where...Ch. 11 - Which of the following molecules is incorrectly...Ch. 11 - Prob. 14TYKCh. 11 - Prob. 15TYK
Knowledge Booster
Learn more about
Need a deep-dive on the concept behind this application? Look no further. Learn more about this topic, biology and related others by exploring similar questions and additional content below.Similar questions
- Describe the effects of the over-expression of mdm2 on cell proliferation and apoptosis on cell signaling pathways and metabolism or cell cycle control. Briefly explain the normal role of each component in the context of the pathway and why its loss or modification would have the expected effect.arrow_forwardIn typical women of reproductive age, the epithelial cells lining the uterus (the endometrium) die and are shed from the body roughly once every month, in the process of menstruation. The triggers in this process include chemical (hormonal) changes and contraction of the blood vessels in the lining. Cell death may be a result of either apoptosis or necrosis. a. Describe the characteristics of each process (apoptosis and necrosis). b.Develop an argument for classifying the process of menstruation as an example of apoptosis, and then argue for classifying it as necrosis. c.Explain which is correct, and provide the supporting evidence?arrow_forward(a) the cell cycle is divided into different phases (referred to as M, G0, G1, S and G2); Can you account for what happens during the different parts of the cycle and out of it is checked? ( b) What is the difference between apoptosis and necrosis? ( c) What are caspases and what is their function in the cell?arrow_forward
- Hi, can you elaborate about the following: certain characterize mutations in p63 and compare that to known mutations in p53.arrow_forwardHelp constructing a concept map? Example provided below. Many studies have indicated that autophagy and apoptosis play an important role in the pathogenesis of spinal cord injury. In recent years, research on autophagy-related signal transduction pathways has demonstrated that the phosphatidylinositol-3-kinase (PI3K)/protein kinase B (Akt)/mammalian target of rapamycin (mTOR) signaling pathway is closely associated with the initiation of autophagy. However, the mechanism of the pathological relationship between this signaling pathway and apoptosis in spinal cord injury is unclear. In this study, we used an in vitro model of spinal cord injury to observe the mechanism of the PI3K/Akt/mTOR signaling pathway and the apoptosis of neurons via the mitochondrial pathway. Mitochondrial pathway apoptosis-related proteins were detected by western blot. Akt and mTOR phosphorylation levels peaked 4 h after mechanical damage and then decreased. Following administration of the PI3K-specific…arrow_forwardNeoplasia in Fisha. In a table, compare and contrast the differences occurring in a cell undergoing hyperplasiaand neoplasia.b. Describe in details how to diagnose a hyperplasic tissue from a neoplastic growth.c. Discuss how the sex of an organism is a factor in the occurrence of neoplasia in fish.d. Compare and contrast how chemical radiation and viruses causes neoplastic diseases in fish.e. List and describe the viruses that are known to cause cancer in fish and their mode ofpathogenesis.f. Describe the bioactivities of aflatoxin, PAH and halogenated compounds that could led tocancer development in fish.arrow_forward
- What would be the likely result of a mutation of the bcl-2 gene on the level of apoptosis? a. No change b. A decrease in apoptosis c. An increase in apoptosis d. An initial decrease, followed by an increase in apoptosisarrow_forward1) Discuss the roles of epidermal growth factor receptor (EGFR) in normal cell signalling transduction and in cancer development. 2) Describe uniparental inheritance of mitochondrial DNA and discuss its importance in human disease and potential treatment strategiesarrow_forwardDiscuss/explain, in details, the relationship between cell viability and cell vitality and cell apoptosis as suggested by neutral red uptake,arrow_forward
- Cancer-promoting mutations are likely to have different effects on the activity of proteins encoded byproto-oncogenes than they do on proteins encodedby tumor-suppressor genes. Explain.arrow_forwardMost forms of cancer involvea. the activation of a single oncogene.b. the inactivation of a single tumor-suppressor gene.c. the activation of multiple oncogenes.d. the activation of multiple oncogenes and the inactivation ofmultiple tumor-suppressor genes.arrow_forwardDescribe/explain the role of thromboxane (TXA4) in animal cells/organsarrow_forward
arrow_back_ios
SEE MORE QUESTIONS
arrow_forward_ios
Recommended textbooks for you
- BiochemistryBiochemistryISBN:9781305577206Author:Reginald H. Garrett, Charles M. GrishamPublisher:Cengage LearningBiology 2eBiologyISBN:9781947172517Author:Matthew Douglas, Jung Choi, Mary Ann ClarkPublisher:OpenStax
Biochemistry
Biochemistry
ISBN:9781305577206
Author:Reginald H. Garrett, Charles M. Grisham
Publisher:Cengage Learning
Biology 2e
Biology
ISBN:9781947172517
Author:Matthew Douglas, Jung Choi, Mary Ann Clark
Publisher:OpenStax
cell division of meiosis and mitosis; Author: Stated Clearly;https://www.youtube.com/watch?v=A-mFPZLLbHI;License: Standard youtube license