Prescott's Microbiology
11th Edition
ISBN: 9781260211887
Author: WILLEY, Sandman, Wood
Publisher: McGraw Hill
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Chapter 15, Problem 4AL
In the chapter opening story, it was stated that the next step in using algae to produce plastics is to maximize expression of the bacterial poly hydroxyalkanoate-synthesizing gene in algal cells. Suggest one genetic change that might be made to increase its expression.
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Chapter 15 Solutions
Prescott's Microbiology
Ch. 15.2 - Prob. 1CCCh. 15.2 - Prob. 2CCCh. 15.2 - Prob. 3CCCh. 15.2 - Prob. 4CCCh. 15.2 - Prob. 5CCCh. 15.3 - What functions are served by the 5 cap and the 3...Ch. 15.3 - Retrieve, Infer, Apply What elements in archaeal...Ch. 15.3 - Prob. 2CCCh. 15.3 - Prob. 3CCCh. 15.4 - Prob. 1CC
Ch. 15.4 - Prob. 2CCCh. 15.4 - Prob. 3CCCh. 15.5 - Retrieve, Infer, Apply List two similarities and...Ch. 15.5 - Prob. 2CCCh. 15.5 - Retrieve, Infer, Apply How are cis-encoded RNAs,...Ch. 15 - Prob. 1RCCh. 15 - Prob. 2RCCh. 15 - Prob. 3RCCh. 15 - Prob. 4RCCh. 15 - Prob. 5RCCh. 15 - Prob. 6RCCh. 15 - Prob. 7RCCh. 15 - Prob. 8RCCh. 15 - Prob. 9RCCh. 15 - Prob. 1ALCh. 15 - All of the subunits in bacterial RNA polymerases...Ch. 15 - Would you expect that one day microbiologists...Ch. 15 - In the chapter opening story, it was stated that...Ch. 15 - Prob. 5AL
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- What would happen to the regulation of the tryptophan operon in bacterial cells that express a mutant form of the tryptophan repressor that (1) cannot bind to DNA, (2) cannot bind tryptophan, or (3) binds to DNA even in the absence of tryptophan?arrow_forwardA crucial step in the regulation of many bacterial genes is the binding of RNA polymerase to DNA at the promoter. Why might it be advantageous for bacteria to regulate the expression of their genes at this particular step?arrow_forwardThe researchers claim that treatment with a compound that inhibits dephosphorylation of the intracellular domain of the Brec protein will inhibit expression of A in Brec-WT cells but not in Brec-MUT cells. Based on the information provided, provide reasoning to refute their claim.arrow_forward
- An individual carries a somatic mutation that changes a lysinecodon into a glutamic acid codon. Prior to acquiring this mutation,the individual had been exposed to UV light, proflavin, and5-bromouracil. Which of these three agents would be the mostlikely to have caused this somatic mutation? Explain your answer.arrow_forwardDescribe the positive outcome of mutation to the regulatory DNA for expression of the lactase gene. Why was this mutation important to the human population?arrow_forwardWhile investigating the function of a specific growth factor receptor gene from humans, researchers found that two types of proteins are synthesized from this gene. A larger protein containing a membrane spanning domain recognizes growth factors at the cell surface, stimulating a specific downstream signaling pathway. In contrast, a related, smaller protein is secreted from the cell and binds available growth factor circulating in the blood, thus inhibiting the downstream signaling pathway. Speculate on how the cell synthesizes these disparate proteins.arrow_forward
- You suspect that the mRNA encoding the guanine biosynthetic genes of Bacillus subtilus contains an aptamer that binds guanine leading to premature termination of transcription. Propose a way you can test this hypothesisarrow_forwardArginine is one of the amino acids that serves as a building block for protein synthesis. Arginine biosynthesis depends on the expression of the arg operon, which encodes ArgC (a reductase) and ArgB (acetylglutamate kinase). Which of the following statements regarding mutation of the arginine biosynthesis repressor ArgR is true? O A 1-bp insertion in the argR gene would result in a mutant able to grow in the absence of arginine, O A 1-bp deletion in the argB gene would result in a mutant unable to transcriptionally regulate the arg operon encoding argB. O A 1bp insertion in the argC gene would result in a mutant able to grow in the absence of arginine. O A 1-bp deletion in the argR gene would result in a mutant that represses the arg operon when grown in the presence of arginine. None of the above.arrow_forwardSuppose that E. coli sustains a mutation in its gene for the lac operon repressor making the repressor ineffective . How would this mutation affect the bacterium's ability to catabolize lactose? Would the mutant strain have an advantage over the wild-type strain? Explain your answer.arrow_forward
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